(Q43507336)

24 November 2017

title

A bacterial pore-forming toxin forms aggregates in cells that resemble those associated with neurodegenerative diseases (English)

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24 November 2017

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4

Daniel A Portnoy

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24 November 2017

Julie P M Viala

1

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24 November 2017

Sofia N Mochegova

series ordinal

2

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24 November 2017

Nicole Meyer-Morse

series ordinal

3

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24 November 2017

publication date

6 December 2007

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24 November 2017

published in

Cellular Microbiology

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24 November 2017

volume

10

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24 November 2017

issue

4

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24 November 2017

page(s)

985-993

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Listeria and atypical presentations of Listeria in the central nervous system

24 November 2017

cites work

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p62/SQSTM1 forms protein aggregates degraded by autophagy and has a protective effect on huntingtin-induced cell death

21 January 2018

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Cutting edge: microbial products elicit formation of dendritic cell aggresome-like induced structures in macrophages.

21 January 2018

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Autophagy-mediated reentry of Francisella tularensis into the endocytic compartment after cytoplasmic replication

21 January 2018

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A PEST-like sequence in listeriolysin O essential for Listeria monocytogenes pathogenicity

21 January 2018

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Impairment of starvation-induced and constitutive autophagy in Atg7-deficient mice

21 January 2018

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Aggresomes, inclusion bodies and protein aggregation

21 January 2018

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LC3, an autophagosome marker, can be incorporated into protein aggregates independent of autophagy: caution in the interpretation of LC3 localization

21 January 2018

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Are amyloid diseases caused by protein aggregates that mimic bacterial pore-forming toxins?

21 January 2018

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Transient aggregation of ubiquitinated proteins during dendritic cell maturation

21 January 2018

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Dendritic cell aggresome-like induced structures are dedicated areas for ubiquitination and storage of newly synthesized defective proteins

21 January 2018

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Cryopyrin activates the inflammasome in response to toxins and ATP

21 January 2018

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The pleiotropic role of autophagy: from protein metabolism to bactericide.

21 January 2018

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Increased expression of p62 in expanded polyglutamine-expressing cells and its association with polyglutamine inclusions.

21 January 2018

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Autophagy defends cells against invading group A Streptococcus

21 January 2018

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p62/SQSTM1 binds directly to Atg8/LC3 to facilitate degradation of ubiquitinated protein aggregates by autophagy

21 January 2018

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Recognition of bacteria in the cytosol of Mammalian cells by the ubiquitin system

21 January 2018

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Protein aggregation and neurodegenerative disease

21 January 2018

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Opinion: What is the role of protein aggregation in neurodegeneration?

21 January 2018

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Listeriolysin O: a phagosome-specific lysin

21 January 2018

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Regulated translation of listeriolysin O controls virulence of Listeria monocytogenes

21 January 2018

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Phosphorylation, ubiquitination and degradation of listeriolysin O in mammalian cells: role of the PEST-like sequence

21 January 2018

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Listeriolysin O secreted by Listeria monocytogenes into the host cell cytosol is degraded by the N-end rule pathway

21 January 2018

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Molecular basis of listeriolysin O pH dependence

21 January 2018

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Sequestosome 1/p62 is a polyubiquitin chain binding protein involved in ubiquitin proteasome degradation

21 January 2018

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ALIS are stress-induced protein storage compartments for substrates of the proteasome and autophagy

21 January 2018

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Cholesterol-dependent cytolysins, a family of versatile pore-forming toxins

21 January 2018

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Listeria pathogenesis and molecular virulence determinants

21 January 2018

1 reference

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Signaling, polyubiquitination, trafficking, and inclusions: sequestosome 1/p62's role in neurodegenerative disease.

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1462-5822.2007.01100.X

p62 Is a common component of cytoplasmic inclusions in protein aggregation diseases

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1462-5822.2007.01100.X

10.1111/J.1462-5822.2007.01100.X

21 January 2018

Identifiers

DOI

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24 November 2017

1 reference