(Q44622883)

4 December 2017

title

Differences in the activation of the GFAP gene promoter by prion and viral infections (English)

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4 December 2017

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3

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4 December 2017

Matthias Titeux

1

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author name string

Maria Galou

2

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4 December 2017

Dominique Dormont

4

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4 December 2017

Vivaldo Moura Neto

5

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4 December 2017

Denise Paulin

6

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4 December 2017

publication date

1 December 2002

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4 December 2017

published in

Molecular Brain Research

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4 December 2017

volume

109

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4 December 2017

issue

1-2

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4 December 2017

page(s)

119-127

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Mice devoid of PrP are resistant to scrapie

4 December 2017

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Glial fibrillary acidic protein (GFAP): the major protein of glial intermediate filaments in differentiated astrocytes

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Neurons induce GFAP gene promoter of cultured astrocytes from transgenic mice

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Mice devoid of the glial fibrillary acidic protein develop normally and are susceptible to scrapie prions

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Cellular prion protein binds laminin and mediates neuritogenesis

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Glial fibrillary acidic protein: regulation by hormones, cytokines, and growth factors

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Strain specific and common pathogenic events in murine models of scrapie and bovine spongiform encephalopathy

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Complementary hydropathy identifies a cellular prion protein receptor

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Inactivation of the glial fibrillary acidic protein gene, but not that of vimentin, improves neuronal survival and neurite growth by modifying adhesion molecule expression

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Astrocyte-specific expression of hamster prion protein (PrP) renders PrP knockout mice susceptible to hamster scrapie

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7 January 2021

Rabies viruses infect primary cultures of murine, feline, and human microglia and astrocytes

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Cloning of a gene whose expression is increased in scrapie and in senile plaques in human brain

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7 January 2021

Immunocytochemical appearance of cytokines, prostaglandin E2 and lipocortin-1 in the CNS during the incubation period of murine scrapie correlates with progressive PrP accumulations.

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Identifiers

DOI

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4 December 2017

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