(Q40183668)

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Clostridium difficile toxin B causes apoptosis in epithelial cells by thrilling mitochondria. Involvement of ATP-sensitive mitochondrial potassium channels.

scientific article published on 12 January 2007

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24 December 2019

Clostridium difficile toxin B causes apoptosis in epithelial cells by thrilling mitochondria. Involvement of ATP-sensitive mitochondrial potassium channels (English)

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24 December 2019

Clostridium difficile

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Carla Fiorentini

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24 December 2019

Loredana Falzano

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24 December 2019

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24 December 2019

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Claudio Frank

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24 December 2019

Michel R. Popoff

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Michel R Popoff

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24 December 2019

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Paola Matarrese

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24 December 2019

Lucrezia Gambardella

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24 December 2019

Blandine Geny

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12 January 2007

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24 December 2019

Journal of Biological Chemistry

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24 December 2019

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24 December 2019

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24 December 2019

9029-9041

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24 December 2019

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The biochemistry of apoptosis

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The N-terminal 34 kDa fragment of Helicobacter pylori vacuolating cytotoxin targets mitochondria and induces cytochrome c release

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VDAC and the bacterial porin PorB of Neisseria gonorrhoeae share mitochondrial import pathways

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Staphylococcus aureus alpha-toxin induces apoptosis in peripheral blood mononuclear cells: role of endogenous tumour necrosis factor-alpha and the mitochondrial death pathway

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Large clostridial cytotoxins

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Lethal toxin fromClostridium sordelliiinduces apoptotic cell death by disruption of mitochondrial homeostasis in HL-60 cells

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Clostridium difficile toxin B activates dual caspase-dependent and caspase-independent apoptosis in intoxicated cells

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Large clostridial cytotoxins--a family of glycosyltransferases modifying small GTP-binding proteins

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Glucosylation of Rho proteins by Clostridium difficile toxin B.

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Rho GTPases in cell biology

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Role of mitochondria in the pheromone- and amiodarone-induced programmed death of yeast

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Purification of two high molecular weight toxins of Clostridium difficile which are antigenically related

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Measurement of membrane permeability and permeability transition of mitochondria

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Tissue transglutaminase is a multifunctional BH3-only protein

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Mitochondria hyperpolarization is an early event in oxidized low-density lipoprotein-induced apoptosis in Caco-2 intestinal cells

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How to clean the dirtiest place in the cell: cationic antioxidants as intramitochondrial ROS scavengers

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Cellular uptake of Clostridium difficile toxin B. Translocation of the N-terminal catalytic domain into the cytosol of eukaryotic cells

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Biochemical and genetic analysis of the mitochondrial response of yeast to BAX and BCL-X(L)

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Mitochondrial control of cell death

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Mitochondrial membrane hyperpolarization hijacks activated T lymphocytes toward the apoptotic-prone phenotype: homeostatic mechanisms of HIV protease inhibitors

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ATP-sensitive K+ channel in the mitochondrial inner membrane

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Mitochondrial K(ATP) channel as an end effector of cardioprotection during late preconditioning: triggering role of nitric oxide

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Potassium channel openers depolarize hippocampal mitochondria

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Sarcolemmal versus mitochondrial ATP-sensitive K+ channels and myocardial preconditioning

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Mitochondrial ATP-sensitive potassium channel activation protects cerebellar granule neurons from apoptosis induced by oxidative stress

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Mitochondrial ATP-dependent potassium channels. Viable candidate effectors of ischemic preconditioning.

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Involvement of mitochondrial K+ release and cellular efflux in ischemic and apoptotic neuronal death

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Identification of Clostridium difficile toxin B cardiotoxicity using a zebrafish embryo model of intoxication

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Mitochondrial ATP-sensitive potassium channels attenuate matrix Ca(2+) overload during simulated ischemia and reperfusion: possible mechanism of cardioprotection

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10.1074/JBC.M607614200

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Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:17220295%20AND%20SRC:MED&resulttype=core&format=json

24 December 2019

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:17220295%20AND%20SRC:MED&resulttype=core&format=json

24 December 2019

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