(Q42519228)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:17890426%20AND%20SRC:MED&resulttype=core&format=json

8 January 2020

title

Triadin is a critical determinant of cellular Ca cycling and contractility in the heart (English)

1 reference

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8 January 2020

5

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8 January 2020

author name string

Uwe Kirchhefer

1

1 reference

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8 January 2020

Jan Klimas

2

1 reference

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8 January 2020

Hideo A Baba

3

1 reference

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8 January 2020

Igor B Buchwalow

4

1 reference

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8 January 2020

Marion Hüls

6

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8 January 2020

Marek Matus

7

1 reference

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8 January 2020

Frank U Müller

8

1 reference

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8 January 2020

Wilhelm Schmitz

9

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8 January 2020

Joachim Neumann

10

1 reference

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8 January 2020

publication date

21 September 2007

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:17890426%20AND%20SRC:MED&resulttype=core&format=json

American Journal of Physiology Heart and Circulatory Physiology

8 January 2020

published in

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8 January 2020

volume

293

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8 January 2020

issue

5

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8 January 2020

page(s)

H3165-74

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Cardiac excitation-contraction coupling

8 January 2020

cites work

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A mutation in calsequestrin, CASQ2D307H, impairs Sarcoplasmic Reticulum Ca2+ handling and causes complex ventricular arrhythmias in mice

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Junctin is a prominent regulator of contractility in cardiomyocytes

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On the role of junctin in cardiac Ca2+ handling, contractility, and heart failure

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The role of calsequestrin, triadin, and junctin in conferring cardiac ryanodine receptor responsiveness to luminal calcium

21 January 2018

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21 January 2018

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Purification, primary structure, and immunological characterization of the 26-kDa calsequestrin binding protein (junctin) from cardiac junctional sarcoplasmic reticulum

21 January 2018

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Overexpression of junctin causes adaptive changes in cardiac myocyte Ca(2+) signaling

21 January 2018

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Transgenic triadin 1 overexpression alters SR Ca2+ handling and leads to a blunted contractile response to beta-adrenergic agonists

21 January 2018

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Cardiac hypertrophy and impaired relaxation in transgenic mice overexpressing triadin 1

21 January 2018

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Impaired relaxation in transgenic mice overexpressing junctin.

21 January 2018

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Localization and characterization of the calsequestrin-binding domain of triadin 1. Evidence for a charged beta-strand in mediating the protein-protein interaction

21 January 2018

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Identification of triadin 1 as the predominant triadin isoform expressed in mammalian myocardium

21 January 2018

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Catecholaminergic polymorphic ventricular tachycardia: recent mechanistic insights

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Role of Ca2+/calmodulin-dependent protein kinase (CaMK) in excitation-contraction coupling in the heart

21 January 2018

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Transgenic CaMKIIdeltaC overexpression uniquely alters cardiac myocyte Ca2+ handling: reduced SR Ca2+ load and activated SR Ca2+ release

21 January 2018

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PKA phosphorylation dissociates FKBP12.6 from the calcium release channel (ryanodine receptor): defective regulation in failing hearts

21 January 2018

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A calcineurin-dependent transcriptional pathway for cardiac hypertrophy

21 January 2018

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CaM kinase signaling induces cardiac hypertrophy and activates the MEF2 transcription factor in vivo

21 January 2018

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21 January 2018

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Cardiac-specific overexpression of mouse cardiac calsequestrin is associated with depressed cardiovascular function and hypertrophy in transgenic mice

21 January 2018

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Regulation of cardiac sarcoplasmic reticulum Ca release by luminal [Ca] and altered gating assessed with a mathematical model.

21 January 2018

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Triadin overexpression stimulates excitation-contraction coupling and increases predisposition to cellular arrhythmia in cardiac myocytes

21 January 2018

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Protein protein interactions between triadin and calsequestrin are involved in modulation of sarcoplasmic reticulum calcium release in cardiac myocytes

21 January 2018

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Ryanodine receptor/calcium release channel PKA phosphorylation: a critical mediator of heart failure progression

21 January 2018

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21 January 2018

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Complex formation between junctin, triadin, calsequestrin, and the ryanodine receptor. Proteins of the cardiac junctional sarcoplasmic reticulum membrane

21 January 2018

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21 January 2018

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