(Q40054867)

English

NF-kappaB inhibition reveals differential mechanisms of TNF versus TRAIL-induced apoptosis upstream or at the level of caspase-8 activation independent of cIAP2.

scientific article published on 8 November 2007

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scholarly article

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9 January 2020

NF-kappaB inhibition reveals differential mechanisms of TNF versus TRAIL-induced apoptosis upstream or at the level of caspase-8 activation independent of cIAP2 (English)

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9 January 2020

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9 January 2020

Martin R. Sprick

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9 January 2020

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9 January 2020

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Philip Diessenbacher

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9 January 2020

Andreas Kerstan

4

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9 January 2020

Peter Geserick

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9 January 2020

Tina Wachter

7

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9 January 2020

Manfred Neumann

8

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9 January 2020

Henning Walczak

9

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9 January 2020

Martin Leverkus

11

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8 November 2007

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9 January 2020

Journal of Investigative Dermatology

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9 January 2020

128

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9 January 2020

1134-1147

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9 January 2020

5

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9 January 2020

https://scigraph.springernature.com/pub.10.1038/sj.jid.5701141

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Abnormal NF-kappaB signaling pathway with enhanced susceptibility to apoptosis in immortalized keratinocytes

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Posttranscriptional downregulation of c-IAP2 by the ubiquitin protein ligase c-IAP1 in vivo

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Activation of NF-κB via the IκB Kinase Complex Is Both Essential and Sufficient for Proinflammatory Gene Expression in Primary Endothelial Cells

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The human anti-apoptotic proteins cIAP1 and cIAP2 bind but do not inhibit caspases

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NF-kappaB at the crossroads of life and death

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NF-kappaB inducers upregulate cFLIP, a cycloheximide-sensitive inhibitor of death receptor signaling

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TRAIL-induced apoptosis and gene induction in HaCaT keratinocytes: differential contribution of TRAIL receptors 1 and 2.

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Proteasome inhibition results in TRAIL sensitization of primary keratinocytes by removing the resistance-mediating block of effector caspase maturation

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Fas/Fas ligand interaction contributes to UV-induced apoptosis in human keratinocytes

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Aberrant NF-kappaB activity in HaCaT cells alters their response to UVB signaling

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Signal-specific and phosphorylation-dependent RelB degradation: a potential mechanism of NF-kappaB control

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NF-kappaB signals induce the expression of c-FLIP.

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A rapid and simple method for measuring thymocyte apoptosis by propidium iodide staining and flow cytometry

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Regulation of death receptor expression and TRAIL/Apo2L-induced apoptosis by NF-kappaB

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Ultraviolet-B-Induced Apoptosis of Keratinocytes: Evidence for Partial Involvement of Tumor Necrosis Factor-α in the Formation of Sunburn Cells

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Identifiers

10.1038/SJ.JID.5701141

1 reference

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9 January 2020

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