(Q39703814)

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Activation of the PI3K-Akt pathway by human T cell leukemia virus type 1 (HTLV-1) oncoprotein Tax increases Bcl3 expression, which is associated with enhanced growth of HTLV-1-infected T cells

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Europe PubMed Central

PubMed publication ID

14 September 2017

Activation of the PI3K-Akt pathway by human T cell leukemia virus type 1 (HTLV-1) oncoprotein Tax increases Bcl3 expression, which is associated with enhanced growth of HTLV-1-infected T cells (English)

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14 September 2017

Human T-lymphotropic virus 1

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Kousuke Saito

1

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Europe PubMed Central

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14 September 2017

Mineki Saito

2

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14 September 2017

Naoko Taniura

3

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14 September 2017

Takako Okuwa

4

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14 September 2017

Yoshiro Ohara

5

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14 September 2017

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14 May 2010

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14 September 2017

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14 September 2017

403

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14 September 2017

2

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14 September 2017

173-180

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Europe PubMed Central

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14 September 2017

Negative regulation of toll-like receptor signaling by NF-kappaB p50 ubiquitination blockade

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HTLV-1 Tax and adult T-cell leukemia

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Respiratory syncytial virus-inducible BCL-3 expression antagonizes the STAT/IRF and NF-kappaB signaling pathways by inducing histone deacetylase 1 recruitment to the interleukin-8 promoter

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Activated AKT regulates NF-kappaB activation, p53 inhibition and cell survival in HTLV-1-transformed cells

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The proto-oncogene bcl-3 encodes an I kappa B protein

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The proto-oncogene Bcl3, induced by Tax, represses Tax-mediated transcription via p300 displacement from the human T-cell leukemia virus type 1 promoter

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Epstein-Barr virus latent membrane protein 1 induces expression of the epidermal growth factor receptor through effects on Bcl-3 and STAT3.

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Impact of HTLV-I Tax on cell cycle progression and the cellular DNA damage repair response

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Human T-cell leukaemia virus type 1 (HTLV-1) infectivity and cellular transformation

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Cloning of the chromosome translocation breakpoint junction of the t(14;19) in chronic lymphocytic leukemia

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HTLV-I-associated myelopathy: analysis of 213 patients based on clinical features and laboratory findings.

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The latent membrane protein 1 (LMP1) encoded by Epstein-Barr virus induces expression of the putative oncogene Bcl-3 through activation of the nuclear factor-kappaB.

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Cell cycle dysregulation by HTLV-I: role of the tax oncoprotein

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The candidate proto-oncogene bcl-3 is related to genes implicated in cell lineage determination and cell cycle control

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HTLV-I associated myelopathy, a new clinical entity.

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Bcl-3, a multifaceted modulator of NF-kappaB-mediated gene transcription

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Up-regulation of cyclin D1 by HBx is mediated by NF-kappaB2/BCL3 complex through kappaB site of cyclin D1 promoter.

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p53 represses cyclin D1 transcription through down regulation of Bcl-3 and inducing increased association of the p52 NF-kappaB subunit with histone deacetylase 1.

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Human T-cell leukemia virus type 1 tax dysregulates beta-catenin signaling

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10.1016/J.VIROL.2010.04.018

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Europe PubMed Central

PubMed publication ID

14 September 2017

PubMed publication ID

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Europe PubMed Central

PubMed publication ID

14 September 2017

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