(Q40131977)

17 September 2017

title

Arsenic trioxide inhibits growth of As4.1 juxtaglomerular cells via cell cycle arrest and caspase-independent apoptosis (English)

1 reference

17 September 2017

Yong Hwan Han

1

1 reference

17 September 2017

Sung Zoo Kim

series ordinal

2

1 reference

17 September 2017

Suhn Hee Kim

series ordinal

3

1 reference

17 September 2017

Woo Hyun Park

series ordinal

4

1 reference

17 September 2017

publication date

16 May 2007

1 reference

American Journal of Physiology - Renal Physiology

17 September 2017

published in

1 reference

17 September 2017

volume

293

1 reference

17 September 2017

issue

2

1 reference

17 September 2017

page(s)

F511-20

1 reference

The role of ROS and RNS in regulating life and death of blood monocytes

17 September 2017

cites work

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

Involvement of caspase-3, lipid peroxidation and TNF-alpha in causing apoptosis of macrophages by coordinately expressed Salmonella phenotype under stress conditions.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

Quercetin inhibition of ROS-dependent and -independent apoptosis in rat glioma C6 cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

Role of NADPH oxidase in arsenic-induced reactive oxygen species formation and cytotoxicity in myeloid leukemia cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

Caspases: the executioners of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

The tyrphostin adaphostin interacts synergistically with proteasome inhibitors to induce apoptosis in human leukemia cells through a reactive oxygen species (ROS)-dependent mechanism

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

Significance of ROS in oxygen sensing in cell systems with sensitivity to physiological hypoxia

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

Bcl-x(L) prevents the initial decrease in mitochondrial membrane potential and subsequent reactive oxygen species production during tumor necrosis factor alpha-induced apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

Involvement of mitochondrial aggregation in arsenic trioxide (As2O3)-induced apoptosis in human glioblastoma cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

Arsenic trioxide represses constitutive activation of NF-kappaB and COX-2 expression in human acute myeloid leukemia, HL-60.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

Arsenic trioxide inhibits the growth of A498 renal cell carcinoma cells via cell cycle arrest or apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

Essential role of the mitochondrial apoptosis-inducing factor in programmed cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

Caspase-independent cell death by arsenic trioxide in human cervical cancer cells: reactive oxygen species-mediated poly(ADP-ribose) polymerase-1 activation signals apoptosis-inducing factor release from mitochondria.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

Involvement of CD95-independent caspase 8 activation in arsenic trioxide-induced apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

Role of oxidative stress in the apoptosis of hepatocellular carcinoma induced by combination of arsenic trioxide and ascorbic acid

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

Arsenic trioxide produces polymerization of microtubules and mitotic arrest before apoptosis in human tumor cell lines

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

Arsenic trioxide-induced apoptosis in myeloma cells: p53-dependent G1 or G2/M cell cycle arrest, activation of caspase-8 or caspase-9, and synergy with APO2/TRAIL.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

Flow cytometric analysis of cell cycle-dependent changes in cell thiol level by combining a new laser dye with Hoechst 33342.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

De novo synthesis of glutathione is required for both entry into and progression through the cell cycle

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

Glutathione depletion causes cell growth inhibition and enhanced apoptosis in pancreatic cancer cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

Arsenic trioxide induces regulated, death receptor-independent cell death through a Bcl-2-controlled pathway

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

Arsenic trioxide triggers a regulated form of caspase-independent necrotic cell death via the mitochondrial death pathway

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

Arsenic trioxide as an inducer of apoptosis and loss of PML/RAR alpha protein in acute promyelocytic leukemia cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

Role of reactive oxygen species (ROS) in apoptosis induction

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

Complete remission after treatment of acute promyelocytic leukemia with arsenic trioxide

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

TRAIL sensitisation by arsenic trioxide is caspase-8 dependent and involves modulation of death receptor components and Akt.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

The role of mitochondrial factors in apoptosis: a Russian roulette with more than one bullet.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

Bleomycin initiates apoptosis of lung epithelial cells by ROS but not by Fas/FasL pathway

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

Reactive oxygen species: roles in blood pressure and kidney function

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

Redox regulation of the afferent arteriole and tubuloglomerular feedback

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

Prevention of apoptosis by Bcl-2: release of cytochrome c from mitochondria blocked

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

The induction of apoptosis and cell cycle arrest by arsenic trioxide in lymphoid neoplasms

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

Mitochondrial ROS-induced ROS release: an update and review

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00385.2006

10.1152/AJPRENAL.00385.2006

21 January 2018

Identifiers

DOI

1 reference

17 September 2017

1 reference