(Q43670222)

25 November 2017

title

C. elegans Kallmann syndrome protein KAL-1 interacts with syndecan and glypican to regulate neuronal cell migrations (English)

1 reference

Probable syndecan;Syndecan CELE_F57C7.3

25 November 2017

main subject

Caenorhabditis elegans

0 references

1 reference

GOA release 2020-03-11

GlyPicaN CELE_F59D12.4

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Human KALlmann syndrome homolog CELE_K03D10.1

GOA release 2020-03-11

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GOA release 2020-03-11

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2

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25 November 2017

4

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25 November 2017

Martin L Hudson

1

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25 November 2017

Hediye Nese Cinar

series ordinal

3

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25 November 2017

language of work or name

English

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publication date

3 May 2006

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25 November 2017

published in

Developmental Biology

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25 November 2017

volume

294

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25 November 2017

issue

2

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25 November 2017

page(s)

352-365

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The NC1/endostatin domain of Caenorhabditis elegans type XVIII collagen affects cell migration and axon guidance.

25 November 2017

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A gene deleted in Kallmann's syndrome shares homology with neural cell adhesion and axonal path-finding molecules

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7 January 2021

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FGF signaling functions in the hypodermis to regulate fluid balance in C. elegans.

7 January 2021

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The lin-15 locus encodes two negative regulators of Caenorhabditis elegans vulval development

7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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Kallmann syndrome: adhesion, afferents, and anosmia

7 January 2021

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Caenorhabditis elegans syndecan (SDN-1) is required for normal egg laying and associates with the nervous system and the vulva

7 January 2021

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7 January 2021

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7 January 2021

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Syndecan regulates cell migration and axon guidance in C. elegans

7 January 2021

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Products of the unc-52 gene in Caenorhabditis elegans are homologous to the core protein of the mammalian basement membrane heparan sulfate proteoglycan

7 January 2021

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Kallmann syndrome and the link between olfactory and reproductive development.

7 January 2021

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Luteinizing hormone-releasing hormone (LHRH)-expressing cells do not migrate normally in an inherited hypogonadal (Kallmann) syndrome

7 January 2021

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Anosmin-1, defective in the X-linked form of Kallmann syndrome, promotes axonal branch formation from olfactory bulb output neurons.

7 January 2021

1 reference

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Caenorhabditis elegans WASP and Ena/VASP proteins play compensatory roles in morphogenesis and neuronal cell migration

7 January 2021

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10.1016/J.YDBIO.2006.02.036

7 January 2021

Identifiers

DOI

1 reference

25 November 2017

1 reference