(Q39973735)

16 September 2017

title

Histone H2AX is a critical factor for cellular protection against DNA alkylating agents (English)

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16 September 2017

J A Meador

1

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16 September 2017

M Zhao

2

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16 September 2017

Y Su

3

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16 September 2017

G Narayan

4

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16 September 2017

C R Geard

5

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16 September 2017

A S Balajee

6

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16 September 2017

language of work or name

English

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publication date

9 June 2008

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16 September 2017

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16 September 2017

volume

27

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16 September 2017

issue

43

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16 September 2017

page(s)

5662-5671

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Distribution of methyl and ethyl adducts following alkylation with monofunctional alkylating agents

16 September 2017

cites work

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

Chromatin-bound PCNA complex formation triggered by DNA damage occurs independent of the ATM gene product in human cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

Replication protein A and gamma-H2AX foci assembly is triggered by cellular response to DNA double-strand breaks

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

Increased ionizing radiation sensitivity and genomic instability in the absence of histone H2AX

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

Histone H2AX: a dosage-dependent suppressor of oncogenic translocations and tumors

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

N-methyl-N'-nitro-N-nitrosoguanidine activates cell-cycle arrest through distinct mechanisms activated in a dose-dependent manner

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

PARP-1, a determinant of cell survival in response to DNA damage

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

H2AX haploinsufficiency modifies genomic stability and tumor susceptibility

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

Histone H2AX phosphorylation is dispensable for the initial recognition of DNA breaks

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

Genomic instability in mice lacking histone H2AX

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

Tubulozole-induced G2/M cell cycle arrest in human colon cancer cells through formation of microtubule polymerization mediated by ERK1/2 and Chk1 kinase activation.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

DNA-independent PARP-1 activation by phosphorylated ERK2 increases Elk1 activity: a link to histone acetylation

31 March 2020

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https://api.crossref.org/works/10.1038%2FONC.2008.187

A DNA damage checkpoint response in telomere-initiated senescence

31 March 2020

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https://api.crossref.org/works/10.1038%2FONC.2008.187

A role for Saccharomyces cerevisiae histone H2A in DNA repair.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

PARP-1-induced cell death through inhibition of the MEK/ERK pathway in MNNG-treated HeLa cells

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

DNA damage-induced G2-M checkpoint activation by histone H2AX and 53BP1

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

H2AX: the histone guardian of the genome

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

Phosphorylation of histone H2AX and activation of Mre11, Rad50, and Nbs1 in response to replication-dependent DNA double-strand breaks induced by mammalian DNA topoisomerase I cleavage complexes.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

Genome maintenance mechanisms for preventing cancer

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

Poly(ADP-ribose) polymerase activity prevents signaling pathways for cell cycle arrest after DNA methylating agent exposure.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

DNA and its associated processes as targets for cancer therapy

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

Phosphorylation of histone H2AX at M phase in human cells without DNA damage response

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

Human mismatch repair, drug-induced DNA damage, and secondary cancer.

31 March 2020

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https://api.crossref.org/works/10.1038%2FONC.2008.187

Direct phosphorylation and regulation of poly(ADP-ribose) polymerase-1 by extracellular signal-regulated kinases 1/2.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

Efficiency of repair of an abasic site within DNA clustered damage sites by mammalian cell nuclear extracts

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

ATM-dependent DNA damage-independent mitotic phosphorylation of H2AX in normally growing mammalian cells.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

Actinomycin D induces histone gamma-H2AX foci and complex formation of gamma-H2AX with Ku70 and nuclear DNA helicase II.

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

Yeast histone 2A serine 129 is essential for the efficient repair of checkpoint-blind DNA damage

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

Megabase chromatin domains involved in DNA double-strand breaks in vivo

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

Initiation of DNA fragmentation during apoptosis induces phosphorylation of H2AX histone at serine 139

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

DNA double-stranded breaks induce histone H2AX phosphorylation on serine 139

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

Poly(ADP-ribose) polymerase-1 cleavage during apoptosis: an update

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

ATM and DNA-PK function redundantly to phosphorylate H2AX after exposure to ionizing radiation

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

DNA damage foci at dysfunctional telomeres

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

The stress-activated protein kinase pathways

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

Complex H2AX phosphorylation patterns by multiple kinases including ATM and DNA-PK in human cells exposed to ionizing radiation and treated with kinase inhibitors

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

Histone H2AX is phosphorylated in an ATR-dependent manner in response to replicational stress

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

Two molecularly distinct G(2)/M checkpoints are induced by ionizing irradiation

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

Irradiation-induced G2/M checkpoint response requires ERK1/2 activation

31 March 2020

1 reference

https://api.crossref.org/works/10.1038%2FONC.2008.187

31 March 2020

Identifiers

DOI

10.1038/ONC.2008.187

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Dimensions Publication ID

16 September 2017

1024245434

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1 reference