(Q41071837)

26 September 2017

title

Regions outside of the Bcl-2 homology domains, BH1 and BH2 protect a dopaminergic neuronal cell line from staurosporine-induced cell death (English)

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26 September 2017

0 references

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Oh YJ

1

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26 September 2017

Uhland-Smith A

series ordinal

2

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26 September 2017

Kim JE

series ordinal

3

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26 September 2017

O'Malley KL

series ordinal

4

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26 September 2017

publication date

1 November 1997

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26 September 2017

published in

Molecular Brain Research

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26 September 2017

volume

51

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26 September 2017

issue

1-2

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26 September 2017

page(s)

133-142

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The proto-oncogene bcl-2 can selectively rescue neurotrophic factor-dependent neurons from apoptosis

26 September 2017

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Inhibitors of protein synthesis and RNA synthesis prevent neuronal death caused by nerve growth factor deprivation

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Endothelial cell inflammatory responses to tumor necrosis factor alpha. Ceramide-dependent and -independent mitogen-activated protein kinase cascades

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7 January 2021

Overexpression of Bcl-2 attenuates MPP+, but not 6-ODHA, induced cell death in a dopaminergic neuronal cell line

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7 January 2021

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7 January 2021

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7 January 2021

Neither macromolecular synthesis nor myc is required for cell death via the mechanism that can be controlled by Bcl-2.

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

BH1 and BH2 domains of Bcl-2 are required for inhibition of apoptosis and heterodimerization with Bax.

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7 January 2021

Proapoptotic protein Bax heterodimerizes with Bcl-2 and homodimerizes with Bax via a novel domain (BH3) distinct from BH1 and BH2

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7 January 2021

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7 January 2021

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Identifiers

DOI

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26 September 2017

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