(Q40838820)

English

Primary mouse fibroblasts deficient for c-Fos, p53 or for both proteins are hypersensitive to UV light and alkylating agent-induced chromosomal breakage and apoptosis

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

Primary mouse fibroblasts deficient for c-Fos, p53 or for both proteins are hypersensitive to UV light and alkylating agent-induced chromosomal breakage and apoptosis (English)

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

apoptotic process

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author name string

Lackinger D

1

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

Kaina B

2

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

language of work or name

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publication date

1 December 2000

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

Mutation Research

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Europe PubMed Central

PubMed publication ID

24 September 2017

457

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

1-2

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

113-123

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

Induction of metallothionein and other mRNA species by carcinogens and tumor promoters in primary human skin fibroblasts

1 reference

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7 January 2021

Purified transcription factor AP-1 interacts with TPA-inducible enhancer elements

1 reference

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7 January 2021

The jun and fos protein families are both required for cell cycle progression in fibroblasts

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

Proto-oncogenes of the fos/jun family of transcription factors are positive regulators of myeloid differentiation

1 reference

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7 January 2021

Extended life span and tumorigenicity of nonestablished mouse connective tissue cells transformed by the fos oncogene of FBR-MuSV.

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

Continuous c-fos expression precedes programmed cell death in vivo

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

c-Fos is involved in the cellular defence against the genotoxic effect of UV radiation

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

Enhanced phosphorylation of p53 by ATM in response to DNA damage

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

DNA-dependent protein kinase acts upstream of p53 in response to DNA damage

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

p53, the cellular gatekeeper for growth and division

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

p53: puzzle and paradigm

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

Wild-type p53 is a cell cycle checkpoint determinant following irradiation

1 reference

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Expression of wild-type p53 is required for efficient global genomic nucleotide excision repair in UV-irradiated human fibroblasts

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

p53-mediated DNA repair responses to UV radiation: studies of mouse cells lacking p53, p21, and/or gadd45 genes

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

Role of heteroduplex joints in the functional interactions between human Rad51 and wild-type p53.

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

p53 is involved in regulation of the DNA repair gene O6-methylguanine-DNA methyltransferase (MGMT) by DNA damaging agents

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

Altered cell cycle arrest and gene amplification potential accompany loss of wild-type p53

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

Delayed DNA damage associated with mitotic catastrophe following X-irradiation of HeLa S3 cells.

1 reference

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7 January 2021

A novel assay for apoptosis. Flow cytometric detection of phosphatidylserine expression on early apoptotic cells using fluorescein labelled Annexin V

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

DNA double-strand breaks, chromosomal rearrangements, and genomic instability

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

Critical steps in alkylation-induced aberration formation

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

DNA double-strand breaks induced by sparsely ionizing radiation and endonucleases as critical lesions for cell death, chromosomal aberrations, mutations and oncogenic transformation

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

Apoptosis induced by X-rays and chemical agents in murine fibroblastic cell lines with a defect in repair of DNA double-strand breaks

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

Presence of double-strand breaks with single-base 3' overhangs in cells undergoing apoptosis but not necrosis

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

Fewer chromosome aberrations and earlier apoptosis induced by DNA synthesis inhibitors, a topoisomerase II inhibitor or alkylating agents in human cells with normal compared with mutant p53.

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

DNA damage, oncogenesis and the p53 tumour-suppressor gene

1 reference

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7 January 2021

p53 is required for radiation-induced apoptosis in mouse thymocytes

1 reference

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7 January 2021

p53 mutations increase resistance to ionizing radiation

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

p53 activates the CD95 (APO-1/Fas) gene in response to DNA damage by anticancer drugs

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

Wild-type function of the p53 tumor suppressor protein is not required for apoptosis of mouse hepatoma cells

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

Role of DNA mismatch repair and p53 in signaling induction of apoptosis by alkylating agents.

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

Two domains of p53 interact with the TATA-binding protein, and the adenovirus 13S E1A protein disrupts the association, relieving p53-mediated transcriptional repression

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

Modulation of activity of the promoter of the human MDR1 gene by Ras and p53.

1 reference

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7 January 2021

Ribozyme-mediated reversal of the multidrug-resistant phenotype

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

Inhibition of c-Fos expression in the UV-irradiated epidermis by topical application of antisense oligodeoxynucleotides suppresses activation of proliferating cell nuclear antigen

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

Wild-type human p53 transactivates the human proliferating cell nuclear antigen promoter

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

DNA damage-induced gene expression: signal transduction and relation to growth factor signaling

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

based on heuristic

inferred from DOI database lookup

DNA damage and cell cycle checkpoints

1 reference

https://api.crossref.org/works/10.1016%2FS0027-5107%2800%2900133-0

7 January 2021

based on heuristic

inferred from DOI database lookup

Identifiers

10.1016/S0027-5107(00)00133-0

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

24 September 2017

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