(Q42475308)

1 November 2017

title

Tumor necrosis factor-alpha and ceramides in insulin resistance (English)

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1 November 2017

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Brindley DN

1

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1 November 2017

Wang CN

2

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1 November 2017

Mei J

3

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1 November 2017

Xu J

4

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1 November 2017

Hanna AN

5

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1 November 2017

publication date

1 January 1999

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1 November 2017

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1 November 2017

volume

34 Suppl

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1 November 2017

page(s)

S85-8

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https://scigraph.springernature.com/pub.10.1007/bf02562240

1 November 2017

exact match

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cites work

Tumor necrosis factor-alpha suppresses insulin-induced tyrosine phosphorylation of insulin receptor and its substrates.

1 reference

12 December 2020

The growth factor-like effects of tumor necrosis factor-alpha. Stimulation of glucose transport activity and induction of glucose transporter and immediate early gene expression in 3T3-L1 preadipocytes

1 reference

12 December 2020

Tumor necrosis factor alpha-induced phosphorylation of insulin receptor substrate-1 (IRS-1). Possible mechanism for suppression of insulin-stimulated tyrosine phosphorylation of IRS-1

1 reference

12 December 2020

The insulin-dependent biosynthesis of GLUT1 and GLUT3 glucose transporters in L6 muscle cells is mediated by distinct pathways. Roles of p21ras and pp70 S6 kinase

1 reference

12 December 2020

Role of p21ras in insulin-stimulated glucose transport in 3T3-L1 adipocytes.

1 reference

12 December 2020

Differential regulation of GLUT1 and GLUT4 glucose transporters in skeletal muscle of a new model of type II diabetes. The obese SHR/N-cp rat

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12 December 2020

Antidiabetic thiazolidinediones block the inhibitory effect of tumor necrosis factor-alpha on differentiation, insulin-stimulated glucose uptake, and gene expression in 3T3-L1 cells

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12 December 2020

Effects of cell-permeable ceramides and tumor necrosis factor-alpha on insulin signaling and glucose uptake in 3T3-L1 adipocytes

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12 December 2020

Tumor necrosis factor enhances glucose uptake by peripheral tissues

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12 December 2020

Banting Lecture. Insulin action, diabetogenes, and the cause of type II diabetes

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12 December 2020

IRS-1-mediated inhibition of insulin receptor tyrosine kinase activity in TNF-alpha- and obesity-induced insulin resistance

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12 December 2020

Reduced tyrosine kinase activity of the insulin receptor in obesity-diabetes. Central role of tumor necrosis factor-alpha

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12 December 2020

Tumor necrosis factor-alpha regulation of glucose transporter (GLUT1) mRNA turnover. Contribution of the 3'-untranslated region of the GLUT1 message

1 reference

12 December 2020

Tumor necrosis factor alpha: a key component of the obesity-diabetes link

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12 December 2020

A novel pathway for tumor necrosis factor-alpha and ceramide signaling involving sequential activation of tyrosine kinase, p21(ras), and phosphatidylinositol 3-kinase

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12 December 2020

Tumor necrosis factor alpha inhibits signaling from the insulin receptor

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12 December 2020

The sphingomyelin cycle and the second messenger function of ceramide.

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12 December 2020

Sphingomyelinase and ceramide suppress insulin-induced tyrosine phosphorylation of the insulin receptor substrate-1

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12 December 2020

Tumor necrosis factor promotes phosphorylation and binding of insulin receptor substrate 1 to phosphatidylinositol 3-kinase in 3T3-L1 adipocytes

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12 December 2020

Tumor necrosis factor-alpha-induced insulin resistance in 3T3-L1 adipocytes is accompanied by a loss of insulin receptor substrate-1 and GLUT4 expression without a loss of insulin receptor-mediated signal transduction.

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12 December 2020

Effect of tumor necrosis factor-alpha on insulin action in cultured rat skeletal muscle cells

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12 December 2020

Tumor necrosis factor (TNF)-alpha inhibits insulin signaling through stimulation of the p55 TNF receptor and activation of sphingomyelinase

1 reference

12 December 2020

Identifiers

DOI

10.1007/BF02562240

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1 November 2017

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