(Q42439505)

31 October 2017

title

Platelet-derived growth factor isoform expression in carbon tetrachloride-induced chronic liver injury (English)

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31 October 2017

Ralf Weiskirchen

9

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author name string

Erawan Borkham-Kamphorst

1

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31 October 2017

Evgenia Kovalenko

2

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31 October 2017

Claudia R C van Roeyen

3

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31 October 2017

Nikolaus Gassler

4

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31 October 2017

Michael Bomble

5

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31 October 2017

Tammo Ostendorf

6

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31 October 2017

Jürgen Floege

7

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31 October 2017

Axel M Gressner

8

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31 October 2017

publication date

28 July 2008

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31 October 2017

published in

Laboratory Investigation

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31 October 2017

volume

88

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31 October 2017

issue

10

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31 October 2017

page(s)

1090-1100

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The PDGF family: four gene products form five dimeric isoforms

31 October 2017

cites work

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Hepatotoxicity and Mechanism of Action of Haloalkanes: Carbon Tetrachloride as a Toxicological Model

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Activation of Kupffer cells during the course of carbon tetrachloride-induced liver injury and fibrosis in rats

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A significant part of macrophage-derived growth factor consists of at least two forms of PDGF.

21 January 2018

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Activation of cultured rat hepatic lipocytes by Kupffer cell conditioned medium. Direct enhancement of matrix synthesis and stimulation of cell proliferation via induction of platelet-derived growth factor receptors

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Expression patterns of PDGF-A, -B, -C and -D and the PDGF-receptors alpha and beta in activated rat hepatic stellate cells (HSC)

21 January 2018

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Translocation of platelets into Disse spaces and their entry into hepatocytes in response to lipopolysaccharides, interleukin-1 and tumour necrosis factor: the role of Kupffer cells

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PDGF C is a selective alpha platelet-derived growth factor receptor agonist that is highly expressed in platelet alpha granules and vascular smooth muscle

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Platelet-derived growth factor C induces liver fibrosis, steatosis, and hepatocellular carcinoma

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Platelet-derived growth factor receptor-alpha: a novel therapeutic target in human hepatocellular cancer.

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Dominant-negative soluble PDGF-beta receptor inhibits hepatic stellate cell activation and attenuates liver fibrosis

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Oral imatinib treatment reduces early fibrogenesis but does not prevent progression in the long term.

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Platelets Strongly Induce Hepatocyte Proliferation with IGF-1 and HGF In Vitro.

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7 January 2021

Expression of platelet-derived growth factor in a model of acute liver injury

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7 January 2021

Chapter 4 Preparation of Isolated Rat Liver Cells

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7 January 2021

Differential effects of TGF-β on connective tissue growth factor (CTGF/CCN2) expression in hepatic stellate cells and hepatocytes

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7 January 2021

Tissue injury and repair as parallel and opposing responses to CCl4 hepatotoxicity: a novel dose-response

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7 January 2021

Neutrophil adhesion is impaired in a rat model of cholestasis

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7 January 2021

Regulation of TGF beta gene expression in rat liver intoxicated with carbon tetrachloride

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7 January 2021

Cell population kinetics of Kupffer cells during the onset of fibrosis in rat liver by chronic carbon tetrachloride administration

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7 January 2021

Zonal location of compensatory hepatocyte proliferation following chemically induced hepatotoxicity in rats and humans

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7 January 2021

10.1038/LABINVEST.2008.71

Identifiers

DOI

1 reference

31 October 2017

1 reference