(Q43443509)

English

Cdc20 turnover rate: a key determinant in cancer patient response to anti-mitotic therapies?

scientific article published on 16 May 2013

In more languages

edit

Statements

instance of

scholarly article

1 reference

stated in

Europe PubMed Central

PubMed publication ID

23681847

retrieved

23 November 2017

title

Cdc20 turnover rate: a key determinant in cancer patient response to anti-mitotic therapies? (English)

1 reference

stated in

Europe PubMed Central

PubMed publication ID

23 November 2017

Andrew M Fry

1

1 reference

stated in

Europe PubMed Central

PubMed publication ID

23681847

retrieved

23 November 2017

language of work or name

English

0 references

publication date

16 May 2013

1 reference

stated in

Europe PubMed Central

PubMed publication ID

23 November 2017

1 reference

Europe PubMed Central

PubMed publication ID

23681847

retrieved

23 November 2017

volume

35

1 reference

stated in

Europe PubMed Central

PubMed publication ID

23681847

retrieved

23 November 2017

issue

9

1 reference

stated in

Europe PubMed Central

PubMed publication ID

23681847

retrieved

23 November 2017

page(s)

762

1 reference

stated in

Europe PubMed Central

PubMed publication ID

23681847

retrieved

23 November 2017

cites work

Cdc20 control of cell fate during prolonged mitotic arrest: do Cdc20 protein levels affect cell fate in response to antimitotic compounds?

1 reference

stated in

Crossref

reference URL

https://api.crossref.org/works/10.1002%2FBIES.201300053

retrieved

21 January 2018

APC15 drives the turnover of MCC-CDC20 to make the spindle assembly checkpoint responsive to kinetochore attachment

1 reference

stated in

Crossref

reference URL

https://api.crossref.org/works/10.1002%2FBIES.201300053

retrieved

21 January 2018

APC15 mediates CDC20 autoubiquitylation by APC/C(MCC) and disassembly of the mitotic checkpoint complex.

1 reference

stated in

Crossref

reference URL

https://api.crossref.org/works/10.1002%2FBIES.201300053

retrieved

21 January 2018

The APC/C subunit Mnd2/Apc15 promotes Cdc20 autoubiquitination and spindle assembly checkpoint inactivation.

1 reference

stated in

Crossref

reference URL

https://api.crossref.org/works/10.1002%2FBIES.201300053

retrieved

21 January 2018

Identifiers

DOI

10.1002/BIES.201300053

1 reference

stated in

Europe PubMed Central

PubMed publication ID

23681847

retrieved

23 November 2017

PubMed publication ID

23681847

1 reference

stated in

Europe PubMed Central

PubMed publication ID

23681847

retrieved

23 November 2017

ResearchGate publication ID

236916331

0 references

(Q43443509)

Cdc20 turnover rate: a key determinant in cancer patient response to anti-mitotic therapies?

Statements

Identifiers

Sitelinks

Wikipedia(0 entries)

Wikibooks(0 entries)

Wikinews(0 entries)

Wikiquote(0 entries)

Wikisource(0 entries)

Wikiversity(0 entries)

Wikivoyage(0 entries)

Wiktionary(0 entries)

Multilingual sites(0 entries)