(Q43976517)

28 November 2017

title

Focal segmental glomerulosclerosis plays a major role in the progression of IgA nephropathy. II. Light microscopic and clinical studies (English)

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focal segmental glomerulosclerosis

28 November 2017

main subject

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7

Patrick Bruneval

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28 November 2017

Khalil El Karoui

1

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28 November 2017

Gary S Hill

2

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28 November 2017

Alexandre Karras

3

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28 November 2017

Luc Moulonguet

4

1 reference

28 November 2017

Valérie Caudwell

5

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28 November 2017

Alexandre Loupy

6

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28 November 2017

Christian Jacquot

8

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28 November 2017

Dominique Nochy

9

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28 November 2017

language of work or name

English

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publication date

22 December 2010

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28 November 2017

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28 November 2017

volume

79

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28 November 2017

issue

6

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28 November 2017

page(s)

643-654

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https://scigraph.springernature.com/pub.10.1038/ki.2010.460

28 November 2017

exact match

0 references

cites work

IgA nephropathy histologically resembling focal-segmental glomerulosclerosis: a clinicopathologic study of 18 cases.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

Focal segmental glomerulosclerosis in mild IgA nephropathy: a clinical-pathologic study

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

Segmental glomerulosclerosis in IgA nephropathy after renal transplantation: relationship with proteinuria and therapeutic response to enalapril.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

Histologic subclassification of IgA nephropathy: a clinicopathologic study of 244 cases.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

The Oxford classification of IgA nephropathy: pathology definitions, correlations, and reproducibility

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

The Oxford classification of IgA nephropathy: rationale, clinicopathological correlations, and classification

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

Mesangial medium with IgA1 from IgA nephropathy inhibits nephrin expression in mouse podocytes.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

Effect of aggregated immunoglobulin A1 from immunoglobulin A nephropathy patients on nephrin expression in podocytes

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

Podocyte injury induced by mesangial-derived cytokines in IgA nephropathy

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

Serum IgA1 from patients with IgA nephropathy up-regulates integrin-linked kinase synthesis and inhibits adhesive capacity in podocytes through indirect pathways.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

Activation of podocytes by mesangial-derived TNF-alpha: glomerulo-podocytic communication in IgA nephropathy

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

Downregulation of Bcl-2 by podocytes is associated with progressive glomerular injury and clinical indices of poor renal prognosis in human IgA nephropathy

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

Parietal podocytes in normal human glomeruli

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

The parietal epithelial cell is crucially involved in human idiopathic focal segmental glomerulosclerosis

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

Podocytes undergo phenotypic changes and express macrophagic-associated markers in idiopathic collapsing glomerulopathy

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

Transdifferentiation of epithelial glomerular cells

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

Significance of early phenotypic change of glomerular podocytes detected by Pax2 in primary focal segmental glomerulosclerosis

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

Proliferating cells in HIV and pamidronate-associated collapsing focal segmental glomerulosclerosis are parietal epithelial cells

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

Tracing the origin of glomerular extracapillary lesions from parietal epithelial cells.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

Regeneration of glomerular podocytes by human renal progenitors

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

Renal progenitor cells contribute to hyperplastic lesions of podocytopathies and crescentic glomerulonephritis

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

Pathologic classification of focal segmental glomerulosclerosis: a working proposal

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

Reduced glomerular epithelial protein 1 expression and podocyte injury in immunoglobulin A nephropathy

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

Targeted Downregulation of Extracellular Nephrin in Human IgA Nephropathy

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

Expression of podocyte-associated molecules in acquired human kidney diseases

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

Expression profile of nephrin, podocin, and CD2AP in Chinese children with MCNS and IgA nephropathy

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

Expression of nephrin in acquired human glomerular disease.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

The role of podocyte injury in the pathogenesis of focal segmental glomerulosclerosis

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

Activated parietal epithelial cells or dedifferentiated podocytes in FSGS: can we make the difference?

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

ACE gene polymorphism in childhood IgA nephropathy: association with clinicopathologic findings

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

Podocyte depletion causes glomerulosclerosis: diphtheria toxin-induced podocyte depletion in rats expressing human diphtheria toxin receptor transgene.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

Cumulative excretion of urinary podocytes reflects disease progression in IgA nephropathy and Schönlein-Henoch purpura nephritis

1 reference

https://api.crossref.org/works/10.1038%2FKI.2010.460

7 January 2021

Identifiers

DOI

10.1038/KI.2010.460

1 reference

28 November 2017

1 reference