(Q39754547)

14 September 2017

The Parkinson-associated protein PINK1 interacts with Beclin1 and promotes autophagy (English)

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14 September 2017

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14

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14 September 2017

L Cassina

12

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Giuseppe Arena

G Arena

10

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Liliana Torosantucci

L Torosantucci

11

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Riccardo Vago

R Vago

9

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Patrizio Sale

P Sale

8

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author name string

S Michiorri

1

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14 September 2017

V Gelmetti

2

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14 September 2017

E Giarda

3

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14 September 2017

F Lombardi

4

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14 September 2017

F Romano

5

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14 September 2017

R Marongiu

6

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14 September 2017

S Nerini-Molteni

7

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14 September 2017

M A Russo

13

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14 September 2017

E M Valente

15

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14 September 2017

G Casari

16

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14 September 2017

8 January 2010

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Cell Death & Differentiation

14 September 2017

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14 September 2017

17

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14 September 2017

6

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14 September 2017

962-974

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https://scigraph.springernature.com/pub.10.1038/cdd.2009.200

14 September 2017

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cites work

Mendelian forms of Parkinson's disease

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https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Hereditary early-onset Parkinson's disease caused by mutations in PINK1

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Pathogenic mutations in Parkinson disease

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Wild-type PINK1 prevents basal and induced neuronal apoptosis, a protective effect abrogated by Parkinson disease-related mutations

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

PINK1 mutants associated with recessive Parkinson's disease are defective in inhibiting mitochondrial release of cytochrome c.

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

PINK1 is necessary for long term survival and mitochondrial function in human dopaminergic neurons

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https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Small interfering RNA targeting the PINK1 induces apoptosis in dopaminergic cells SH-SY5Y.

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Mitochondrial alterations in PINK1 deficient cells are influenced by calcineurin-dependent dephosphorylation of dynamin-related protein 1.

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

PINK1 protects against oxidative stress by phosphorylating mitochondrial chaperone TRAP1

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https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Cytoplasmic Pink1 activity protects neurons from dopaminergic neurotoxin MPTP.

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Characterization of PINK1 processing, stability, and subcellular localization

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https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Pink1 Parkinson mutations, the Cdc37/Hsp90 chaperones and Parkin all influence the maturation or subcellular distribution of Pink1.

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

The kinase domain of mitochondrial PINK1 faces the cytoplasm

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Parkin blushed by PINK1.

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Loss-of-function of human PINK1 results in mitochondrial pathology and can be rescued by parkin

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https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Pink1 regulates mitochondrial dynamics through interaction with the fission/fusion machinery

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https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

The PINK1-Parkin pathway is involved in the regulation of mitochondrial remodeling process.

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https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Loss of parkin or PINK1 function increases Drp1-dependent mitochondrial fragmentation

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https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Loss of PINK1 function promotes mitophagy through effects on oxidative stress and mitochondrial fission

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https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Parkin is recruited selectively to impaired mitochondria and promotes their autophagy

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https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Selective degradation of mitochondria by mitophagy

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Constitutive autophagy: vital role in clearance of unfavorable proteins in neurons.

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Autophagy and neurodegeneration: when the cleaning crew goes on strike

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Autophagy in neurodegeneration and development

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

High levels of Fis1, a pro-fission mitochondrial protein, trigger autophagy

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Mitochondrial fusion, fission and autophagy as a quality control axis: the bioenergetic view

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Pink1 forms a multiprotein complex with Miro and Milton, linking Pink1 function to mitochondrial trafficking

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Mitochondrial import and enzymatic activity of PINK1 mutants associated to recessive parkinsonism.

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Physiological functions of Atg6/Beclin 1: a unique autophagy-related protein

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Two Beclin 1-binding proteins, Atg14L and Rubicon, reciprocally regulate autophagy at different stages

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Effects of dopamine on LC3-II activation as a marker of autophagy in a neuroblastoma cell model.

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

How to interpret LC3 immunoblotting

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Methods for monitoring autophagy from yeast to human

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

OPA1 requires mitofusin 1 to promote mitochondrial fusion

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Bcl-2 antiapoptotic proteins inhibit Beclin 1-dependent autophagy

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Regulation of intracellular accumulation of mutant Huntingtin by Beclin 1

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

BH3-only proteins and BH3 mimetics induce autophagy by competitively disrupting the interaction between Beclin 1 and Bcl-2/Bcl-X(L)

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

DAP-kinase-mediated phosphorylation on the BH3 domain of beclin 1 promotes dissociation of beclin 1 from Bcl-XL and induction of autophagy

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Distinct classes of phosphatidylinositol 3'-kinases are involved in signaling pathways that control macroautophagy in HT-29 cells

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Discovery of Atg5/Atg7-independent alternative macroautophagy

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https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Beclin 1-independent pathway of damage-induced mitophagy and autophagic stress: implications for neurodegeneration and cell death

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https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Two-hybrid reporter vectors for gap repair cloning

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Studying inner ear protein-protein interactions using FRET and FLIM.

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https://api.crossref.org/works/10.1038%2FCDD.2009.200

7 January 2021

Identifiers

DOI

10.1038/CDD.2009.200

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Dimensions Publication ID

14 September 2017

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