(Q44178896)

English

Withdrawal duration differentially affects c-fos expression in the medial prefrontal cortex and discrete subregions of the nucleus accumbens in cocaine-sensitized rats

scientific article published in January 2002

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

Withdrawal duration differentially affects c-fos expression in the medial prefrontal cortex and discrete subregions of the nucleus accumbens in cocaine-sensitized rats (English)

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

nucleus accumbens

1 reference

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author name string

Todtenkopf MS

1

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

Mihalakopoulos A

2

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

Stellar JR

3

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

language of work or name

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publication date

1 January 2002

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

114

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

4

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

1061-1069

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

A10 somatodendritic dopamine autoreceptor sensitivity following withdrawal from repeated cocaine treatment.

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Blockade of neurotensin receptors suppresses the dopamine D1/D2 synergism on immediate early gene expression in the rat brain

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Functional heterogeneity in dopamine release and in the expression of Fos-like proteins within the rat striatal complex.

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Selective sensitization to the psychosis-inducing effects of cocaine: a possible marker for addiction relapse vulnerability?

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Topographical organization and relationship with ventral striatal compartments of prefrontal corticostriatal projections in the rat

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Dopamine and glutamate agonists stimulate neuron-specific expression of Fos-like protein in the striatum

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Cortically driven Fos induction in the striatum is amplified by local dopamine D2-class receptor blockade

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D-amphetamine-induced behavioral sensitization: implication of a glutamatergic medial prefrontal cortex-ventral tegmental area innervation

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Psychomotor stimulant- and opiate-induced c-fos mRNA expression patterns in the rat forebrain: comparisons between acute drug treatment and a drug challenge in sensitized animals

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Amphetamine induction of c-fos in the nucleus accumbens is not inhibited by glutamate antagonists

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Amphetamine and cocaine induce drug-specific activation of the c-fos gene in striosome-matrix compartments and limbic subdivisions of the striatum

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The accumbens: beyond the core-shell dichotomy

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Specificity in the projection patterns of accumbal core and shell in the rat.

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The persistence of behavioral sensitization to cocaine parallels enhanced inhibition of nucleus accumbens neurons.

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Regulation of immediate early gene expression and AP-1 binding in the rat nucleus accumbens by chronic cocaine

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Evidence for a multi-compartmental histochemical organization of the nucleus accumbens in the rat

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Effect of acute and daily cocaine treatment on extracellular dopamine in the nucleus accumbens.

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Expression of the transcription factor deltaFosB in the brain controls sensitivity to cocaine

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Amphetamine and dopamine-induced immediate early gene expression in striatal neurons depends on postsynaptic NMDA receptors and calcium.

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The expression of cocaine sensitization is not prevented by MK-801 or ibotenic acid lesions of the medial prefrontal cortex.

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Transcriptional regulation in mammalian cells by sequence-specific DNA binding proteins

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Network-level changes in expression of inducible Fos-Jun proteins in the striatum during chronic cocaine treatment and withdrawal

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Coordinate expression of c-fos and jun B is induced in the rat striatum by cocaine.

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Cellular responses to psychomotor stimulant and neuroleptic drugs are abnormal in mice lacking the D1 dopamine receptor

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Repeated cocaine augments excitatory amino acid transmission in the nucleus accumbens only in rats having developed behavioral sensitization.

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A circuitry model of the expression of behavioral sensitization to amphetamine-like psychostimulants

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Ibotenic acid lesions of the dorsal prefrontal cortex disrupt the expression of behavioral sensitization to cocaine.

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Striatonigral projection neurons contain D1 dopamine receptor-activated c-fos.

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Alterations in the morphology of dendrites and dendritic spines in the nucleus accumbens and prefrontal cortex following repeated treatment with amphetamine or cocaine

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Amphetamine- and cocaine-induced fos in the rat striatum depends on D2 dopamine receptor activation

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Identifiers

10.1016/S0306-4522(02)00272-5

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

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