(Q40750283)

English

Downregulation of c-fos gene transcription in cells transformed by E1A and cHa-ras oncogenes: a role of sustained activation of MAP/ERK kinase cascade and of inactive chromatin structure at c-fos promoter

scientific article published on January 2002

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Downregulation of c-fos gene transcription in cells transformed by E1A and cHa-ras oncogenes: a role of sustained activation of MAP/ERK kinase cascade and of inactive chromatin structure at c-fos promoter (English)

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

author name string

Alexander N Kukushkin

1

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Maria V Abramova

2

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Svetlana B Svetlikova

3

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Zalfia A Darieva

4

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Tatiana V Pospelova

5

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Valery A Pospelov

6

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Europe PubMed Central

PubMed publication ID

23 September 2017

language of work or name

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publication date

1 January 2002

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

21

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

5

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

719-730

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

https://scigraph.springernature.com/pub.10.1038/sj.onc.1205118

0 references

Activation of SRF-regulated chromosomal templates by Rho-family GTPases requires a signal that also induces H4 hyperacetylation

1 reference

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Signal transduction pathways activated and required for mammary carcinogenesis in response to specific oncogenes.

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A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

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Single-Step Method of RNA Isolation by Acid Guanidinium Thiocyanate–Phenol–Chloroform Extraction

1 reference

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21 January 2018

Epidermal growth factor induction of the c-jun promoter by a Rac pathway

1 reference

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Activation of MAP kinase kinase is necessary and sufficient for PC12 differentiation and for transformation of NIH 3T3 cells

1 reference

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Net, a negative Ras-switchable TCF, contains a second inhibition domain, the CID, that mediates repression through interactions with CtBP and de-acetylation

1 reference

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ERK activation induces phosphorylation of Elk-1 at multiple S/T-P motifs to high stoichiometry

1 reference

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MEK/ERK-mediated proliferation is negatively regulated by P38 map kinase in the human pancreatic cancer cell line, PANC-1.

1 reference

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Multiple ras effector pathways contribute to G(1) cell cycle progression

1 reference

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Net-b, a Ras-insensitive factor that forms ternary complexes with serum response factor on the serum response element of the fos promoter

1 reference

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TC21 and Ras share indistinguishable transforming and differentiating activities

1 reference

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Histone acetylation in chromatin structure and transcription

1 reference

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Effects of the inhibition of p38/RK MAP kinase on induction of five fos and jun genes by diverse stimuli

1 reference

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Occupation of the c-fos serum response element in vivo by a multi-protein complex is unaltered by growth factor induction

1 reference

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Identification of an oncoprotein- and UV-responsive protein kinase that binds and potentiates the c-Jun activation domain

1 reference

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The Rho family GTPases RhoA, Rac1, and CDC42Hs regulate transcriptional activation by SRF

1 reference

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Attenuated expression of the serum responsive T1 gene in ras transformed fibroblasts due to the inhibition of c-fos gene activity

1 reference

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Histone acetylases and deacetylases in cell proliferation

1 reference

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21 January 2018

E2F and histone deacetylase mediate transforming growth factor beta repression of cdc25A during keratinocyte cell cycle arrest

1 reference

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21 January 2018

Rb interacts with histone deacetylase to repress transcription

1 reference

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21 January 2018

Transformation by ras modifies AP1 composition and activity

1 reference

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Transcriptional repression by wild-type p53 utilizes histone deacetylases, mediated by interaction with mSin3a.

1 reference

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21 January 2018

Stabilization of p53 by adenovirus E1A occurs through its amino-terminal region by modification of the ubiquitin-proteasome pathway

1 reference

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The transcriptional coactivators p300 and CBP are histone acetyltransferases

1 reference

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The Bcr-Abl leukemia oncogene activates Jun kinase and requires Jun for transformation

1 reference

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Extracellular signal-regulated kinase 1/2-mediated phosphorylation of JunD and FosB is required for okadaic acid-induced activator protein 1 activation

1 reference

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In vivo protein-DNA interactions at the c-jun promoter in quiescent and serum-stimulated fibroblasts

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Ligand-induced recruitment of a histone deacetylase in the negative-feedback regulation of the thyrotropin beta gene

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Rapid detection of octamer binding proteins with 'mini-extracts', prepared from a small number of cells

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Characterization of downstream Ras signals that induce alternative protease-dependent invasive phenotypes

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Ternary complex factors: growth factor regulated transcriptional activators

1 reference

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21 January 2018

Regulation of transcription by MAP kinase cascades

1 reference

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21 January 2018

Autocrine growth and anchorage independence: two complementing Jun-controlled genetic programs of cellular transformation.

1 reference

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p38 mitogen-activated protein kinase-dependent activation of protein phosphatases 1 and 2A inhibits MEK1 and MEK2 activity and collagenase 1 (MMP-1) gene expression

1 reference

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Role of p38 and JNK mitogen-activated protein kinases in the activation of ternary complex factors

1 reference

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Expression of dominant negative Erk2 inhibits AP-1 transactivation and neoplastic transformation

1 reference

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Oncogenic Ras activates c-Jun via a separate pathway from the activation of extracellular signal-regulated kinases

1 reference

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v-src induces prostaglandin synthase 2 gene expression by activation of the c-Jun N-terminal kinase and the c-Jun transcription factor

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Differential targeting of MAP kinases to the ETS-domain transcription factor Elk-1.

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The mechanism of phosphorylation-inducible activation of the ETS-domain transcription factor Elk-1.

1 reference

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Temporal recruitment of the mSin3A-histone deacetylase corepressor complex to the ETS domain transcription factor Elk-1.

1 reference

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Id helix-loop-helix proteins inhibit nucleoprotein complex formation by the TCF ETS-domain transcription factors

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Trichostatin A and trapoxin: novel chemical probes for the role of histone acetylation in chromatin structure and function

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Attenuation of serum inducibility of immediate early genes by oncoproteins in tyrosine kinase signaling pathways

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Stress-induced inhibition of ERK1 and ERK2 by direct interaction with p38 MAP kinase

1 reference

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Integration of MAP kinase signal transduction pathways at the serum response element

1 reference

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Deregulation of p53/p21Cip1/Waf1 pathway contributes to polyploidy and apoptosis of E1A+cHa-ras transformed cells after gamma-irradiation

1 reference

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inferred from DOI database lookup

Chromatin structure analysis of the human c-fos promoter reveals a centrally positioned nucleosome

1 reference

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based on heuristic

inferred from DOI database lookup

Activation of ternary complex factor Elk-1 by MAP kinases

1 reference

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7 January 2021

based on heuristic

inferred from DOI database lookup

Autoregulation of fos: the dyad symmetry element as the major target of repression.

1 reference

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inferred from DOI database lookup

F9 embryonal carcinoma cells fail to stop at G1/S boundary of the cell cycle after gamma-irradiation due to p21WAF1/CIP1 degradation

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based on heuristic

inferred from DOI database lookup

Inhibition of p38 MAP kinase increases okadaic acid mediated AP-1 expression and DNA binding but has no effect on TRE dependent transcription

1 reference

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based on heuristic

inferred from DOI database lookup

One-way cross-talk between p38(MAPK) and p42/44(MAPK). Inhibition of p38(MAPK) induces low density lipoprotein receptor expression through activation of the p42/44(MAPK) cascade

1 reference

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based on heuristic

inferred from DOI database lookup

Activation of the c-fos enhancer by the erk MAP kinase pathway through two sequence elements: the c-fos AP-1 and p62TCF sites

1 reference

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7 January 2021

based on heuristic

inferred from DOI database lookup

Identifiers

10.1038/SJ.ONC.1205118

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Dimensions Publication ID

0 references

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

ResearchGate publication ID

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