(Q44217206)

English

Inhibition of glutathione-related enzymes augments LPS-mediated cytokine biosynthesis: involvement of an IkappaB/NF-kappaB-sensitive pathway in the alveolar epithelium

scientific article published in October 2002

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scholarly article

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Europe PubMed Central

PubMed publication ID

30 November 2017

Inhibition of glutathione-related enzymes augments LPS-mediated cytokine biosynthesis: involvement of an IkappaB/NF-kappaB-sensitive pathway in the alveolar epithelium (English)

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

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Ryszard Lauterbach

4

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Ryszard Lauterbach

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

author name string

John J Haddad

1

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

Bared Safieh-Garabedian

2

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

Nayef E Saadé

3

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

publication date

1 October 2002

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Europe PubMed Central

PubMed publication ID

30 November 2017

International Immunopharmacology

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Europe PubMed Central

PubMed publication ID

30 November 2017

2

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

11

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

1567-1583

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

Immunological functions of the pulmonary epithelium

1 reference

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Targeting the alveolar epithelium in acute lung injury: keratinocyte growth factor and regulation of the alveolar epithelial barrier

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Alveolar epithelial barrier. Role in lung fluid balance in clinical lung injury

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Chemioxyexcitation (delta pO2/ROS)-dependent release of IL-1 beta, IL-6 and TNF-alpha: evidence of cytokines as oxygen-sensitive mediators in the alveolar epithelium

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Cytokines of the lung

1 reference

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Proinflammatory cytokines

1 reference

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Immunopharmacological potential of selective phosphodiesterase inhibition. I. Differential regulation of lipopolysaccharide-mediated proinflammatory cytokine (interleukin-6 and tumor necrosis factor-alpha) biosynthesis in alveolar epithelial cells

1 reference

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Immunopharmacological potential of selective phosphodiesterase inhibition. II. Evidence for the involvement of an inhibitory-kappaB/nuclear factor-kappaB-sensitive pathway in alveolar epithelial cells

1 reference

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Glutathione depletion is associated with augmenting a proinflammatory signal: evidence for an antioxidant/pro-oxidant mechanism regulating cytokines in the alveolar epithelium

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O(2)-evoked regulation of HIF-1alpha and NF-kappaB in perinatal lung epithelium requires glutathione biosynthesis.

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Antioxidant/pro-oxidant equilibrium regulates HIF-1alpha and NF-kappa B redox sensitivity. Evidence for inhibition by glutathione oxidation in alveolar epithelial cells.

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The involvement of L-gamma-glutamyl-L-cysteinyl-glycine (glutathione/GSH) in the mechanism of redox signaling mediating MAPK(p38)-dependent regulation of pro-inflammatory cytokine production

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Redox/ROS regulation of lipopolysaccharide-induced mitogen-activated protein kinase (MAPK) activation and MAPK-mediated TNF-alpha biosynthesis

1 reference

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Glutathione and glutathione-dependent enzymes represent a co-ordinately regulated defence against oxidative stress.

1 reference

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Functions of glutathione and glutathione disulfide in immunology and immunopathology.

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Glutathione deficiency in the epithelial lining fluid of the lower respiratory tract in idiopathic pulmonary fibrosis

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Increased transcription of the regulatory subunit of gamma-glutamylcysteine synthetase in rat lung epithelial L2 cells exposed to oxidative stress or glutathione depletion

1 reference

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Oxidized glutathione is increased in the alveolar fluid of patients with the adult respiratory distress syndrome

1 reference

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Bronchopulmonary dysplasia and oxidative stress: are we closer to an understanding of the pathogenesis of BPD?

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Systemic deficiency of glutathione in cystic fibrosis

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Multiple nuclear factors interact with the immunoglobulin enhancer sequences

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Series introduction: the transcription factor NF-kappaB and human disease

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Alpha-melanocyte-related tripeptide, Lys-d-Pro-Val, ameliorates endotoxin-induced nuclear factor kappaB translocation and activation: evidence for involvement of an interleukin-1beta193-195 receptor antagonism in the alveolar epithelium

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The differential expression of apoptosis factors in the alveolar epithelium is redox sensitive and requires NF-kappaB (RelA)-selective targeting

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7 January 2021

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Carbamoylation of glutathione reductase by N,N-bis(2-chloroethyl)-N- nitrosourea associated with inhibition of multidrug resistance protein (MRP) function

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Escherichia coli FPG and human OGG1 reduce DNA damage and cytotoxicity by BCNU in human lung cells

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Redox signaling and the emerging therapeutic potential of thiol antioxidants

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Role of redox potential and reactive oxygen species in stress signaling

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Assessing oxygen radicals as mediators in activation of inducible eukaryotic transcription factor NF-kappa B.

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Antioxidant and redox regulation of gene transcription

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Overexpression of gamma-glutamylcysteine synthetase suppresses tumor necrosis factor-induced apoptosis and activation of nuclear transcription factor-kappa B and activator protein-1

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Is NF-kappaB the sensor of oxidative stress?

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Dithiocarbamates as potent inhibitors of nuclear factor kappa B activation in intact cells

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Gene expression and the thiol redox state

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Separation of oxidant-initiated and redox-regulated steps in the NF-kappa B signal transduction pathway

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2-mercaptoethanol restores the ability of nuclear factor kappa B (NF kappa B) to bind DNA in nuclear extracts from interleukin 1-treated cells incubated with pyrollidine dithiocarbamate (PDTC). Evidence for oxidation of glutathione in the mechanism

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A cytokine-responsive IkappaB kinase that activates the transcription factor NF-kappaB

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Tumor necrosis factor and interleukin-1 lead to phosphorylation and loss of I kappa B alpha: a mechanism for NF-kappa B activation

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The p38 mitogen-activated protein kinase is required for NF-kappaB-dependent gene expression. The role of TATA-binding protein (TBP)

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Identifiers

10.1016/S1567-5769(02)00117-0

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

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