(Q43845628)

27 November 2017

title

Tumor necrosis factor receptor p55 and p75 deficiency protects mice from developing experimental autoimmune myasthenia gravis (English)

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27 November 2017

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Elzbieta Goluszko

1

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27 November 2017

Caishu Deng

series ordinal

2

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27 November 2017

Mathilde A Poussin

series ordinal

3

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27 November 2017

Premkumar Christadoss

series ordinal

4

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27 November 2017

language of work or name

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1 January 2002

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Journal of Neuroimmunology

27 November 2017

published in

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27 November 2017

volume

122

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27 November 2017

issue

1-2

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27 November 2017

page(s)

85-93

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Interferon gamma (IFN-gamma) is necessary for the genesis of acetylcholine receptor-induced clinical experimental autoimmune myasthenia gravis in mice

27 November 2017

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Muscle acetylcholine receptor loss in murine experimental autoimmune myasthenia gravis: correlated with cellular, humoral and clinical responses

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Abnormal development of peripheral lymphoid organs in mice deficient in lymphotoxin

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Inhibition of tumor necrosis factor activity minimizes target organ damage in experimental autoimmune uveoretinitis despite quantitatively normal activated T cell traffic to the retina

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Neutralizing TNF-alpha activity modulates T-cell phenotype and function in experimental autoimmune uveoretinitis

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Decreased sensitivity to tumour-necrosis factor but normal T-cell development in TNF receptor-2-deficient mice

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7 January 2021

Neuromuscular Junction in Myasthenia Gravis: Decreased Acetylcholine Receptors

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7 January 2021

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Lymphotoxin-alpha deficiency completely protects C57BL/6 mice from developing clinical experimental autoimmune myasthenia gravis

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7 January 2021

Polymorphism in tumor necrosis factor genes associated with myasthenia gravis

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Myasthenia gravis muscle antibodies examined by ELISA: IgG and IgM antibodies characterize different patient subgroups

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7 January 2021

Prevention of autoimmune diabetes mellitus in NOD mice by transgenic expression of soluble tumor necrosis factor receptor p55

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7 January 2021

Isotype-specific regulation of MHC class II gene expression in human monocytes by exogenous and endogenous tumor necrosis factor

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7 January 2021

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7 January 2021

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7 January 2021

Tumor necrosis factor-alpha, lymphotoxin, interleukin (IL)-6, IL-10, IL-12 and perforin mRNA expression in mononuclear cells in response to acetylcholine receptor is augmented in myasthenia gravis

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7 January 2021

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7 January 2021

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7 January 2021

Mice deficient for the 55 kd tumor necrosis factor receptor are resistant to endotoxic shock, yet succumb to L. monocytogenes infection

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7 January 2021

Suppression of experimental autoimmune myasthenia gravis in IL-10 gene-disrupted mice is associated with reduced B cells and serum cytotoxicity on mouse cell line expressing AChR.

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7 January 2021

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7 January 2021

Antiviral activity of tumor necrosis factor (TNF) is mediated via p55 and p75 TNF receptors

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7 January 2021

Experimental autoimmune encephalomyelitis: immunotherapy with anti-tumor necrosis factor antibodies and soluble tumor necrosis factor receptors

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7 January 2021

Failure to down-regulate Bcl-2 protein in thymic germinal center B cells in myasthenia gravis

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7 January 2021

Tumour necrosis factor alpha and its soluble receptors parallel clinical disease and autoimmune activity in systemic lupus erythematosus

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7 January 2021

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7 January 2021

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7 January 2021

In-vitro synthesis of anti-acetylcholine-receptor antibody by thymic lymphocytes in myasthenia gravis

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7 January 2021

Tumor necrosis factor receptor-1 is critically involved in the development of experimental autoimmune myasthenia gravis

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7 January 2021

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Identifiers

DOI

1 reference

27 November 2017

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