(Q44006678)

28 November 2017

title

Stress-induced increase in extracellular sucrose space in rats is mediated by nitric oxide (English)

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María Ángeles Moro Sánchez

28 November 2017

author

series ordinal

2

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28 November 2017

3

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28 November 2017

Juan C Leza

5

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author name string

José L M Madrigal

series ordinal

1

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28 November 2017

Pedro Lorenzo

series ordinal

4

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28 November 2017

language of work or name

English

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publication date

1 May 2002

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28 November 2017

number of pages

5

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based on heuristic

inferred from page(s)

published in

Brain Research

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28 November 2017

volume

938

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28 November 2017

issue

1-2

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28 November 2017

page(s)

87-91

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Potential role of cerebral glutathione in the maintenance of blood-brain barrier integrity in rat.

28 November 2017

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7 January 2021

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N-(3-(aminomethyl)benzyl)acetamidine, an inducible nitric oxide synthase inhibitor, decreases colonic inflammation induced by trinitrobenzene sulphonic acid in rats.

7 January 2021

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7 January 2021

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The restraint stress-induced elevation in plasma interleukin-6 negatively regulates the plasma TNF-alpha level.

7 January 2021

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Chronic Stress Induces the Expression of Inducible Nitric Oxide Synthase in Rat Brain Cortex

7 January 2021

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Evaluation of the effect of stress on the blood--brain barrier: critical role of the brain perfusion time

7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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Involvement of interleukin-1 in immobilization stress-induced increase in plasma adrenocorticotropic hormone and in release of hypothalamic monoamines in the rat

7 January 2021

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Stress-induced increase in blood-brain barrier permeability in control and monosodium glutamate-treated rats.

7 January 2021

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Phosphorylation of vasodilator-stimulated phosphoprotein: a consequence of nitric oxide- and cGMP-mediated signal transduction in brain capillary endothelial cells and astrocytes.

7 January 2021

1 reference

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Nitric oxide and blood-brain barrier integrity

7 January 2021

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10.1016/S0006-8993(02)02467-8

7 January 2021

Identifiers

DOI

1 reference

28 November 2017

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