(Q39767949)

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Development of a flow cytometric method for quantification of BCL-2 family members in chronic lymphocytic leukemia and correlation with sensitivity to BCL-2 family inhibitors

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scholarly article

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Europe PubMed Central

PubMed publication ID

14 September 2017

Development of a flow cytometric method for quantification of BCL-2 family members in chronic lymphocytic leukemia and correlation with sensitivity to BCL-2 family inhibitors (English)

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

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1 reference

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1 reference

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inferred from title

chronic lymphocytic leukemia

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author name string

Morey L Smith

1

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

Brenda Chyla

2

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Europe PubMed Central

PubMed publication ID

14 September 2017

Evelyn McKeegan

3

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Europe PubMed Central

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14 September 2017

Stephen K Tahir

4

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Europe PubMed Central

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14 September 2017

publication date

14 May 2016

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Europe PubMed Central

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14 September 2017

Cytometry Part B: Clinical Cytometry

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Europe PubMed Central

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14 September 2017

92

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Europe PubMed Central

PubMed publication ID

14 September 2017

5

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Europe PubMed Central

PubMed publication ID

14 September 2017

331-339

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Europe PubMed Central

PubMed publication ID

14 September 2017

Regulating cell death at, on, and in membranes.

1 reference

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The BCL-2 family reunion

1 reference

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Control of apoptosis by the BCL-2 protein family: implications for physiology and therapy.

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Discovery of a Potent and Selective BCL-XL Inhibitor with in Vivo Activity

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Genomic analysis and selective small molecule inhibition identifies BCL-X(L) as a critical survival factor in a subset of colorectal cancer

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Structure-guided design of a series of MCL-1 inhibitors with high affinity and selectivity

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Potent and selective small-molecule MCL-1 inhibitors demonstrate on-target cancer cell killing activity as single agents and in combination with ABT-263 (navitoclax).

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An inhibitor of Bcl-2 family proteins induces regression of solid tumours

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ABT-263: a potent and orally bioavailable Bcl-2 family inhibitor

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Integrative genomic analysis of small-cell lung carcinoma reveals correlates of sensitivity to bcl-2 antagonists and uncovers novel chromosomal gains

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Influence of Bcl-2 family members on the cellular response of small-cell lung cancer cell lines to ABT-737.

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Identification of expression signatures predictive of sensitivity to the Bcl-2 family member inhibitor ABT-263 in small cell lung carcinoma and leukemia/lymphoma cell lines

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Navitoclax (ABT-263) reduces Bcl-x(L)-mediated chemoresistance in ovarian cancer models

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A flow cytometric method to detect apoptosis-related protein expression in minimal residual disease in acute myeloid leukemia.

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Phase I study of oblimersen sodium, an antisense to Bcl-2, in untreated older patients with acute myeloid leukemia: pharmacokinetics, pharmacodynamics, and clinical activity.

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Amount of spontaneous apoptosis detected by Bax/Bcl-2 ratio predicts outcome in acute myeloid leukemia (AML).

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Stromal cells prevent apoptosis of AML cells by up-regulation of anti-apoptotic proteins

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Common polymorphism G(-248)A in the promoter region of the bax gene results in significantly shorter survival in patients with chronic lymphocytic Leukemia once treatment is initiated.

1 reference

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A New Bliss Independence Model to Analyze Drug Combination Data

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Antibody validation

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Recommendations for the validation of flow cytometric testing during drug development: II assays

1 reference

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Quantitative fluorescence cytometric analysis of Bcl-2 levels in tumor cells exhibiting a wide range of inherent Bcl-2 protein expression: correlation with Western blot analysis

1 reference

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7 January 2021

based on heuristic

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Quantitative analysis of bcl-2 expression in normal and leukemic human B-cell differentiation

1 reference

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7 January 2021

based on heuristic

inferred from DOI database lookup

Identifiers

10.1002/CYTO.B.21383

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

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