(Q40425153)

English

Use of a fluorescent phosphoprotein dye to characterize oxidative stress-induced signaling pathway components in macrophage and epithelial cultures exposed to diesel exhaust particle chemicals

scientific article published in June 2005

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

20 September 2017

Use of a fluorescent phosphoprotein dye to characterize oxidative stress-induced signaling pathway components in macrophage and epithelial cultures exposed to diesel exhaust particle chemicals (English)

1 reference

Europe PubMed Central

PubMed publication ID

20 September 2017

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author name string

Meiying Wang

1

1 reference

Europe PubMed Central

PubMed publication ID

20 September 2017

Gary Guishan Xiao

2

1 reference

Europe PubMed Central

PubMed publication ID

20 September 2017

Ning Li

3

1 reference

Europe PubMed Central

PubMed publication ID

20 September 2017

Yongming Xie

4

1 reference

Europe PubMed Central

PubMed publication ID

20 September 2017

Joseph A Loo

5

1 reference

Europe PubMed Central

PubMed publication ID

20 September 2017

Andre E Nel

6

1 reference

Europe PubMed Central

PubMed publication ID

20 September 2017

publication date

1 June 2005

1 reference

Europe PubMed Central

PubMed publication ID

20 September 2017

Electrophoresis

1 reference

Europe PubMed Central

PubMed publication ID

20 September 2017

26

1 reference

Europe PubMed Central

PubMed publication ID

20 September 2017

11

1 reference

Europe PubMed Central

PubMed publication ID

20 September 2017

2092-2108

1 reference

Europe PubMed Central

PubMed publication ID

20 September 2017

The role of a mitochondrial pathway in the induction of apoptosis by chemicals extracted from diesel exhaust particles

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Thiol antioxidants inhibit the adjuvant effects of aerosolized diesel exhaust particles in a murine model for ovalbumin sensitization

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Nrf2 is a key transcription factor that regulates antioxidant defense in macrophages and epithelial cells: protecting against the proinflammatory and oxidizing effects of diesel exhaust chemicals

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Comparison of the pro-oxidative and proinflammatory effects of organic diesel exhaust particle chemicals in bronchial epithelial cells and macrophages

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Regulation of TNF expression by multiple mitogen-activated protein kinase pathways

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Regulation of human airway epithelial cell IL-8 expression by MAP kinases

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Benzene-extracted components are important for the major activity of diesel exhaust particles: effect on interleukin-8 gene expression in human bronchial epithelial cells

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The level of intracellular glutathione is a key regulator for the induction of stress-activated signal transduction pathways including Jun N-terminal protein kinases and p38 kinase by alkylating agents

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ASK1 is required for sustained activations of JNK/p38 MAP kinases and apoptosis

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Organic compounds from diesel exhaust particles elicit a proinflammatory response in human airway epithelial cells and induce cytochrome p450 1A1 expression

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Diesel exhaust enhances allergic airway inflammation and hyperresponsiveness in mice

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Diesel exhaust inhalation enhances airway hyperresponsiveness in mice

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Diesel exhaust particles exert acute effects on airway inflammation and function in murine allergen provocation models

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Use of a stratified oxidative stress model to study the biological effects of ambient concentrated and diesel exhaust particulate matter

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Generation of reactive oxygen species during interaction of diesel exhaust particle components with NADPH-cytochrome P450 reductase and involvement of the bioactivation in the DNA damage

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Cytokine production by human airway epithelial cells after exposure to an air pollution particle is metal-dependent

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Metals associated with both the water-soluble and insoluble fractions of an ambient air pollution particle catalyze an oxidative stress

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The identification of polynuclear aromatic hydrocarbon (PAH) derivatives in mutagenic fractions of diesel particulate extracts

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Ultrafine particulate pollutants induce oxidative stress and mitochondrial damage

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Quinones and aromatic chemical compounds in particulate matter induce mitochondrial dysfunction: implications for ultrafine particle toxicity

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Use of Proteomics to Demonstrate a Hierarchical Oxidative Stress Response to Diesel Exhaust Particle Chemicals in a Macrophage Cell Line

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Direct ampholyte-free liquid-phase isoelectric peptide focusing: application to the human serum proteome

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Global quantitative phosphoprotein analysis using Multiplexed Proteomics technology

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In vivo nasal challenge with diesel exhaust particles enhances expression of the CC chemokines rantes, MIP-1alpha, and MCP-3 in humans

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Mass Spectrometric Sequencing of Proteins from Silver-Stained Polyacrylamide Gels

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Analysis of steady-state protein phosphorylation in mitochondria using a novel fluorescent phosphosensor dye.

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Dihydrodiol dehydrogenases and polycyclic aromatic hydrocarbon activation: generation of reactive and redox active o-quinones.

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Lung expression of cytochrome P450 1A1 as a possible biomarker of exposure to diesel exhaust particles.

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DNA binding of polycyclic aromatic hydrocarbons in a human bronchial epithelial cell line treated with diesel and gasoline particulate extracts and benzo[a]pyrene

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Transcriptional and posttranscriptional roles for p38 mitogen-activated protein kinase in upregulation of TNF-alpha expression by deoxynivalenol (vomitoxin).

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Diesel exhaust particles activate p38 MAP kinase to produce interleukin 8 and RANTES by human bronchial epithelial cells and N-acetylcysteine attenuates p38 MAP kinase activation.

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1 reference

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Identifiers

10.1002/ELPS.200410428

1 reference

Europe PubMed Central

PubMed publication ID

20 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

20 September 2017

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