(Q40546172)

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Monocyte chemoattractant protein-1 and macrophage inflammatory protein-2 are involved in both excitotoxin-induced neurodegeneration and regeneration

scientific article published on July 2004

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scholarly article

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Europe PubMed Central

PubMed publication ID

21 September 2017

Monocyte chemoattractant protein-1 and macrophage inflammatory protein-2 are involved in both excitotoxin-induced neurodegeneration and regeneration (English)

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Europe PubMed Central

PubMed publication ID

21 September 2017

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neurodegeneration

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based on heuristic

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author name string

Kalehua AN

1

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Europe PubMed Central

PubMed publication ID

21 September 2017

Nagel JE

2

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Europe PubMed Central

PubMed publication ID

21 September 2017

Whelchel LM

3

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Europe PubMed Central

PubMed publication ID

21 September 2017

Gides JJ

4

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Europe PubMed Central

PubMed publication ID

21 September 2017

Pyle RS

5

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Europe PubMed Central

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21 September 2017

Smith RJ

6

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Europe PubMed Central

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21 September 2017

Kusiak JW

7

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Europe PubMed Central

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21 September 2017

Taub DD

8

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21 September 2017

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publication date

1 July 2004

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21 September 2017

Experimental Cell Research

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21 September 2017

297

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21 September 2017

1

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Europe PubMed Central

PubMed publication ID

21 September 2017

197-211

1 reference

Europe PubMed Central

PubMed publication ID

21 September 2017

Production and function of monocyte chemoattractant protein-1 and other β-chemokines in murine glial cells

1 reference

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7 January 2021

Expression of multiple functional chemokine receptors and monocyte chemoattractant protein-1 in human neurons

1 reference

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Murine astrocytes express a functional chemokine receptor

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Chemokines and chemokine receptors in CNS pathology.

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Expression of specific chemokines and chemokine receptors in the central nervous system of multiple sclerosis patients

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Immunohistochemical study of the beta-chemokine receptors CCR3 and CCR5 and their ligands in normal and Alzheimer's disease brains

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Differential and time-dependent expression of monocyte chemoattractant protein-1 mRNA by astrocytes and macrophages in rat brain: effects of ischemia and peripheral lipopolysaccharide administration

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Differential production of MCP-1 and cytokine-induced neutrophil chemoattractant in the ischemic brain after transient focal ischemia in rats

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Selective chemokine mRNA expression following brain injury

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Early and specific expression of monocyte chemoattractant protein-1 in the thalamus induced by cortical injury

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Production of monocyte chemotactic protein-1 by rat brain macrophages

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Early response of brain resident microglia to kainic acid-induced hippocampal lesions

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Microglial and astroglial reactions to ischemic and kainic acid-induced lesions of the adult rat hippocampus

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The neuronal death induced by endotoxic shock but not that induced by excitatory amino acids requires TNF-alpha

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Neuronal death in cytokine-activated primary human brain cell culture: role of tumor necrosis factor-alpha

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Monocyte chemoattractant protein-1 is a mediator of acute excitotoxic injury in neonatal rat brain

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Overriding the brain's intrinsic resistance to leukocyte recruitment with intraparenchymal injections of recombinant chemokines

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An investigation into the early stages of the inflammatory response following ibotenic acid-induced neuronal degeneration

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Production of basic fibroblast growth factor in cultured rat brain microglia

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Basic FGF in astroglial, microglial, and neuronal cultures: characterization of binding sites and modulation of release by lymphokines and trophic factors

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Seizure-associated induction of basic fibroblast growth factor and its receptor in the rat brain

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Development and selective neurodegeneration in cell cultures from different hippocampal regions

1 reference

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Glia protect hippocampal neurons against excitatory amino acid-induced degeneration: involvement of fibroblast growth factor

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Neuroprotective effect of chronic infusion of basic fibroblast growth factor on seizure-associated hippocampal damage

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Basic fibroblast growth factor protects against hypoxia-ischemia and NMDA neurotoxicity in neonatal rats

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Delayed administration of basic fibroblast growth factor protects against N-methyl-D-aspartate neurotoxicity in neonatal rats

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Basic fibroblast growth factor is highly neuroprotective against seizure-induced long-term behavioural deficits

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Immortal rat hippocampal cell lines exhibit neuronal and glial lineages and neurotrophin gene expression

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Directed establishment of rat brain cell lines with the phenotypic characteristics of type 1 astrocytes

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Loss and reacquisition of hippocampal synapses after selective destruction of CA3-CA4 afferents with kainic acid

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Reactive synaptogenesis in hippocampal area CA1 of aged and young adult rats

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Combined Hoechst 33342 and merocyanine 540 staining to examine murine B cell cycle stage, viability and apoptosis

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Interleukin-1 stimulation of astroglial proliferation after brain injury

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Interleukin-1 injected into mammalian brain stimulates astrogliosis and neovascularization

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Interleukin-1 is an astroglial growth factor in the developing brain.

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Anatomical studies of DNA fragmentation in rat brain after systemic kainate administration

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Degeneration of hippocampal CA3 pyramidal cells induced by intraventricular kainic acid

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Selective neuronal death in the contralateral hippocampus following unilateral kainate injections into the CA3 subfield.

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Intracellular signalling pathways induced by chemokines in natural killer cells

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A novel heterodimeric cysteine protease is required for interleukin-1 beta processing in monocytes

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Both caspase-dependent and caspase-independent pathways may be involved in hippocampal CA1 neuronal death because of loss of cytochrome c From mitochondria in a rat forebrain ischemia model

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R-deprenyl and R-2-heptyl-N-methylpropargylamine prevent apoptosis in cerebellar granule neurons induced by cytosine arabinoside but not low extracellular potassium.

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Identifiers

10.1016/J.YEXCR.2004.02.031

1 reference

Europe PubMed Central

PubMed publication ID

21 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

21 September 2017

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