(Q44147847)

English

Effect of β-amyloid (25-35) on mitochondrial function and expression of mitochondrial permeability transition pore proteins in rat hippocampal neurons

scientific article published in May 2011

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scholarly article

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Europe PubMed Central

PubMed publication ID

30 November 2017

Effect of β-amyloid (25-35) on mitochondrial function and expression of mitochondrial permeability transition pore proteins in rat hippocampal neurons (English)

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

author name string

Rui Ren

1

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

Yang Zhang

2

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

Benchang Li

3

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

Yanping Wu

4

1 reference

Europe PubMed Central

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30 November 2017

Baixiang Li

5

1 reference

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PubMed publication ID

30 November 2017

publication date

1 May 2011

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30 November 2017

Journal of Cellular Biochemistry

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30 November 2017

112

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Europe PubMed Central

PubMed publication ID

30 November 2017

5

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Europe PubMed Central

PubMed publication ID

30 November 2017

1450-1457

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

Beta-amyloid peptides induce mitochondrial dysfunction and oxidative stress in astrocytes and death of neurons through activation of NADPH oxidase.

1 reference

https://api.crossref.org/works/10.1002%2FJCB.23062

21 January 2018

Calcium elevation in mitochondria is the main Ca2+ requirement for mitochondrial permeability transition pore (mPTP) opening.

1 reference

https://api.crossref.org/works/10.1002%2FJCB.23062

21 January 2018

Targeting mitochondrial dysfunction in neurodegenerative disease: Part II.

1 reference

https://api.crossref.org/works/10.1002%2FJCB.23062

21 January 2018

Beta-amyloid fragment 25-35 causes mitochondrial dysfunction in primary cortical neurons

1 reference

https://api.crossref.org/works/10.1002%2FJCB.23062

21 January 2018

Mitochondria, calcium and cell death: a deadly triad in neurodegeneration

1 reference

https://api.crossref.org/works/10.1002%2FJCB.23062

21 January 2018

Mitochondrial permeability transition pore in Alzheimer's disease: cyclophilin D and amyloid beta

1 reference

https://api.crossref.org/works/10.1002%2FJCB.23062

21 January 2018

Cyclophilin D deficiency attenuates mitochondrial and neuronal perturbation and ameliorates learning and memory in Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1002%2FJCB.23062

21 January 2018

Neurodegenerative diseases and oxidative stress

1 reference

https://api.crossref.org/works/10.1002%2FJCB.23062

21 January 2018

Assay for apoptosis using the mitochondrial probes, Rhodamine123 and 10-N-nonyl acridine orange

1 reference

https://api.crossref.org/works/10.1002%2FJCB.23062

21 January 2018

The release of calcium from the endoplasmic reticulum induced by amyloid-beta and prion peptides activates the mitochondrial apoptotic pathway.

1 reference

https://api.crossref.org/works/10.1002%2FJCB.23062

21 January 2018

The peripheral benzodiazepine receptor: a promising therapeutic drug target

1 reference

https://api.crossref.org/works/10.1002%2FJCB.23062

21 January 2018

Long-term changes in the aggregation state and toxic effects of beta-amyloid injected into the rat brain

1 reference

https://api.crossref.org/works/10.1002%2FJCB.23062

21 January 2018

What is the mitochondrial permeability transition pore?

1 reference

https://api.crossref.org/works/10.1002%2FJCB.23062

21 January 2018

Mitochondrial permeability transition pore opening during myocardial reperfusion--a target for cardioprotection

1 reference

https://api.crossref.org/works/10.1002%2FJCB.23062

21 January 2018

The mitochondrial permeability transition pore complex as a pharmacological target. An introduction

1 reference

https://api.crossref.org/works/10.1002%2FJCB.23062

21 January 2018

Conformations and biological activities of amyloid beta peptide 25-35.

1 reference

https://api.crossref.org/works/10.1002%2FJCB.23062

21 January 2018

Amyloid beta-peptide promotes permeability transition pore in brain mitochondria

1 reference

https://api.crossref.org/works/10.1002%2FJCB.23062

21 January 2018

The consequences of mitochondrial amyloid beta-peptide in Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1002%2FJCB.23062

21 January 2018

Amyloid beta, mitochondrial structural and functional dynamics in Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1002%2FJCB.23062

21 January 2018

Possible role of amyloid-beta, adenine nucleotide translocase and cyclophilin-D interaction in mitochondrial dysfunction of Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1002%2FJCB.23062

21 January 2018

Phospholipases A2 mediate amyloid-beta peptide-induced mitochondrial dysfunction.

1 reference

https://api.crossref.org/works/10.1002%2FJCB.23062

21 January 2018

Regulation and pharmacology of the mitochondrial permeability transition pore

1 reference

https://api.crossref.org/works/10.1002%2FJCB.23062

21 January 2018

Identifiers

10.1002/JCB.23062

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

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