(Q40937408)

English

The PIM-1 serine kinase prolongs survival and inhibits apoptosis-related mitochondrial dysfunction in part through a bcl-2-dependent pathway

scientific article published on July 1999

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scholarly article

1 reference

Europe PubMed Central

25 September 2017

The PIM-1 serine kinase prolongs survival and inhibits apoptosis-related mitochondrial dysfunction in part through a bcl-2-dependent pathway (English)

1 reference

Europe PubMed Central

25 September 2017

author name string

Lilly M

1

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Europe PubMed Central

25 September 2017

Sandholm J

2

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Europe PubMed Central

25 September 2017

Cooper JJ

3

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Europe PubMed Central

25 September 2017

Koskinen PJ

4

1 reference

Europe PubMed Central

25 September 2017

Kraft A

5

1 reference

Europe PubMed Central

25 September 2017

language of work or name

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publication date

1 July 1999

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Europe PubMed Central

25 September 2017

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Europe PubMed Central

25 September 2017

18

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Europe PubMed Central

25 September 2017

4022-4031

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Europe PubMed Central

25 September 2017

27

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Europe PubMed Central

25 September 2017

https://scigraph.springernature.com/pub.10.1038/sj.onc.1202741

0 references

Collaboration of pim-1 and bcl-2 in lymphomagenesis

1 reference

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Transduction of interleukin-2 antiapoptotic and proliferative signals via Akt protein kinase

1 reference

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Complementation tagging of cooperating oncogenes in knockout mice

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The human protooncogene product p33pim is expressed during fetal hematopoiesis and in diverse leukemias

1 reference

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Mitochondrial cytochrome c release in apoptosis occurs upstream of DEVD-specific caspase activation and independently of mitochondrial transmembrane depolarization.

1 reference

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Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery

1 reference

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Interleukin-3-induced phosphorylation of BAD through the protein kinase Akt

1 reference

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Suppression of apoptosis allows differentiation and development of a multipotent hemopoietic cell line in the absence of added growth factors

1 reference

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KID-1, a protein kinase induced by depolarization in brain

1 reference

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Chapter 3 Glutathione and Cellular Resistance to Anti-Cancer Drugs

1 reference

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Role of the mitochondrial permeability transition pore in apoptosis

1 reference

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7 January 2021

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Bcl-2 functions in an antioxidant pathway to prevent apoptosis

1 reference

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Raf/MAPK and rapamycin-sensitive pathways mediate the anti-apoptotic function of p21Ras in IL-3-dependent hematopoietic cells

1 reference

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The AML1/ETO fusion protein activates transcription of BCL-2

1 reference

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The release of cytochrome c from mitochondria: a primary site for Bcl-2 regulation of apoptosis

1 reference

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Bcl-2 and Bax function independently to regulate cell death

1 reference

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Pim-1 kinase and p100 cooperate to enhance c-Myb activity

1 reference

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A discontinuous method for catalase determination at 'near physiological' concentrations of H2O2 and its application to the study of H2O2 fluxes within cells

1 reference

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The kinase domain of Jak2 mediates induction of bcl-2 and delays cell death in hematopoietic cells

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202741

7 January 2021

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Resistance to apoptosis in CTLL-2 cells constitutively expressing c-Myb is associated with induction of BCL-2 expression and Myb-dependent regulation of bcl-2 promoter activity

1 reference

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Tumorigenic activity of the BCR-ABL oncogenes is mediated by BCL2.

1 reference

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7 January 2021

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Bcl-2 prevents apoptotic mitochondrial dysfunction by regulating proton flux

1 reference

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7 January 2021

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Electroporation in ‘intracellular’ buffer increases cell survival

1 reference

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7 January 2021

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Predisposition to lymphomagenesis in pim-1 transgenic mice: cooperation with c-myc and N-myc in murine leukemia virus-induced tumors

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202741

7 January 2021

based on heuristic

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Bcl-2 targets the protein kinase Raf-1 to mitochondria

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1202741

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Bad, a heterodimeric partner for Bcl-XL and Bcl-2, displaces Bax and promotes cell death

1 reference

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Sequential reduction of mitochondrial transmembrane potential and generation of reactive oxygen species in early programmed cell death

1 reference

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Mitochondrial control of nuclear apoptosis.

1 reference

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Identifiers

10.1038/SJ.ONC.1202741

1 reference

Europe PubMed Central

25 September 2017

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1 reference

Europe PubMed Central

25 September 2017

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