(Q39561716)

13 September 2017

title

Measles virus C protein suppresses gamma-activated factor formation and virus-induced cell growth arrest (English)

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13 September 2017

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3

Nobuhiro Fujii

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13 September 2017

2

Tamaki Okabayashi

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13 September 2017

Shin-Ichi Yokota

1

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13 September 2017

language of work or name

English

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publication date

7 April 2011

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13 September 2017

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13 September 2017

volume

414

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13 September 2017

issue

1

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13 September 2017

page(s)

74-82

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Identification of naturally occurring amino acid variations that affect the ability of the measles virus C protein to regulate genome replication and transcription

13 September 2017

cites work

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2011.03.010

Inhibition of IFN-alpha/beta signaling by two discrete peptides within measles virus V protein that specifically bind STAT1 and STAT2.

7 January 2021

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https://api.crossref.org/works/10.1016%2FJ.VIROL.2011.03.010

Measles virus V protein blocks Jak1-mediated phosphorylation of STAT1 to escape IFN-alpha/beta signaling

7 January 2021

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https://api.crossref.org/works/10.1016%2FJ.VIROL.2011.03.010

Measles virus editing provides an additional cysteine-rich protein

7 January 2021

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Measles virus phosphoprotein gene products: conformational flexibility of the P/V protein amino-terminal domain and C protein infectivity factor function

7 January 2021

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https://api.crossref.org/works/10.1016%2FJ.VIROL.2011.03.010

Regulation of interferon signaling by the C and V proteins from attenuated and wild-type strains of measles virus

7 January 2021

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Interferons: cell signalling, immune modulation, antiviral response and virus countermeasures

7 January 2021

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Identification of a novel cytokine response element in the human IFN regulatory factor-1 gene promoter

7 January 2021

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7 January 2021

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Mechanisms of protein kinase PKR-mediated amplification of beta interferon induction by C protein-deficient measles virus

7 January 2021

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https://api.crossref.org/works/10.1016%2FJ.VIROL.2011.03.010

Translational inhibition and increased interferon induction in cells infected with C protein-deficient measles virus

7 January 2021

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Measles virus circumvents the host interferon response by different actions of the C and V proteins.

7 January 2021

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https://api.crossref.org/works/10.1016%2FJ.VIROL.2011.03.010

The C protein of wild-type measles virus has the ability to shuttle between the nucleus and the cytoplasm

7 January 2021

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https://api.crossref.org/works/10.1016%2FJ.VIROL.2011.03.010

Dissection of measles virus V protein in relation to its ability to block alpha/beta interferon signal transduction

7 January 2021

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Dissociation of paramyxovirus interferon evasion activities: universal and virus-specific requirements for conserved V protein amino acids in MDA5 interference

7 January 2021

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The C protein of measles virus inhibits the type I interferon response

7 January 2021

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How cells respond to interferons

7 January 2021

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Measles virus V protein blocks interferon (IFN)-alpha/beta but not IFN-gamma signaling by inhibiting STAT1 and STAT2 phosphorylation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2011.03.010

Protein kinase PKR mediates the apoptosis induction and growth restriction phenotypes of C protein-deficient measles virus

7 January 2021

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Growth arrest of epithelial cells during measles virus infection is caused by upregulation of interferon regulatory factor 1.

7 January 2021

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Measles virus P protein suppresses Toll-like receptor signal through up-regulation of ubiquitin-modifying enzyme A20.

7 January 2021

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https://api.crossref.org/works/10.1016%2FJ.VIROL.2011.03.010

Measles virus suppresses interferon-alpha signaling pathway: suppression of Jak1 phosphorylation and association of viral accessory proteins, C and V, with interferon-alpha receptor complex

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2011.03.010

Cytosolic chaperonin-containing t-complex polypeptide 1 changes the content of a particular subunit species concomitant with substrate binding and folding activities during the cell cycle

7 January 2021

1 reference

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Herpes simplex virus type 1 suppresses the interferon signaling pathway by inhibiting phosphorylation of STATs and janus kinases during an early infection stage

7 January 2021

1 reference

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10.1016/J.VIROL.2011.03.010

7 January 2021

Identifiers

DOI

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13 September 2017

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