(Q41108579)

26 September 2017

title

Caspase-11 activates a canonical NLRP3 inflammasome by promoting K(+) efflux (English)

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26 September 2017

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2

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26 September 2017

Sebastian Rühl

1

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26 September 2017

language of work or name

English

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publication date

14 July 2015

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European Journal of Immunology

26 September 2017

published in

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26 September 2017

volume

45

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26 September 2017

issue

10

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26 September 2017

page(s)

2927-2936

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Recognition of bacteria by inflammasomes

26 September 2017

cites work

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201545772

Differential requirement for Caspase-1 autoproteolysis in pathogen-induced cell death and cytokine processing

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201545772

Deregulated inflammasome signaling in disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201545772

Structures of the HIN Domain:DNA Complexes Reveal Ligand Binding and Activation Mechanisms of the AIM2 Inflammasome and IFI16 Receptor

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201545772

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201545772

Activation of the NALP3 inflammasome is triggered by low intracellular potassium concentration

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201545772

K⁺ efflux is the common trigger of NLRP3 inflammasome activation by bacterial toxins and particulate matter

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201545772

Silica crystals and aluminum salts activate the NALP3 inflammasome through phagosomal destabilization

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201545772

The NALP3 inflammasome is involved in the innate immune response to amyloid-beta

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201545772

Oxidized mitochondrial DNA activates the NLRP3 inflammasome during apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201545772

A role for mitochondria in NLRP3 inflammasome activation

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201545772

Non-canonical inflammasome activation targets caspase-11

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201545772

Caspase-11 increases susceptibility to Salmonella infection in the absence of caspase-1

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201545772

TRIF licenses caspase-11-dependent NLRP3 inflammasome activation by gram-negative bacteria

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201545772

Noncanonical inflammasome activation by intracellular LPS independent of TLR4.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201545772

Cytoplasmic LPS activates caspase-11: implications in TLR4-independent endotoxic shock

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201545772

The role of potassium in inflammasome activation by bacteria

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201545772

Inflammatory caspases are innate immune receptors for intracellular LPS.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201545772

inferred from DOI database lookup

7 January 2021

based on heuristic

A primary role for K+ and Na+ efflux in the activation of apoptosis

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201545772

inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1002/EJI.201545772

1 reference

26 September 2017

1 reference