(Q40803029)

English

Enhanced reporter gene expression in the rat brain from helper virus-free HSV-1 vectors packaged in the presence of specific mutated HSV-1 proteins that affect the virion

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Enhanced reporter gene expression in the rat brain from helper virus-free HSV-1 vectors packaged in the presence of specific mutated HSV-1 proteins that affect the virion (English)

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

vector-borne disease

0 references

1 reference

based on heuristic

inferred from title

Herpes simplex virus type 1

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based on heuristic

inferred from title

author name string

Yang T

1

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Zhang GR

2

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Zhang W

3

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Sun M

4

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Wang X

5

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Geller AI

6

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

publication date

1 May 2001

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Molecular Brain Research

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Europe PubMed Central

PubMed publication ID

23 September 2017

90

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Europe PubMed Central

PubMed publication ID

23 September 2017

1

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

1-16

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Europe PubMed Central

PubMed publication ID

23 September 2017

Mutational analysis of the herpes simplex virus type 1 trans-inducing factor Vmw65.

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Construction and characterization of a herpes simplex virus type 1 mutant unable to transinduce immediate-early gene expression

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Organization and evolution of the rat tyrosine hydroxylase gene

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A mutant of herpes simplex virus type 1 in which the UL13 protein kinase gene is disrupted

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A cosmid-based system for constructing mutants of herpes simplex virus type 1.

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The UL13 virion protein of herpes simplex virus type 1 is phosphorylated by a novel virus-induced protein kinase

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Identification of a target protein of US3 protein kinase of herpes simplex virus type 2.

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Post-transcriptional transactivation of human retroviral envelope glycoprotein expression by herpes simplex virus Us11 protein

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The use of a retroviral vector to identify foetal striatal neurones transplanted into the adult striatum

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Genetic analysis of the role of protein kinase C signaling pathways in behaviors by direct gene transfer with HSV-1 vectors

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A defective HSV-1 vector expresses Escherichia coli beta-galactosidase in cultured peripheral neurons

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Herpes simplex virus Vmw65-octamer binding protein interaction: a paradigm for combinatorial control of transcription

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Establishment of a noradrenergic clonal line of rat adrenal pheochromocytoma cells which respond to nerve growth factor

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The protein kinase family: conserved features and deduced phylogeny of the catalytic domains

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Allele replacement: an application that permits rapid manipulation of herpes simplex virus type 1 genomes

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Prolonged in vivo gene expression driven by a tyrosine hydroxylase promoter in a defective herpes simplex virus amplicon vector

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Cytotoxicity of a replication-defective mutant of herpes simplex virus type 1

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Sequence of the lacZ gene of Escherichia coli

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Preproenkephalin promoter yields region-specific and long-term expression in adult brain after direct in vivo gene transfer via a defective herpes simplex viral vector

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An efficient in vivo recombination cloning procedure for modifying and combining HSV-1 cosmids

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The herpes simplex virus 1 protein kinase US3 is required for protection from apoptosis induced by the virus

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Generation of high-titer defective HSV-1 vectors using an IE 2 deletion mutant and quantitative study of expression in cultured cortical cells.

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Herpes simplex virus type 1 ICP27 deletion mutants exhibit altered patterns of transcription and are DNA deficient

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The complete DNA sequence of the long unique region in the genome of herpes simplex virus type 1.

1 reference

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UL13 protein kinase of herpes simplex virus 1 complexes with glycoprotein E and mediates the phosphorylation of the viral Fc receptor: glycoproteins E and I.

1 reference

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Infection of sympathetic and sensory neurones with herpes simplex virus does not elicit a shut-off of cellular protein synthesis: implications for viral latency and herpes vectors

1 reference

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The UL13 protein kinase and the infected cell type are determinants of posttranslational modification of ICP0

1 reference

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Herpes simplex virus 1 protein kinase is encoded by open reading frame US3 which is not essential for virus growth in cell culture

1 reference

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The UL13 gene of herpes simplex virus 1 encodes the functions for posttranslational processing associated with phosphorylation of the regulatory protein alpha 22

1 reference

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Structure and function in the herpes simplex virus 1 RNA-binding protein U(s)11: mapping of the domain required for ribosomal and nucleolar association and RNA binding in vitro

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Herpes simplex virus type 1 DNA amplified as bacterial artificial chromosome in Escherichia coli: rescue of replication-competent virus progeny and packaging of amplicon vectors

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Evidence that the herpes simplex virus immediate early protein ICP27 acts post-transcriptionally during infection to regulate gene expression

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An HSV-1 vector containing the rat tyrosine hydroxylase promoter enhances both long-term and cell type-specific expression in the midbrain.

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Improved titers for helper virus-free herpes simplex virus type 1 plasmid vectors by optimization of the packaging protocol and addition of noninfectious herpes simplex virus-related particles (previral DNA replication enveloped particles) to the pa

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Diverse stabilities of expression in the rat brain from different cellular promoters in a helper virus-free herpes simplex virus type 1 vector system

1 reference

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Herpes simplex virus type 1 infection stimulates p38/c-Jun N-terminal mitogen-activated protein kinase pathways and activates transcription factor AP-1.

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Identifiers

10.1016/S0169-328X(01)00059-6

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

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