(Q42832797)

8 November 2017

Evodiamine inhibits adipogenesis via the EGFR-PKCalpha-ERK signaling pathway (English)

1 reference

8 November 2017

1 reference

Ting Wang

1

1 reference

PubMed ID

19854188

8 November 2017

Youxue Wang

series ordinal

2

1 reference

PubMed ID

19854188

8 November 2017

Hitoshi Yamashita

series ordinal

3

1 reference

PubMed ID

19854188

8 November 2017

language of work or name

English

0 references

publication date

23 October 2009

1 reference

8 November 2017

1 reference

8 November 2017

583

1 reference

8 November 2017

22

1 reference

8 November 2017

3655-3659

1 reference

Molecular regulation of adipogenesis

8 November 2017

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2009.10.046

Evodiamine abolishes constitutive and inducible NF-kappaB activation by inhibiting IkappaBalpha kinase activation, thereby suppressing NF-kappaB-regulated antiapoptotic and metastatic gene expression, up-regulating apoptosis, and inhibiting invasion

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2009.10.046

Evodiamine and rutaecarpine inhibit migration by LIGHT via suppression of NADPH oxidase activation

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2009.10.046

Evodiamine functions as an agonist for the vanilloid receptor TRPV1.

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2009.10.046

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2009.10.046

Capsaicin-like anti-obese activities of evodiamine from fruits of Evodia rutaecarpa, a vanilloid receptor agonist

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2009.10.046

Evodiamine improves diet-induced obesity in a uncoupling protein-1-independent manner: involvement of antiadipogenic mechanism and extracellularly regulated kinase/mitogen-activated protein kinase signaling

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2009.10.046

Signaling pathways through which insulin regulates CCAAT/enhancer binding protein alpha (C/EBPalpha) phosphorylation and gene expression in 3T3-L1 adipocytes. Correlation with GLUT4 gene expression

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2009.10.046

Protein kinase C-epsilon promotes adipogenic commitment and is essential for terminal differentiation of 3T3-F442A preadipocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2009.10.046

Activation of transient receptor potential vanilloid type-1 channel prevents adipogenesis and obesity

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2009.10.046

TRPV1-null mice are protected from diet-induced obesity

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2009.10.046

Role of epidermal growth factor and ErbB2 receptors in 3T3-L1 adipogenesis

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2009.10.046

Multiple roles of phosphoinositide-specific phospholipase C isozymes

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2009.10.046

PKC isozymes and diacylglycerol-regulated proteins as effectors of growth factor receptors

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2009.10.046

The role of MAPKs in adipocyte differentiation and obesity.

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2009.10.046

10.1016/J.FEBSLET.2009.10.046

21 January 2018

Identifiers

DOI

1 reference

Fatcat

https://api.fatcat.wiki/v0/release/lb3eg5huabdlbmcujllfhyw6wy

mapped directly with Wikidata item

30 July 2022

based on heuristic

Fatcat ID

release_lb3eg5huabdlbmcujllfhyw6wy

1 reference

Fatcat

https://api.fatcat.wiki/v0/release/lb3eg5huabdlbmcujllfhyw6wy

mapped directly with Wikidata item

30 July 2022

based on heuristic

1 reference

https://api.fatcat.wiki/v0/release/lb3eg5huabdlbmcujllfhyw6wy