(Q44350757)

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Effects of high-dose methadone maintenance on cocaine place conditioning, cocaine self-administration, and mu-opioid receptor mRNA expression in the rat brain

scientific article published on 12 October 2005

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scholarly article

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Europe PubMed Central

PubMed publication ID

2 December 2017

Effects of high-dose methadone maintenance on cocaine place conditioning, cocaine self-administration, and mu-opioid receptor mRNA expression in the rat brain (English)

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Europe PubMed Central

PubMed publication ID

2 December 2017

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2 December 2017

Mary Jeanne Kreek

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object named as

Mary Jeanne Kreek

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Europe PubMed Central

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2 December 2017

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Francesco Leri

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2 December 2017

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Erin Cummins

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2 December 2017

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Benjamin Goddard

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publication date

12 October 2005

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2 December 2017

Neuropsychopharmacology

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2 December 2017

31

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2 December 2017

7

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2 December 2017

1462-1474

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Europe PubMed Central

PubMed publication ID

2 December 2017

https://scigraph.springernature.com/pub.10.1038/sj.npp.1300927

0 references

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Conditioned place preference using opiate and stimulant drugs: a meta-analysis.

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Opioidergic modulation of cocaine conditioned place preferences.

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Cocaine abuse sharply reduced in an effective methadone maintenance program

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Quantitation of preproenkephalin mRNA levels in brain regions from male Fischer rats following chronic cocaine treatment using a recently developed solution hybridization assay

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The basolateral amygdala-ventral striatal system and conditioned place preference: further evidence of limbic-striatal interactions underlying reward-related processes

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Immunocytochemical Localization of β-Endorphin-Containing Neurons in the Rat Brain

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Differential involvement of the core and shell subregions of the nucleus accumbens in conditioned cue-induced reinstatement of cocaine seeking in rats

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The d- and l-isomers of methadone bind to the non-competitive site on the N-methyl-D-aspartate (NMDA) receptor in rat forebrain and spinal cord.

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Methadone disrupts performance on the working memory version of the Morris water task.

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The use of place conditioning in studying the neuropharmacology of drug reinforcement.

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Differential control over cocaine-seeking behavior by nucleus accumbens core and shell

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DNQX in the nucleus accumbens inhibits cocaine-induced conditioned place preference.

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Reconditioning of drug-related cues: a potential contributor to relapse after drug reexposure.

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Methadone maintenance reduces heroin- and cocaine-induced relapse without affecting stress-induced relapse in a rodent model of poly-drug use.

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Pattern of cocaine use in methadone-maintained individuals applying for research studies.

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Mu, delta, and kappa opioid receptor mRNA expression in the rat CNS: an in situ hybridization study

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Optimizing response to methadone maintenance treatment: use of higher-dose methadone

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Cocaine induced secretion of ACTH, beta-endorphin, and corticosterone

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Persistence and drug-induced reinstatement of a morphine-induced conditioned place preference.

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Cocaine-induced conditioned place preference: reinstatement by priming injections of cocaine after extinction.

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Effects of chronic methadone treatment on cocaine- and food-maintained responding under second-order, progressive-ratio and concurrent-choice schedules in rhesus monkeys

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Reinstatement of both a conditioned place preference and a conditioned place aversion with drug primes.

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Cocaine use immediately prior to entry in an inpatient heroin detoxification unit as a predictor of discharges against medical advice.

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Enhancement of cocaine's abuse liability in methadone maintenance patients.

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Methadone versus buprenorphine with contingency management or performance feedback for cocaine and opioid dependence

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Regulation of drug-taking and -seeking behaviors by neuroadaptations in the mesolimbic dopamine system

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Cocaine abstinence during methadone maintenance: effects of repeated brief exposure to voucher-based reinforcement

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Nucleus accumbens beta-endorphin levels are not elevated by brain stimulation reward but do increase with extinction

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Steady-state methadone in rats does not change mRNA levels of corticotropin-releasing factor, its pituitary receptor or proopiomelanocortin.

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Identifiers

10.1038/SJ.NPP.1300927

1 reference

Europe PubMed Central

PubMed publication ID

2 December 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

2 December 2017

ResearchGate publication ID

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