(Q39996387)

English

Telmisartan, an angiotensin II type 1 receptor blocker, inhibits advanced glycation end-product (AGE)-elicited hepatic insulin resistance via peroxisome proliferator-activated receptor-gamma activation

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

Telmisartan, an angiotensin II type 1 receptor blocker, inhibits advanced glycation end-product (AGE)-elicited hepatic insulin resistance via peroxisome proliferator-activated receptor-gamma activation (English)

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

insulin resistance

1 reference

based on heuristic

inferred from title

author name string

T Yoshida

1

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

S Yamagishi

2

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

T Matsui

3

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

K Nakamura

4

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

T Ueno

5

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

M Takeuchi

6

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

M Sata

7

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

publication date

1 March 2008

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

Journal of International Medical Research

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Europe PubMed Central

PubMed publication ID

16 September 2017

36

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Europe PubMed Central

PubMed publication ID

16 September 2017

2

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

237-243

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

TAGE (toxic AGEs) theory in diabetic complications

1 reference

https://api.crossref.org/works/10.1177%2F147323000803600204

21 January 2018

Recent progress in advanced glycation and diabetic vascular disease: role of advanced glycation end product receptors

1 reference

https://api.crossref.org/works/10.1177%2F147323000803600204

21 January 2018

Diabetic vascular complications: pathophysiology, biochemical basis and potential therapeutic strategy

1 reference

https://api.crossref.org/works/10.1177%2F147323000803600204

21 January 2018

Advanced glycation end products attenuate cellular insulin sensitivity by increasing the generation of intracellular reactive oxygen species in adipocytes

1 reference

https://api.crossref.org/works/10.1177%2F147323000803600204

21 January 2018

Human glycated albumin affects glucose metabolism in L6 skeletal muscle cells by impairing insulin-induced insulin receptor substrate (IRS) signaling through a protein kinase C alpha-mediated mechanism

1 reference

https://api.crossref.org/works/10.1177%2F147323000803600204

21 January 2018

Improved insulin sensitivity is associated with restricted intake of dietary glycoxidation products in the db/db mouse.

1 reference

https://api.crossref.org/works/10.1177%2F147323000803600204

21 January 2018

Advanced-glycation end products in insulin-resistant states

1 reference

https://api.crossref.org/works/10.1177%2F147323000803600204

21 January 2018

Role of the renin-angiotensin- aldosterone system in the metabolic syndrome

1 reference

https://api.crossref.org/works/10.1177%2F147323000803600204

21 January 2018

Telmisartan inhibits AGE-induced C-reactive protein production through downregulation of the receptor for AGE via peroxisome proliferator-activated receptor-gamma activation

1 reference

https://api.crossref.org/works/10.1177%2F147323000803600204

21 January 2018

Potential utility of telmisartan, an angiotensin II type 1 receptor blocker with peroxisome proliferator-activated receptor-gamma (PPAR-gamma)-modulating activity for the treatment of cardiometabolic disorders

1 reference

https://api.crossref.org/works/10.1177%2F147323000803600204

21 January 2018

Telmisartan inhibits expression of a receptor for advanced glycation end products (RAGE) in angiotensin-II-exposed endothelial cells and decreases serum levels of soluble RAGE in patients with essential hypertension

1 reference

https://api.crossref.org/works/10.1177%2F147323000803600204

21 January 2018

Angiotensin II augments advanced glycation end product-induced pericyte apoptosis through RAGE overexpression

1 reference

https://api.crossref.org/works/10.1177%2F147323000803600204

21 January 2018

Hyperglycemia potentiates collagen-induced platelet activation through mitochondrial superoxide overproduction

1 reference

https://api.crossref.org/works/10.1177%2F147323000803600204

21 January 2018

Interleukin-6 induces cellular insulin resistance in hepatocytes

1 reference

https://api.crossref.org/works/10.1177%2F147323000803600204

21 January 2018

The c-Jun NH(2)-terminal kinase promotes insulin resistance during association with insulin receptor substrate-1 and phosphorylation of Ser(307)

1 reference

https://api.crossref.org/works/10.1177%2F147323000803600204

21 January 2018

Myosin motor Myo1c and its receptor NEMO/IKK-gamma promote TNF-alpha-induced serine307 phosphorylation of IRS-1.

1 reference

https://api.crossref.org/works/10.1177%2F147323000803600204

21 January 2018

Pigment epithelium-derived factor (PEDF) ameliorates advanced glycation end product (AGE)-induced hepatic insulin resistance in vitro by suppressing Rac-1 activation

1 reference

https://api.crossref.org/works/10.1177%2F147323000803600204

21 January 2018

Hypoglycemic action of thiazolidinediones/peroxisome proliferator-activated receptor gamma by inhibition of the c-Jun NH2-terminal kinase pathway

1 reference

https://api.crossref.org/works/10.1177%2F147323000803600204

21 January 2018

Phosphorylation of Ser307 in insulin receptor substrate-1 blocks interactions with the insulin receptor and inhibits insulin action

1 reference

https://api.crossref.org/works/10.1177%2F147323000803600204

21 January 2018

Insulin resistance due to phosphorylation of insulin receptor substrate-1 at serine 302.

1 reference

https://api.crossref.org/works/10.1177%2F147323000803600204

21 January 2018

Identifiers

10.1177/147323000803600204

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

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