(Q40834415)

24 September 2017

title

Variability in gene expression patterns of Ewing tumor cell lines differing in EWS-FLI1 fusion type (English)

1 reference

24 September 2017

Aryee DN

1

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24 September 2017

Sommergruber W

2

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24 September 2017

Muehlbacher K

3

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24 September 2017

Dockhorn-Dworniczak B

4

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24 September 2017

Zoubek A

5

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24 September 2017

Kovar H

6

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24 September 2017

publication date

1 December 2000

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24 September 2017

published in

Laboratory Investigation

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24 September 2017

volume

80

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24 September 2017

issue

12

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24 September 2017

page(s)

1833-1844

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https://scigraph.springernature.com/pub.10.1038/labinvest.3780194

24 September 2017

exact match

0 references

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EWS-FLI1 fusion transcript structure is an independent determinant of prognosis in Ewing's sarcoma

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Among genes involved in the RB dependent cell cycle regulatory cascade, the p16 tumor suppressor gene is frequently lost in the Ewing family of tumors.

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The S3a ribosomal protein gene is identical to the Fte-1 (v-fos transformation effector) gene and the TNF-alpha-induced TU-11 gene, and its transcript level is altered in transformed and tumor cells

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EWS/FLI1-induced manic fringe renders NIH 3T3 cells tumorigenic

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Ewing sarcoma 11;22 translocation produces a chimeric transcription factor that requires the DNA-binding domain encoded by FLI1 for transformation.

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Identifiers

DOI

1 reference

24 September 2017

1 reference