(Q39802987)

14 September 2017

title

Disruption of transepithelial resistance by enterotoxigenic Escherichia coli (English)

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14 September 2017

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3

Philip R Hardwidge

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14 September 2017

Amber M Johnson

1

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14 September 2017

Radhey S Kaushik

series ordinal

2

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14 September 2017

publication date

20 August 2009

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14 September 2017

published in

Veterinary Microbiology

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14 September 2017

volume

141

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14 September 2017

issue

1-2

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14 September 2017

page(s)

115-119

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Relative importance of heat-labile enterotoxin in the causation of severe diarrheal disease in the gnotobiotic piglet model by a strain of enterotoxigenic Escherichia coli that produces multiple enterotoxins

14 September 2017

cites work

1 reference

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Salmonella enterica serovar Typhimurium effectors SopB, SopE, SopE2 and SipA disrupt tight junction structure and function

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Disruption of tight junctions and induction of proinflammatory cytokine responses in colonic epithelial cells by Campylobacter jejuni

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Identification of tight junction modulating lipids

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Intestinal barrier dysfunction by enteropathogenic Escherichia coli is mediated by two effector molecules and a bacterial surface protein.

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Tight junctional structure and permeability of porcine jejunum after enterotoxic Escherichia coli infection.

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The secreted autotransporter toxin, Sat, functions as a virulence factor in Afa/Dr diffusely adhering Escherichia coli by promoting lesions in tight junction of polarized epithelial cells.

7 January 2021

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7 January 2021

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Microbes and microbial toxins: paradigms for microbial-mucosal interactions. VII. Enteropathogenic Escherichia coli: physiological alterations from an extracellular position

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Heat-labile enterotoxin promotes Escherichia coli adherence to intestinal epithelial cells.

7 January 2021

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Enterotoxigenic Escherichia coli modulates host intestinal cell membrane asymmetry and metabolic activity

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Porcine intestinal epithelial cell lines as a new in vitro model for studying adherence and pathogenesis of enterotoxigenic Escherichia coli

7 January 2021

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ClC-2 is required for rapid restoration of epithelial tight junctions in ischemic-injured murine jejunum

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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Enteropathogenic E. coli disrupts tight junction barrier function and structure in vivo.

7 January 2021

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ZO-1 and ZO-2 independently determine where claudins are polymerized in tight-junction strand formation.

7 January 2021

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The density of small tight junction pores varies among cell types and is increased by expression of claudin-2.

7 January 2021

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Adhesion in vitro and in vivo associated with an adhesive antigen (F41) produced by a K99 mutant of the reference strain Escherichia coli B41

7 January 2021

1 reference

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inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1016/J.VETMIC.2009.08.020

1 reference

14 September 2017

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