(Q44557811)

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Suppression of fatty acid synthase in MCF-7 breast cancer cells by tea and tea polyphenols: a possible mechanism for their hypolipidemic effects

scientific article published in January 2003

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Europe PubMed Central

PubMed publication ID

4 December 2017

Suppression of fatty acid synthase in MCF-7 breast cancer cells by tea and tea polyphenols: a possible mechanism for their hypolipidemic effects (English)

1 reference

Europe PubMed Central

PubMed publication ID

4 December 2017

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author name string

Yeh CW

1

1 reference

Europe PubMed Central

PubMed publication ID

4 December 2017

Chen WJ

2

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Europe PubMed Central

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4 December 2017

Chiang CT

3

1 reference

Europe PubMed Central

PubMed publication ID

4 December 2017

Lin-Shiau SY

4

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4 December 2017

Lin JK

5

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Europe PubMed Central

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4 December 2017

publication date

1 January 2003

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Europe PubMed Central

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4 December 2017

The Pharmacogenomics Journal

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Europe PubMed Central

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4 December 2017

3

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Europe PubMed Central

PubMed publication ID

4 December 2017

5

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Europe PubMed Central

PubMed publication ID

4 December 2017

267-276

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Europe PubMed Central

PubMed publication ID

4 December 2017

The disease burden associated with overweight and obesity

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Weight-reducing effects of the plasma protein encoded by the obese gene

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Fatty-acid synthase and human cancer: new perspectives on its role in tumor biology

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Block the FAS, lose the fat.

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Green tea epigallocatechin gallate: a natural inhibitor of fatty-acid synthase

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Cancer chemoprevention by tea polyphenols through mitotic signal transduction blockade

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Activation of fatty acid synthesis during neoplastic transformation: role of mitogen-activated protein kinase and phosphatidylinositol 3-kinase

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Suppression of extracellular signals and cell proliferation through EGF receptor binding by (-)-epigallocatechin gallate in human A431 epidermoid carcinoma cells

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Fatty acid synthesis: a potential selective target for antineoplastic therapy

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Oncogenic kinase signalling

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Fatty acid synthesis and its regulation

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Inhibition of xanthine oxidase and suppression of intracellular reactive oxygen species in HL-60 cells by theaflavin-3,3'-digallate, (-)-epigallocatechin-3-gallate, and propyl gallate

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Cancer chemoprevention by tea polyphenols through modulating signal transduction pathways

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G1 events and regulation of cell proliferation

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CDK4 coexpression with Ras generates malignant human epidermal tumorigenesis

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Signaling networks--do all roads lead to the same genes?

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Diverse signaling pathways activated by growth factor receptors induce broadly overlapping, rather than independent, sets of genes

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Inhibition of UVB-induced oxidative stress-mediated phosphorylation of mitogen-activated protein kinase signaling pathways in cultured human epidermal keratinocytes by green tea polyphenol (-)-epigallocatechin-3-gallate

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Inhibitory mechanisms of tea polyphenols on the ultraviolet B-activated phosphatidylinositol 3-kinase-dependent pathway

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The animal fatty acid synthase: one gene, one polypeptide, seven enzymes

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Drug treatment of obesity: don't throw the baby out with the bath water

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Modulation of endocrine systems and food intake by green tea epigallocatechin gallate

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Stimulation of tumor-associated fatty acid synthase expression by growth factor activation of the sterol regulatory element-binding protein pathway

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Physiological and molecular mechanisms involved in nutritional regulation of fatty acid synthesis

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Protein phosphatases limit tumor motility

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Induction of apoptosis by the oolong tea polyphenol theasinensin A through cytochrome c release and activation of caspase-9 and caspase-3 in human U937 cells

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Inhibition of cyclin-dependent kinases 2 and 4 activities as well as induction of Cdk inhibitors p21 and p27 during growth arrest of human breast carcinoma cells by (-)-epigallocatechin-3-gallate

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(-)-Epigallocatechin-3-gallate blocks the induction of nitric oxide synthase by down-regulating lipopolysaccharide-induced activity of transcription factor nuclear factor-kappaB

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Fatty acid synthase is highly expressed in carcinoma, adenoma and in regenerative epithelium and intestinal metaplasia of the stomach

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Mechanisms of inhibition of the Ras-MAP kinase signaling pathway in 30.7b Ras 12 cells by tea polyphenols (-)-epigallocatechin-3-gallate and theaflavin-3,3'-digallate

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Interleukin-6 inhibits transforming growth factor-beta-induced apoptosis through the phosphatidylinositol 3-kinase/Akt and signal transducers and activators of transcription 3 pathways

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Comparison among lipid constituents in native LDL, ultra-water-soluble LDL, and vessel wall, and their significance in arteriosclerosis

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Identifiers

10.1038/SJ.TPJ.6500192

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PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

4 December 2017

ResearchGate publication ID

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