(Q39849267)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:19455703%20AND%20SRC:MED&resulttype=core&format=json

28 January 2020

title

Breaks invisible to the DNA damage response machinery accumulate in ATM-deficient cells (English)

1 reference

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28 January 2020

0 references

2

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28 January 2020

Laura Tusell

4

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28 January 2020

Anna Genesca

5

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28 January 2020

author name string

Marta Martín

1

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28 January 2020

George Iliakis

3

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28 January 2020

language of work or name

1 reference

1 September 2009

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Genes, Chromosomes and Cancer

28 January 2020

published in

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28 January 2020

volume

48

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28 January 2020

issue

9

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28 January 2020

page(s)

745-759

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Aberrant V(D)J recombination in ataxia telangiectasia mutated-deficient lymphocytes is dependent on nonhomologous DNA end joining.

28 January 2020

cites work

1 reference

Artemis links ATM to double strand break rejoining

21 January 2018

1 reference

An imperfect G2M checkpoint contributes to chromosome instability following irradiation of S and G2 phase cells

21 January 2018

1 reference

Cell biology. Guiding ATM to broken DNA.

21 January 2018

1 reference

7 January 2021

Fibroblasts from ataxia telangiectasia (AT) and AT heterozygotes show an enhanced level of residual DNA double-strand breaks after low dose-rate gamma-irradiation as assayed by pulsed field gel electrophoresis

1 reference

7 January 2021

ATM stabilizes DNA double-strand-break complexes during V(D)J recombination.

1 reference

7 January 2021

ATM phosphorylates histone H2AX in response to DNA double-strand breaks

1 reference

7 January 2021

ATM prevents the persistence and propagation of chromosome breaks in lymphocytes

1 reference

7 January 2021

Histone H2AX phosphorylation is dispensable for the initial recognition of DNA breaks

1 reference

7 January 2021

On the nature of a defect in cells from individuals with ataxia-telangiectasia

1 reference

7 January 2021

Hypersensitivity of ataxia telangiectasia fibroblasts to ionizing radiation is associated with a repair deficiency of DNA double-strand breaks

1 reference

7 January 2021

H2AX prevents DNA breaks from progressing to chromosome breaks and translocations

1 reference

7 January 2021

ATM signaling facilitates repair of DNA double-strand breaks associated with heterochromatin

1 reference

7 January 2021

Phosphorylation of histone H2AX at M phase in human cells without DNA damage response

1 reference

7 January 2021

Focus formation of DNA repair proteins in normal and repair-deficient cells irradiated with high-LET ions

1 reference

7 January 2021

Levels of gamma-H2AX Foci after low-dose-rate irradiation reveal a DNA DSB rejoining defect in cells from human ATM heterozygotes in two at families and in another apparently normal individual.

1 reference

7 January 2021

Radiation-Induced Chromosome Aberrations in Ataxia Telangiectasia Cells: High Frequency of Deletions and Misrejoining Detected by FluorescenceIn SituHybridization

1 reference

7 January 2021

A double-strand break repair defect in ATM-deficient cells contributes to radiosensitivity

1 reference

7 January 2021

DNA damage-induced activation of ATM and ATM-dependent signaling pathways

1 reference

7 January 2021

ATM and the Mre11 complex combine to recognize and signal DNA double-strand breaks

1 reference

7 January 2021

Activation and regulation of ATM kinase activity in response to DNA double-strand breaks

1 reference

7 January 2021

DNA amplification by breakage/fusion/bridge cycles initiated by spontaneous telomere loss in a human cancer cell line.

1 reference

7 January 2021

Chromosome instability as a result of double-strand breaks near telomeres in mouse embryonic stem cells

1 reference

7 January 2021

Expression of phosphorylated histone H2AX in cultured cell lines following exposure to X-rays.

1 reference

7 January 2021

Radiation-induced chromosome breaks in ataxia-telangiectasia cells remain open.

1 reference

7 January 2021

Postreplicative Joining of DNA Double-Strand Breaks Causes Genomic Instability in DNA-PKcs–Deficient Mouse Embryonic Fibroblasts

1 reference

7 January 2021

ATM-dependent DNA damage-independent mitotic phosphorylation of H2AX in normally growing mammalian cells.

1 reference

7 January 2021

DNA-PKcs dependence of Artemis endonucleolytic activity, differences between hairpins and 5' or 3' overhangs

1 reference

7 January 2021

A critical role for histone H2AX in recruitment of repair factors to nuclear foci after DNA damage

1 reference

7 January 2021

A pathway of double-strand break rejoining dependent upon ATM, Artemis, and proteins locating to gamma-H2AX foci.

1 reference

7 January 2021

DNA double-stranded breaks induce histone H2AX phosphorylation on serine 139

1 reference

7 January 2021

Megabase chromatin domains involved in DNA double-strand breaks in vivo

1 reference

7 January 2021

Evidence for a lack of DNA double-strand break repair in human cells exposed to very low x-ray doses

1 reference

7 January 2021

Quantitative detection of (125)IdU-induced DNA double-strand breaks with gamma-H2AX antibody

1 reference

7 January 2021

The ATM-mediated DNA-damage response: taking shape.

1 reference

7 January 2021

ATM and DNA-PK function redundantly to phosphorylate H2AX after exposure to ionizing radiation

1 reference

7 January 2021

Accumulation of Checkpoint Protein 53BP1 at DNA Breaks Involves Its Binding to Phosphorylated Histone H2AX

1 reference

7 January 2021

Mre11-Rad50-Nbs1 is a keystone complex connecting DNA repair machinery, double-strand break signaling, and the chromatin template.

1 reference

7 January 2021