(Q40016236)

16 September 2017

Analyses of a novel SCN5A mutation (C1850S): conduction vs. repolarization disorder hypotheses in the Brugada syndrome (English)

1 reference

16 September 2017

1 reference

Jan P Kucera

8

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Hugues Abriel

object named as

Hugues Abriel

7

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Thomas Jespersen

object named as

Thomas Jespersen

2

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author name string

Séverine Petitprez

1

1 reference

16 September 2017

Etienne Pruvot

3

1 reference

16 September 2017

Dagmar I Keller

4

1 reference

16 September 2017

Cora Corbaz

5

1 reference

16 September 2017

Jürg Schläpfer

6

1 reference

16 September 2017

5 February 2008

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16 September 2017

Cardiovascular Research

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16 September 2017

78

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16 September 2017

494-504

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16 September 2017

3

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Pathophysiological mechanisms of Brugada syndrome: depolarization disorder, repolarization disorder, or more?

16 September 2017

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVN023

Proposed diagnostic criteria for the Brugada syndrome: consensus report

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVN023

Brugada syndrome and fever: genetic and molecular characterization of patients carrying SCN5A mutations.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVN023

Brugada syndrome: a decade of progress

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVN023

Cardiac voltage-gated sodium channel Nav1.5 is regulated by Nedd4-2 mediated ubiquitination

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVN023

A dynamic model of the cardiac ventricular action potential. I. Simulations of ionic currents and concentration changes

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVN023

Action potential and contractility changes in [Na(+)](i) overloaded cardiac myocytes: a simulation study

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVN023

Ionic mechanisms responsible for the electrocardiographic phenotype of the Brugada syndrome are temperature dependent

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVN023

Enhanced Na(+) channel intermediate inactivation in Brugada syndrome

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVN023

Cellular basis for the Brugada syndrome and other mechanisms of arrhythmogenesis associated with ST-segment elevation

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVN023

The hyperkalemic Brugada sign

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVN023

Brugada syndrome with ventricular tachycardia and fibrillation related to hypokalemia

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVN023

Quantitative assessment of ST segment elevation in Brugada patients

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVN023

Right ventricular fibrosis and conduction delay in a patient with clinical signs of Brugada syndrome: a combined electrophysiological, genetic, histopathologic, and computational study.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVN023

Cardiac histological substrate in patients with clinical phenotype of Brugada syndrome

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVN023

Basic Mechanisms of Cardiac Impulse Propagation and Associated Arrhythmias

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVN023

Loss-of-function mutations in the cardiac calcium channel underlie a new clinical entity characterized by ST-segment elevation, short QT intervals, and sudden cardiac death

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVN023

Dynamic changes of cardiac conduction during rapid pacing.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVN023

21 January 2018

Identifiers

DOI

10.1093/CVR/CVN023

1 reference

16 September 2017

1 reference