(Q40790684)

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Spontaneously immortalized cell lines obtained from adult Atm null mice retain sensitivity to ionizing radiation and exhibit a mutational pattern suggestive of oxidative stress

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Europe PubMed Central

PubMed publication ID

23 September 2017

Spontaneously immortalized cell lines obtained from adult Atm null mice retain sensitivity to ionizing radiation and exhibit a mutational pattern suggestive of oxidative stress (English)

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Europe PubMed Central

PubMed publication ID

23 September 2017

author name string

Gage BM

1

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23 September 2017

Alroy D

2

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23 September 2017

Shin CY

3

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23 September 2017

Ponomareva ON

4

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23 September 2017

Dhar S

5

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23 September 2017

Sharma GG

6

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23 September 2017

Pandita TK

7

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23 September 2017

Thayer MJ

8

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23 September 2017

Turker MS

9

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23 September 2017

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publication date

1 July 2001

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23 September 2017

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23 September 2017

20

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23 September 2017

32

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23 September 2017

4291-4297

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23 September 2017

https://scigraph.springernature.com/pub.10.1038/sj.onc.1204509

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Atm-deficient mice: a paradigm of ataxia telangiectasia

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204509

21 January 2018

Molecular analysis of APRT deficiency in mouse P19 teratocarcinoma stem cell line

1 reference

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21 January 2018

At least two distinct epigenetic mechanisms are correlated with high-frequency "switching" for APRT phenotypic expression in mouse embryonal carcinoma stem cells

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204509

21 January 2018

Comparative effects of adenine analogs upon metabolic cooperation between Chinese hamster cells with different levels of adenine phosphoribosyltransferase activity

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204509

21 January 2018

Pleiotropic defects in ataxia-telangiectasia protein-deficient mice

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204509

21 January 2018

Neoplastic transformation of mouse C3H10T1/2 cells following exposure to neutrons does not involve mutation of ras gene as analyzed by SSCP and cycle sequencing.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204509

21 January 2018

Ataxia-telangiectasia: chronic activation of damage-responsive functions is reduced by alpha-lipoic acid

1 reference

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21 January 2018

Strange bedfellows in even stranger places: the role of ATM in meiotic cells, lymphocytes, tumors, and its functional links to p53.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204509

21 January 2018

Heterozygous manifestations in four autosomal recessive human cancer-prone syndromes: ataxia telangiectasia, xeroderma pigmentosum, Fanconi anemia, and Bloom syndrome.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204509

21 January 2018

Responses to DNA damage and regulation of cell cycle checkpoints by the ATM protein kinase family

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204509

21 January 2018

A cloning assay for 6-thioguanine resistance provides evidence against certain somatic mutational theories of aging

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204509

21 January 2018

A role for certain mouse Aprt sequences in resistance to toxic adenine analogs

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204509

21 January 2018

Differential induction of transcriptionally active p53 following UV or ionizing radiation: defects in chromosome instability syndromes?

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204509

21 January 2018

High spontaneous intrachromosomal recombination rates in ataxia-telangiectasia

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204509

21 January 2018

Ataxia-telangiectasia, cancer and the pathobiology of the ATM gene

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204509

21 January 2018

Chromosome end associations, telomeres and telomerase activity in ataxia telangiectasia cells

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204509

21 January 2018

Characterization of spontaneous and induced mutations in SV40-transformed normal and ataxia telangiectasia cell lines

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204509

21 January 2018

Ataxia-telangiectasia: is ATM a sensor of oxidative damage and stress?

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204509

21 January 2018

Mitotic recombination produces the majority of recessive fibroblast variants in heterozygous mice.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204509

21 January 2018

Spontaneous and ionizing radiation induced mutations involve large events when selecting for loss of an autosomal locus

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204509

21 January 2018

A cell line selected for resistance to ionizing radiation exhibits cross resistance to other genotoxic agents and a mutator phenotype for loss of heterozygosity events

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204509

21 January 2018

Characterization of ataxia telangiectasia fibroblasts with extended life-span through telomerase expression.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204509

21 January 2018

Targeted disruption of ATM leads to growth retardation, chromosomal fragmentation during meiosis, immune defects, and thymic lymphoma

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204509

21 January 2018

Comparative human cellular radiosensitivity: II. The survival following gamma-irradiation of unstimulated (G0) T-lymphocytes, T-lymphocyte lines, lymphoblastoid cell lines and fibroblasts from normal donors, from ataxia-telangiectasia patients and f

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204509

7 January 2021

based on heuristic

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APRT: a versatile in vivo resident reporter of local mutation and loss of heterozygosity

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1204509

7 January 2021

based on heuristic

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Identifiers

10.1038/SJ.ONC.1204509

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

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PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

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