(Q44413809)

English

N-methyl-D-aspartate receptor blockade after status epilepticus protects against limbic brain damage but not against epilepsy in the kainate model of temporal lobe epilepsy

scientific article published in January 2003

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scholarly article

1 reference

Europe PubMed Central

2 December 2017

N-methyl-D-aspartate receptor blockade after status epilepticus protects against limbic brain damage but not against epilepsy in the kainate model of temporal lobe epilepsy (English)

1 reference

Europe PubMed Central

2 December 2017

status epilepticus

1 reference

based on heuristic

inferred from title

Heidrun Potschka

2

1 reference

Europe PubMed Central

2 December 2017

author name string

Brandt C

1

1 reference

Europe PubMed Central

2 December 2017

Löscher W

3

1 reference

Europe PubMed Central

2 December 2017

Ebert U

4

1 reference

Europe PubMed Central

2 December 2017

language of work or name

0 references

publication date

1 January 2003

1 reference

Europe PubMed Central

2 December 2017

1 reference

Europe PubMed Central

2 December 2017

118

1 reference

Europe PubMed Central

2 December 2017

3

1 reference

Europe PubMed Central

2 December 2017

727-740

1 reference

Europe PubMed Central

2 December 2017

Vigabatrin protects against hippocampal damage but is not antiepileptogenic in the lithium-pilocarpine model of temporal lobe epilepsy

1 reference

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Causes of epilepsy: contributions of the Rochester epidemiology project.

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Limbic seizure and brain damage produced by kainic acid: mechanisms and relevance to human temporal lobe epilepsy

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Apoptosis and proliferation of dentate gyrus neurons after single and intermittent limbic seizures

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7 January 2021

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NMDA receptor antagonists and limbic status epilepticus: a comparison with standard anticonvulsants

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The pathological substrate of limbic epilepsy: neuronal loss in the medial dorsal thalamic nucleus as the consistent change

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7 January 2021

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Comparison of valproate and phenobarbital treatment after status epilepticus in rats.

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Mediodorsal thalamus plays a critical role in the development of limbic motor seizures.

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Ketamine, phencyclidine, and MK-801 protect against kainic acid-induced seizure-related brain damage.

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MK-801 reduced cerebral ischemic injury by inducing hypothermia.

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Chronic epileptogenic cellular alterations in the limbic system after status epilepticus.

1 reference

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Delayed sclerosis, neuroprotection, and limbic epileptogenesis after status epilepticus in the rat.

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Introduction to temporal lobe epilepsy

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Mesial temporal lobe epilepsy: what have we learned?

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Hippocampal sclerosis revisited

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Neuroprotective effects of MK-801 in vivo: selectivity and evidence for delayed degeneration mediated by NMDA receptor activation.

1 reference

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The competitive NMDA receptor antagonist CGP 40116 protects against status epilepticus-induced neuronal damage

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7 January 2021

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Neuroprotective effect of ketamine administered after status epilepticus onset.

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Confusion between neuronal apoptosis and activation of programmed cell death mechanisms in acute necrotic insults

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Status epilepticus-induced neuronal loss in humans without systemic complications or epilepsy

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Seizure-induced neuronal necrosis: implications for programmed cell death mechanisms

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7 January 2021

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Kainic acid-induced seizures produce necrotic, not apoptotic, neurons with internucleosomal DNA cleavage: implications for programmed cell death mechanisms.

1 reference

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Subcortical structures and pathways involved in convulsive seizure generation

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Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation

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Rat strain and age differences in kainic acid induced seizures

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Bilirubin induces apoptosis via activation of NMDA receptors in developing rat brain neurons

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Chronic elevation of brain GABA levels beginning two days after status epilepticus does not prevent epileptogenesis in rats

1 reference

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7 January 2021

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Spontaneous motor seizures of rats with kainate-induced epilepsy: effect of time of day and activity state.

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Cognitive functions after pilocarpine-induced status epilepticus: changes during silent period precede appearance of spontaneous recurrent seizures

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MK-801-induced neuronal damage in rats.

1 reference

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Neuronal death enhanced by N-methyl-D-aspartate antagonists

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9. Excitotoxicity and epileptic brain damage

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1 reference

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Selective protection of neuropeptide containing dentate hilar interneurons by non-NMDA receptor blockade in an animal model of status epilepticus.

1 reference

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1 reference

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1 reference

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based on heuristic

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Kainate-induced apoptotic cell death in hippocampal neurons.

1 reference

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7 January 2021

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Modification of seizure activity by electrical stimulation. II. Motor seizure

1 reference

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1 reference

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NMDA receptor activation during status epilepticus is required for the development of epilepsy

1 reference

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7 January 2021

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1 reference

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1 reference

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NMDA and kainate induce internucleosomal DNA cleavage associated with both apoptotic and necrotic cell death in the neonatal rat brain

1 reference

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Neuroprotective effects of estrogens on hippocampal cells in adult female rats after status epilepticus

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Subregional pathology of the amygdala complex and entorhinal region in surgical specimens from patients with pharmacoresistant temporal lobe epilepsy

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Identifiers

10.1016/S0306-4522(03)00027-7

1 reference

Europe PubMed Central

2 December 2017

1 reference

Europe PubMed Central

2 December 2017

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