(Q44135799)

English

Astrocytes are less efficient in the removal of apoptotic lymphocytes than microglia cells: implications for the role of glial cells in the inflamed central nervous system

scientific article published in September 2002

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scholarly article

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Europe PubMed Central

30 November 2017

Astrocytes are less efficient in the removal of apoptotic lymphocytes than microglia cells: implications for the role of glial cells in the inflamed central nervous system (English)

1 reference

Europe PubMed Central

30 November 2017

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apoptotic process

1 reference

based on heuristic

inferred from title

5

object named as

Ralf Gold

1 reference

Europe PubMed Central

30 November 2017

Klaus Viktor Toyka

4

object named as

Klaus V Toyka

1 reference

Europe PubMed Central

30 November 2017

author name string

Tim Magnus

1

1 reference

Europe PubMed Central

30 November 2017

Andrew Chan

2

1 reference

Europe PubMed Central

30 November 2017

Ralf A Linker

3

1 reference

Europe PubMed Central

30 November 2017

publication date

1 September 2002

1 reference

Europe PubMed Central

30 November 2017

Journal of Neuropathology & Experimental Neurology

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Europe PubMed Central

30 November 2017

61

1 reference

Europe PubMed Central

30 November 2017

760-766

1 reference

Europe PubMed Central

30 November 2017

9

1 reference

Europe PubMed Central

30 November 2017

T-cell apoptosis in inflammatory brain lesions: destruction of T cells does not depend on antigen recognition

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F61.9.760

21 January 2018

T-cell apoptosis in autoimmune diseases: termination of inflammation in the nervous system and other sites with specialized immune-defense mechanisms

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F61.9.760

21 January 2018

Apoptosis in the nervous system in experimental allergic encephalomyelitis

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F61.9.760

21 January 2018

Apoptosis: the importance of being eaten

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F61.9.760

21 January 2018

Macrophages that have ingested apoptotic cells in vitro inhibit proinflammatory cytokine production through autocrine/paracrine mechanisms involving TGF-beta, PGE2, and PAF

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F61.9.760

21 January 2018

Production of nitrite by neonatal rat microglial cells/brain macrophages

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F61.9.760

21 January 2018

Plasma membrane phospholipid asymmetry precedes DNA fragmentation in different apoptotic cell models

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F61.9.760

21 January 2018

Apoptotic elimination of V beta 8.2+ cells from the central nervous system during recovery from experimental autoimmune encephalomyelitis induced by the passive transfer of V beta 8.2+ encephalitogenic T cells

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F61.9.760

21 January 2018

Microglial phagocytosis of apoptotic inflammatory T cells leads to down-regulation of microglial immune activation.

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F61.9.760

21 January 2018

Multiple sclerosis: a role for astroglia in active demyelination suggested by class II MHC expression and ultrastructural study

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F61.9.760

21 January 2018

Astrocytes attenuate oligodendrocyte death in vitro through an alpha(6) integrin-laminin-dependent mechanism

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F61.9.760

21 January 2018

Microglia: a sensor for pathological events in the CNS

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F61.9.760

21 January 2018

Immunosuppressive effects of apoptotic cells

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F61.9.760

21 January 2018

Identifiers

10.1093/JNEN/61.9.760

1 reference

Europe PubMed Central

30 November 2017

1 reference

Europe PubMed Central

30 November 2017

ResearchGate publication ID

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