(Q40184279)

18 September 2017

title

Copper ions disrupt dopamine metabolism via inhibition of V-H+-ATPase: a possible contributing factor to neurotoxicity (English)

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18 September 2017

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D Shyamali Wimalasena

series ordinal

1

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18 September 2017

Thomas J Wiese

series ordinal

2

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18 September 2017

Kandatege Wimalasena

series ordinal

3

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18 September 2017

language of work or name

English

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publication date

18 January 2007

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Journal of Neurochemistry

18 September 2017

published in

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18 September 2017

volume

101

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18 September 2017

issue

2

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18 September 2017

page(s)

313-326

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Neurodegenerative diseases and oxidative stress

18 September 2017

cites work

1 reference

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Copper depletion down-regulates expression of the Alzheimer's disease amyloid-beta precursor protein gene

21 January 2018

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A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

21 January 2018

1 reference

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Copper and zinc cause delivery of the prion protein from the plasma membrane to a subset of early endosomes and the Golgi

21 January 2018

1 reference

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Copper, iron, and zinc imbalances in severely degenerated brain regions in Alzheimer's disease: possible relation to oxidative stress.

21 January 2018

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Cupric-amyloid beta peptide complex stimulates oxidation of ascorbate and generation of hydroxyl radical

21 January 2018

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Copper toxicity, oxidative stress, and antioxidant nutrients

21 January 2018

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The loading of neurotransmitters into synaptic vesicles

21 January 2018

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A modified colorimetric method for the determination of orthophosphate in the presence of high ATP concentrations.

21 January 2018

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Cellular copper transport and metabolism

21 January 2018

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The vesicular monoamine transporter: from chromaffin granule to brain

21 January 2018

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The beta A4 amyloid precursor protein binding to copper.

21 January 2018

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Methods for studying synaptosomal copper release

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Nerve endings from rat brain tissue release copper upon depolarization. A possible role in regulating neuronal excitability

21 January 2018

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Electrogenic epinephrine transport in chromaffin granule ghosts.

21 January 2018

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Copper, iron and zinc in Alzheimer's disease senile plaques.

21 January 2018

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Role of hydrogen peroxide in the aetiology of Alzheimer's disease: implications for treatment.

21 January 2018

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The amyloid precursor protein of Alzheimer's disease in the reduction of copper(II) to copper(I)

21 January 2018

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Bioenergetics of secretory vesicles

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Mechanism of ascorbic acid regeneration mediated by cytochrome b561.

21 January 2018

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Metallochaperones, an intracellular shuttle service for metal ions

21 January 2018

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Dopamine efflux by MPTP and hydroxyl radical generation

21 January 2018

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Copper(II)-induced self-oligomerization of alpha-synuclein.

21 January 2018

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Transport mechanisms in acetylcholine and monoamine storage

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Adventiously-bound redox active iron and copper are at the center of oxidative damage in Alzheimer disease

21 January 2018

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Undetectable intracellular free copper: the requirement of a copper chaperone for superoxide dismutase

21 January 2018

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Spectrophotometric measurements of transmembrane potential and pH gradients in chromaffin granules

21 January 2018

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A highly sensitive method for measurement of myosin ATPase activity by reversed-phase high-performance liquid chromatography

21 January 2018

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Prion protein selectively binds copper(II) ions

21 January 2018

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Copper in disorders with neurological symptoms: Alzheimer's, Menkes, and Wilson diseases.

21 January 2018

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Role of H+-ATPase-mediated acidification in sorting and release of the regulated secretory protein chromogranin A: evidence for a vesiculogenic function

21 January 2018

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Complete separation of adenine nucleotides for ATPase activity assay by ion-pair reversed-phase high-performance liquid chromatography

21 January 2018

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Copper binding to the prion protein: structural implications of four identical cooperative binding sites

21 January 2018

1 reference

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The reduction of membrane-bound dopamine beta-monooxygenase in resealed chromaffin granule ghosts. Is intragranular ascorbic acid a mediator for extragranular reducing equivalents?

21 January 2018

1 reference

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Kinetic evidence for channeling of dopamine between monoamine transporter and membranous dopamine-beta-monooxygenase in chromaffin granule ghosts

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1471-4159.2006.04362.X

10.1111/J.1471-4159.2006.04362.X

21 January 2018

Identifiers

DOI

1 reference

18 September 2017

1 reference