(Q43592786)

25 November 2017

title

Removal of glutathione produces apoptosis and necrosis in HepG2 cells overexpressing CYP2E1. (English)

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25 November 2017

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Wu D

1

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25 November 2017

Cederbaum AI

series ordinal

2

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25 November 2017

publication date

1 April 2001

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Alcoholism: Clinical and Experimental Research

25 November 2017

published in

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25 November 2017

volume

25

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25 November 2017

issue

4

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25 November 2017

page(s)

619-628

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Recent progress on regulation of the mitochondrial permeability transition pore; a cyclosporin-sensitive pore in the inner mitochondrial membrane

25 November 2017

cites work

1 reference

https://api.crossref.org/works/10.1111%2FJ.1530-0277.2001.TB02259.X

The protein bcl-2 alpha does not require membrane attachment, but two conserved domains to suppress apoptosis

21 January 2018

1 reference

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Cytotoxicity and apoptosis produced by arachidonic acid in Hep G2 cells overexpressing human cytochrome P4502E1.

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1530-0277.2001.TB02259.X

21 January 2018

The cell-death machine

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https://api.crossref.org/works/10.1111%2FJ.1530-0277.2001.TB02259.X

Stable expression of human cytochrome P4502E1 in HepG2 cells: characterization of catalytic activities and production of reactive oxygen intermediates

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1530-0277.2001.TB02259.X

Review: The role of glutathione in the regulation of apoptosis

21 January 2018

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https://api.crossref.org/works/10.1111%2FJ.1530-0277.2001.TB02259.X

Dual regulation of caspase activity by hydrogen peroxide: implications for apoptosis

21 January 2018

1 reference

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Cytosol-to-membrane redistribution of Bax and Bcl-X(L) during apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1530-0277.2001.TB02259.X

The glutathione precursor L-2-oxothiazolidine-4-carboxylic acid protects against liver injury due to chronic enteral ethanol exposure in the rat.

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1530-0277.2001.TB02259.X

Reactive oxygen species and programmed cell death

21 January 2018

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Reactive oxygen species and the regulation of cell death by the Bcl-2 gene family

21 January 2018

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Mitochondrial dysfunction in the pathogenesis of necrotic and apoptotic cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1530-0277.2001.TB02259.X

CYP2E1 overexpression in HepG2 cells induces glutathione synthesis by transcriptional activation of gamma-glutamylcysteine synthetase.

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1530-0277.2001.TB02259.X

Protease activation during apoptosis: death by a thousand cuts?

21 January 2018

1 reference

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Implication of free radical mechanisms in ethanol-induced cellular injury

21 January 2018

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Potentiation by chronic ethanol treatment of the mitochondrial permeability transition.

21 January 2018

1 reference

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Differential role of ethanol and acetaldehyde in the induction of oxidative stress in HEP G2 cells: effect on transcription factors AP-1 and NF-kappaB

21 January 2018

1 reference

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NF-kappaB is a target of AKT in anti-apoptotic PDGF signalling

21 January 2018

1 reference

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A comparative study of apoptosis and necrosis in HepG2 cells: oxidant-induced caspase inactivation leads to necrosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1530-0277.2001.TB02259.X

Relative level of expression of Bax and Bcl-XL determines the cellular fate of apoptosis/necrosis induced by the overexpression of Bax.

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1530-0277.2001.TB02259.X

Why are mitochondria involved in apoptosis? Permeability transition pores and apoptosis as selective mechanisms to eliminate superoxide-producing mitochondria and cell.

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1530-0277.2001.TB02259.X

Increased loss and decreased synthesis of hepatic glutathione after acute ethanol administration. Turnover studies

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1530-0277.2001.TB02259.X

Mitochondrial release of caspase-2 and -9 during the apoptotic process

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1530-0277.2001.TB02259.X

Apoptosis and necrosis: intracellular ATP level as a determinant for cell death modes

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1530-0277.2001.TB02259.X

Ethanol cytotoxicity to a transfected HepG2 cell line expressing human cytochrome P4502E1.

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1530-0277.2001.TB02259.X

Prevention of apoptosis by Bcl-2: release of cytochrome c from mitochondria blocked

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1530-0277.2001.TB02259.X

Mitochondrial control of nuclear apoptosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1530-0277.2001.TB02259.X

The mitochondrial permeability transition.

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1530-0277.2001.TB02259.X

Selective glutathione depletion of mitochondria by ethanol sensitizes hepatocytes to tumor necrosis factor

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1530-0277.2001.TB02259.X

10.1111/J.1530-0277.2001.TB02259.X

21 January 2018

Identifiers

DOI

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25 November 2017

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