(Q44317343)

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Reduction of hypothalamic vasopressinergic hyperdrive contributes to clinically relevant behavioral and neuroendocrine effects of chronic paroxetine treatment in a psychopathological rat model.

scientific article published on 25 July 2002

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31 October 2019

Reduction of hypothalamic vasopressinergic hyperdrive contributes to clinically relevant behavioral and neuroendocrine effects of chronic paroxetine treatment in a psychopathological rat model (English)

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31 October 2019

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Manfred Uhr

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Florian Holsboer

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Florian Holsboer

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31 October 2019

Rainer Landgraf

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Rainer Landgraf

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31 October 2019

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Martin E Keck

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Marianne B Müller

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Frauke Ohl

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31 October 2019

Alexandra Wigger

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25 July 2002

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31 October 2019

Neuropsychopharmacology

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235-243

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31 October 2019

https://scigraph.springernature.com/pub.10.1038/sj.npp.1300040

0 references

Chronic fluoxetine treatment reduces hypothalamic vasopressin secretion in vitro.

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Vasopressinergic control of pituitary adrenocorticotropin secretion comes of age.

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Corticotropin releasing activity of the new CRF is potentiated several times by vasopressin

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Anxiolytic- and antidepressant-like effects of the non-peptide vasopressin V1b receptor antagonist, SSR149415, suggest an innovative approach for the treatment of stress-related disorders.

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D-Fenfluramine induces serotonin-mediated Fos expression in corticotropin-releasing factor and oxytocin neurons of the hypothalamus, and serotonin-independent Fos expression in enkephalin and neurotensin neurons of the amygdala.

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Hyperactivity of CRH neuronal circuits as a target for therapeutic interventions in affective disorders

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Neuroendocrine and behavioral effects of repetitive transcranial magnetic stimulation in a psychopathological animal model are suggestive of antidepressant-like effects

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The anxiolytic effect of the CRH(1) receptor antagonist R121919 depends on innate emotionality in rats.

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Vasopressin mediates the response of the combined dexamethasone/CRH test in hyper-anxious rats: implications for pathogenesis of affective disorders.

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Role of glucocorticoid hormones in arginine vasopressin gene regulation

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Behavioral, physiological, and neuroendocrine stress responses and differential sensitivity to diazepam in two Wistar rat lines selectively bred for high- and low-anxiety-related behavior.

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Behavioural profiles of two Wistar rat lines selectively bred for high or low anxiety-related behaviour.

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Differential regulation of corticotropin-releasing hormone and vasopressin transcription by glucocorticoids

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Clinical and neurobiological effects of tianeptine and paroxetine in major depression

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Dimensions of emotionality in a rat model of innate anxiety.

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Increased number of vasopressin- and oxytocin-expressing neurons in the paraventricular nucleus of the hypothalamus in depression.

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Selective serotonin reuptake inhibitors and neuroendocrine function

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The brain mineralocorticoid receptor: greedy for ligand, mysterious in function.

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The distribution and cells of origin of serotonergic inputs to the paraventricular and supraoptic nuclei of the rat

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Regulation of corticotropin-releasing factor neuronal systems and hypothalamic-pituitary-adrenal axis activity by stress and chronic antidepressant treatment

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Phenotypic plasticity of CRF neurons during stress

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Plasma levels of arginine vasopressin elevated in patients with major depression.

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Release of vasopressin from supraoptic neurons within the median eminence in vivo. A combined microdialysis and push-pull perfusion study in the rat.

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Long-term fluoxetine produces behavioral anxiolytic effects without inhibiting neuroendocrine responses to conditioned stress in rats

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Cortisol response in the combined dexamethasone/CRH test as predictor of relapse in patients with remitted depression. a prospective study

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Characterization of a novel, linear radioiodinated vasopressin antagonist: an excellent radioligand for vasopressin V1a receptors

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Identifiers

10.1038/SJ.NPP.1300040

1 reference

Europe PubMed Central

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31 October 2019

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31 October 2019

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