(Q39943570)

15 September 2017

title

SOCS2: inhibitor of JAK2V617F-mediated signal transduction (English)

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15 September 2017

10

H G Drexler

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15 September 2017

H Quentmeier

1

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15 September 2017

R Geffers

2

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15 September 2017

E Jost

3

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15 September 2017

R A F Macleod

4

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15 September 2017

S Nagel

5

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15 September 2017

S Röhrs

6

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15 September 2017

J Romani

7

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15 September 2017

M Scherr

8

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15 September 2017

M Zaborski

9

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15 September 2017

publication date

4 September 2008

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15 September 2017

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15 September 2017

volume

22

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15 September 2017

issue

12

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15 September 2017

page(s)

2169-2175

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https://scigraph.springernature.com/pub.10.1038/leu.2008.226

15 September 2017

exact match

0 references

cites work

Suppressors of cytokine signalling: SOCS.

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A family of cytokine-inducible inhibitors of signalling

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A new protein containing an SH2 domain that inhibits JAK kinases

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https://api.crossref.org/works/10.1038%2FLEU.2008.226

7 January 2021

Structure and function of a new STAT-induced STAT inhibitor

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https://api.crossref.org/works/10.1038%2FLEU.2008.226

7 January 2021

Differential hypermethylation of SOCS genes in ovarian and breast carcinomas.

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https://api.crossref.org/works/10.1038%2FLEU.2008.226

7 January 2021

DNA methylation changes in multiple myeloma

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https://api.crossref.org/works/10.1038%2FLEU.2008.226

7 January 2021

Epigenetic inactivation of tumor suppressor genes in serum of patients with cutaneous melanoma

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https://api.crossref.org/works/10.1038%2FLEU.2008.226

7 January 2021

Epigenetic modification of SOCS-1 differentially regulates STAT3 activation in response to interleukin-6 receptor and epidermal growth factor receptor signaling through JAK and/or MEK in head and neck squamous cell carcinomas

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2008.226

7 January 2021

Hypermethylation of the suppressor of cytokine signalling-1 (SOCS-1) in myelodysplastic syndrome.

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2008.226

7 January 2021

Epigenetic alterations complement mutation of JAK2 tyrosine kinase in patients with BCR/ABL-negative myeloproliferative disorders.

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2008.226

7 January 2021

Rearrangements involving 12q in myeloproliferative disorders: possible role of HMGA2 and SOCS2 genes

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https://api.crossref.org/works/10.1038%2FLEU.2008.226

7 January 2021

Expression of a homodimeric type I cytokine receptor is required for JAK2V617F-mediated transformation

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2008.226

7 January 2021

Dimerization by a cytokine receptor is necessary for constitutive activation of JAK2V617F.

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2008.226

7 January 2021

JAK2 V617F tyrosine kinase mutation in cell lines derived from myeloproliferative disorders

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7 January 2021

New acute myeloid leukemia-derived cell line: MUTZ-8 with 5q-.

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https://api.crossref.org/works/10.1038%2FLEU.2008.226

7 January 2021

Derivation of a new hematopoietic cell line with endothelial features from a patient with transformed myeloproliferative syndrome: a case report

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https://api.crossref.org/works/10.1038%2FLEU.2008.226

7 January 2021

Cloning of human thymic stromal lymphopoietin (TSLP) and signaling mechanisms leading to proliferation

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2008.226

7 January 2021

Cytogenetic harvesting of commonly used tumor cell lines

1 reference

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7 January 2021

Lentiviral gene transfer into peripheral blood-derived CD34+ NOD/SCID-repopulating cells

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2008.226

7 January 2021

Activating mutation in the tyrosine kinase JAK2 in polycythemia vera, essential thrombocythemia, and myeloid metaplasia with myelofibrosis

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2008.226

7 January 2021

Overexpression of SOCS-2 and SOCS-3 genes reverses erythroid overgrowth and IGF-I hypersensitivity of primary polycythemia vera (PV) cells

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2008.226

7 January 2021

SOCS2 can enhance interleukin-2 (IL-2) and IL-3 signaling by accelerating SOCS3 degradation

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2008.226

7 January 2021

The myeloproliferative disorder associated JAK2 V617F mutant escapes negative regulation by suppressor of cytokine signaling 3

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2008.226

7 January 2021

Differential roles of SOCS family members in EpoR signal transduction

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2008.226

7 January 2021

Epigenetic regulation of normal and malignant hematopoiesis

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2008.226

7 January 2021

Identifiers

DOI

10.1038/LEU.2008.226

1 reference

15 September 2017

1 reference