(Q43623835)

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Co-involvement of mitochondria and endoplasmic reticulum in regulation of apoptosis: changes in cytochrome c, Bcl-2 and Bax in the hippocampus of aluminum-treated rabbits

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Europe PubMed Central

PubMed publication ID

25 November 2017

Co-involvement of mitochondria and endoplasmic reticulum in regulation of apoptosis: changes in cytochrome c, Bcl-2 and Bax in the hippocampus of aluminum-treated rabbits (English)

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Europe PubMed Central

PubMed publication ID

25 November 2017

endoplasmic reticulum

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author name string

Ghribi O

1

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Europe PubMed Central

PubMed publication ID

25 November 2017

DeWitt DA

2

1 reference

Europe PubMed Central

PubMed publication ID

25 November 2017

Forbes MS

3

1 reference

Europe PubMed Central

PubMed publication ID

25 November 2017

Herman MM

4

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Europe PubMed Central

PubMed publication ID

25 November 2017

Savory J

5

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Europe PubMed Central

PubMed publication ID

25 November 2017

language of work or name

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publication date

1 June 2001

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Europe PubMed Central

PubMed publication ID

25 November 2017

number of pages

8

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Europe PubMed Central

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25 November 2017

903

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Europe PubMed Central

PubMed publication ID

25 November 2017

1-2

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Europe PubMed Central

PubMed publication ID

25 November 2017

66-73

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Europe PubMed Central

PubMed publication ID

25 November 2017

The Bcl-2 protein family: arbiters of cell survival

1 reference

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The parkinsonian neurotoxin MPP+ opens the mitochondrial permeability transition pore and releases cytochrome c in isolated mitochondria via an oxidative mechanism.

1 reference

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7 January 2021

Regulation of lymphocyte survival by the bcl-2 gene family

1 reference

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Bid-induced conformational change of Bax is responsible for mitochondrial cytochrome c release during apoptosis

1 reference

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7 January 2021

Bax-induced cytochrome C release from mitochondria is independent of the permeability transition pore but highly dependent on Mg2+ ions

1 reference

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7 January 2021

Enforced dimerization of BAX results in its translocation, mitochondrial dysfunction and apoptosis

1 reference

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Apoptotic crosstalk between the endoplasmic reticulum and mitochondria controlled by Bcl-2

1 reference

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7 January 2021

Maintenance of calcium homeostasis in the endoplasmic reticulum by Bcl-2

1 reference

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Neurofibrillary lesions in experimental aluminum-induced encephalopathy and Alzheimer's disease share immunoreactivity for amyloid precursor protein, A beta, alpha 1-antichymotrypsin and ubiquitin-protein conjugates

1 reference

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Neuronal cytoskeletal lesions induced in the CNS by intraventricular and intravenous aluminium maltol in rabbits

1 reference

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The release of cytochrome c from mitochondria: a primary site for Bcl-2 regulation of apoptosis

1 reference

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Reactive oxygen species and the regulation of cell death by the Bcl-2 gene family

1 reference

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Bcl-2: Prolonging Life in a Transgenic Mouse Model of Familial Amyotrophic Lateral Sclerosis

1 reference

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7 January 2021

The mitochondrial permeability transition in cell death: a common mechanism in necrosis, apoptosis and autophagy

1 reference

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7 January 2021

Bcl-2 prevents hippocampal cell death induced by the neuroleptic drug haloperidol

1 reference

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Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

1 reference

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Induction of apoptotic program in cell-free extracts: requirement for dATP and cytochrome c

1 reference

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The permeability transition pore complex: a target for apoptosis regulation by caspases and bcl-2-related proteins

1 reference

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7 January 2021

The magnitude of brain lipid peroxidation correlates with the extent of degeneration but not with density of neuritic plaques or neurofibrillary tangles or with APOE genotype in Alzheimer's disease patients

1 reference

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7 January 2021

Release of mitochondrial cytochrome c and DNA fragmentation after cold injury-induced brain trauma in mice: possible role in neuronal apoptosis

1 reference

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7 January 2021

Caspase-12 mediates endoplasmic-reticulum-specific apoptosis and cytotoxicity by amyloid-beta

1 reference

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7 January 2021

Disturbance of endoplasmic reticulum functions: a key mechanism underlying cell damage?

1 reference

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7 January 2021

Double identity for proteins of the Bcl-2 family

1 reference

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7 January 2021

Bcl-2 family proteins

1 reference

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7 January 2021

Apoptosis and the nervous system

1 reference

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7 January 2021

Tau immunoreactivity associated with aluminum maltolate-induced neurofibrillary degeneration in rabbits

1 reference

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7 January 2021

Quantitative image analysis of temporal changes in tau and neurofilament proteins during the course of acute experimental neurofibrillary degeneration; non-phosphorylated epitopes precede phosphorylation

1 reference

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7 January 2021

Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC

1 reference

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7 January 2021

Autoactivation of procaspase-9 by Apaf-1-mediated oligomerization

1 reference

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Neuronal DNA damage precedes tangle formation and is associated with up-regulation of nitrotyrosine in Alzheimer's disease brain.

1 reference

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7 January 2021

CED-4—The Third Horseman of Apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2801%2902406-4

7 January 2021

Movement of Bax from the cytosol to mitochondria during apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2801%2902406-4

7 January 2021

Prevention of apoptosis by Bcl-2: release of cytochrome c from mitochondria blocked

1 reference

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7 January 2021

Mitochondrial control of nuclear apoptosis.

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2801%2902406-4

7 January 2021

Apaf-1, a human protein homologous to C. elegans CED-4, participates in cytochrome c-dependent activation of caspase-3

1 reference

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7 January 2021

Identifiers

10.1016/S0006-8993(01)02406-4

1 reference

Europe PubMed Central

PubMed publication ID

25 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

25 November 2017

ResearchGate publication ID

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