(Q40473479)

English

Common fragile sites (CFS) and extremely large CFS genes are targets for human papillomavirus integrations and chromosome rearrangements in oropharyngeal squamous cell carcinoma.

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scholarly article

1 reference

Europe PubMed Central

20 September 2017

Common fragile sites (CFS) and extremely large CFS genes are targets for human papillomavirus integrations and chromosome rearrangements in oropharyngeal squamous cell carcinoma. (English)

1 reference

Europe PubMed Central

20 September 2017

author name string

Ge Gao

1

1 reference

Europe PubMed Central

20 September 2017

Sarah H Johnson

2

1 reference

Europe PubMed Central

20 September 2017

George Vasmatzis

3

1 reference

Europe PubMed Central

20 September 2017

Christina E Pauley

4

1 reference

Europe PubMed Central

20 September 2017

Nicole M Tombers

5

1 reference

Europe PubMed Central

20 September 2017

Jan L Kasperbauer

6

1 reference

Europe PubMed Central

20 September 2017

David I Smith

7

1 reference

Europe PubMed Central

20 September 2017

publication date

16 September 2016

1 reference

Europe PubMed Central

20 September 2017

Genes, Chromosomes and Cancer

1 reference

Europe PubMed Central

20 September 2017

56

1 reference

Europe PubMed Central

20 September 2017

1

1 reference

Europe PubMed Central

20 September 2017

59-74

1 reference

Europe PubMed Central

20 September 2017

The haplotype-resolved genome and epigenome of the aneuploid HeLa cancer cell line

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Genome-wide analysis of HPV integration in human cancers reveals recurrent, focal genomic instability

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WWOX in biological control and tumorigenesis

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The landscape of somatic copy-number alteration across human cancers

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Signatures of mutation and selection in the cancer genome

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HPV-16 E6 L83V variant in squamous cell carcinomas of the upper aerodigestive tract.

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Secondary structure formation and DNA instability at fragile site FRA16B

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Incidence trends for human papillomavirus-related and -unrelated oral squamous cell carcinomas in the United States

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Initiation of the breakage-fusion-bridge mechanism through common fragile site activation in human breast cancer cells: the model of PIP gene duplication from a break at FRA7I

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Congenital muscular dystrophy type 1D (MDC1D) due to a large intragenic insertion/deletion, involving intron 10 of the LARGE gene

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LRP1B deletion in high-grade serous ovarian cancers is associated with acquired chemotherapy resistance to liposomal doxorubicin

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The human papilloma virus-16 E7 oncoprotein is able to bind to the retinoblastoma gene product

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Molecular analysis of integrated human papillomavirus 16 sequences in the cervical cancer cell line SiHa.

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Discovery of recurrent t(6;7)(p25.3;q32.3) translocations in ALK-negative anaplastic large cell lymphomas by massively parallel genomic sequencing

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Preferential integration of human papillomavirus type 18 near the c-myc locus in cervical carcinoma.

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Mate pair sequencing of oropharyngeal squamous cell carcinomas reveals that HPV integration occurs much less frequently than in cervical cancer

1 reference

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Very large common fragile site genes and their potential role in cancer development

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WW domain-containing oxidoreductase's role in myriad cancers: clinical significance and future implications

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Induction of pRb degradation by the human papillomavirus type 16 E7 protein is essential to efficiently overcome p16INK4a-imposed G1 cell cycle Arrest

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DNA polymerase alpha inhibition by aphidicolin induces gaps and breaks at common fragile sites in human chromosomes

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Collisions between replication and transcription complexes cause common fragile site instability at the longest human genes

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Genome-wide profiling of HPV integration in cervical cancer identifies clustered genomic hot spots and a potential microhomology-mediated integration mechanism

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Frequent allelic loss and homozygous deletion in chromosome band 8p23 in oral cancer.

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Contribution of germline mutations to PARK2 gene inactivation in lung adenocarcinoma.

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Contig array CGH at 3p14.2 points to the FRA3B/FHIT common fragile region as the target gene in diffuse large B-cell lymphoma.

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Mutations in the parkin gene cause autosomal recessive juvenile parkinsonism

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Identification of submicroscopic genetic changes and precise breakpoint mapping in myelofibrosis using high resolution mate-pair sequencing

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Molecular profiling of common fragile sites in human fibroblasts.

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Common fragile site profiling in epithelial and erythroid cells reveals that most recurrent cancer deletions lie in fragile sites hosting large genes.

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Cancer of the oropharynx

1 reference

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Characterization CSMD1 in a large set of primary lung, head and neck, breast and skin cancer tissues

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HPV-associated head and neck cancer: a virus-related cancer epidemic

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Clinical, pathologic, and mutational spectrum of dystroglycanopathy caused by LARGE mutations

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The FHIT gene, spanning the chromosome 3p14.2 fragile site and renal carcinoma-associated t(3;8) breakpoint, is abnormal in digestive tract cancers

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Comprehensive analysis of HPV16 integration in OSCC reveals no significant impact of physical status on viral oncogene and virally disrupted human gene expression

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Viral load, gene expression and mapping of viral integration sites in HPV16-associated HNSCC cell lines.

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Failure of origin activation in response to fork stalling leads to chromosomal instability at fragile sites

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Whole genome scanning identifies genotypes associated with recurrence and metastasis in prostate tumors

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Acquisition of high-level chromosomal instability is associated with integration of human papillomavirus type 16 in cervical keratinocytes

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Mapping and analysis of HPV16 integration sites in a head and neck cancer cell line

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Relationship between FRA11F and 11q13 gene amplification in oral cancer

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Characterization of a conserved aphidicolin-sensitive common fragile site at human 4q22 and mouse 6C1: possible association with an inherited disease and cancer

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Molecular characterization of common fragile sites as a strategy to discover cancer susceptibility genes

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The E6 oncoprotein encoded by human papillomavirus types 16 and 18 promotes the degradation of p53

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The HPV-16 E6 and E6-AP complex functions as a ubiquitin-protein ligase in the ubiquitination of p53

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The role of CSMD1 in head and neck carcinogenesis

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The molecular basis of common and rare fragile sites

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DNA structure and the Werner protein modulate human DNA polymerase delta-dependent replication dynamics within the common fragile site FRA16D.

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Homozygous deletions define a region of 8p23.2 containing a putative tumor suppressor gene

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CSMD1 exhibits antitumor activity in A375 melanoma cells through activation of the Smad pathway.

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Common fragile sites are preferential targets for HPV16 integrations in cervical tumors

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The presence of multiple regions of homozygous deletion at the CSMD1 locus in oral squamous cell carcinoma question the role of CSMD1 in head and neck carcinogenesis

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Human papillomavirus is a necessary cause of invasive cervical cancer worldwide

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Systematic review of genomic integration sites of human papillomavirus genomes in epithelial dysplasia and invasive cancer of the female lower genital tract

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Differential disruption of genomic integrity and cell cycle regulation in normal human fibroblasts by the HPV oncoproteins.

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Molecular basis for expression of common and rare fragile sites

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Mfold web server for nucleic acid folding and hybridization prediction

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Papillomaviruses and cancer: from basic studies to clinical application

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Allelic loss on chromosomes 2q21 and 19p 13.2 in oxyphilic thyroid tumors

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Loss of heterozygosity at 19p13.2 and 2q21 in tumours from familial clusters of non-medullary thyroid carcinoma

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Chromosome 8 allelic loss and the outcome of patients with squamous cell carcinoma of the supraglottic larynx

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7 January 2021

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10.1002/GCC.22415

1 reference

Europe PubMed Central

20 September 2017

1 reference

Europe PubMed Central

20 September 2017

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