(Q44658837)

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Prolongation of insulin-induced activation of mitogen-activated protein kinases ERK 1/2 and phosphatidylinositol 3-kinase by vanadyl sulfate, a protein tyrosine phosphatase inhibitor

scientific article published on December 1, 2003

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Europe PubMed Central

5 December 2017

Prolongation of insulin-induced activation of mitogen-activated protein kinases ERK 1/2 and phosphatidylinositol 3-kinase by vanadyl sulfate, a protein tyrosine phosphatase inhibitor (English)

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Europe PubMed Central

5 December 2017

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10.1016/J.ABB.2003.09.010

https://api.crossref.org/works/10.1016/J.ABB.2003.09.010

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Jean-François Théberge

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5 December 2017

Mohamad Z. Mehdi

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3 April 2024

Sanjay K. Pandey

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3 April 2024

Ashok K. Srivastava

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1 December 2003

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https://api.elsevier.com/content/article/PII:S0003986103004806?httpAccept=text/plain

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10.1016/J.ABB.2003.09.010

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Archives of Biochemistry and Biophysics

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Molecular determinants of insulin action

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The IRS-signalling system: a network of docking proteins that mediate insulin action

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Phosphatidylinositol 3'-kinase is activated by association with IRS-1 during insulin stimulation

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Insulin-stimulated MAP-2 kinase phosphorylates and activates ribosomal protein S6 kinase II.

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Phosphoinositide 3-kinase: the key switch mechanism in insulin signalling

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Mechanisms and consequences of activation of protein kinase B/Akt

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Primary structure, tissue distribution, and expression of mouse phosphoinositide-dependent protein kinase-1, a protein kinase that phosphorylates and activates protein kinase Czeta

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Serum and glucocorticoid-inducible kinase (SGK) is a target of the PI 3-kinase-stimulated signaling pathway

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1-Phosphatidylinositol 3-kinase activity is required for insulin-stimulated glucose transport but not for RAS activation in CHO cells

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Expression of a constitutively active Akt Ser/Thr kinase in 3T3-L1 adipocytes stimulates glucose uptake and glucose transporter 4 translocation

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Protein kinase C-zeta as a downstream effector of phosphatidylinositol 3-kinase during insulin stimulation in rat adipocytes. Potential role in glucose transport

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Insulin stimulation of glycogen synthesis and glycogen synthase activity is blocked by wortmannin and rapamycin in 3T3-L1 adipocytes: evidence for the involvement of phosphoinositide 3-kinase and p70 ribosomal protein-S6 kinase

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3 April 2024

Vanadyl sulfate-stimulated glycogen synthesis is associated with activation of phosphatidylinositol 3-kinase and is independent of insulin receptor tyrosine phosphorylation

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Potential mechanism(s) involved in the regulation of glycogen synthesis by insulin.

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Requirement of protein kinase C zeta for stimulation of protein synthesis by insulin

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Protein-tyrosine phosphatase-1B acts as a negative regulator of insulin signal transduction.

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Protein-tyrosine phosphatases: biological function, structural characteristics, and mechanism of catalysis

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Effect of vanadate on elevated blood glucose and depressed cardiac performance of diabetic rats

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Anti-diabetic and toxic effects of vanadium compounds

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In vivo and in vitro studies of vanadate in human and rodent diabetes mellitus

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PTP1B inhibition and antihyperglycemic activity in the ob/ob mouse model of novel 11-arylbenzo[b]naphtho[2,3-d]furans and 11-arylbenzo[b]naphtho[2,3-d]thiophenes

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Increased abundance of specific skeletal muscle protein-tyrosine phosphatases in a genetic model of insulin-resistant obesity and diabetes mellitus.

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Skeletal muscle protein tyrosine phosphatase activity and tyrosine phosphatase 1B protein content are associated with insulin action and resistance

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Decreased skeletal muscle phosphotyrosine phosphatase (PTPase) activity towards insulin receptors in insulin-resistant Zucker rats measured by delayed Europium fluorescence

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Marked impairment of protein tyrosine phosphatase 1B activity in adipose tissue of obese subjects with and without type 2 diabetes mellitus

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PTP1B antisense oligonucleotide lowers PTP1B protein, normalizes blood glucose, and improves insulin sensitivity in diabetic mice

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In vivo effects of vanadate on hepatic glycogen metabolizing and lipogenic enzymes in insulin-dependent and insulin-resistant diabetic animals

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Activation of mitogen activated protein (MAP) kinases by vanadate is independent of insulin receptor autophosphorylation

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Vanadium salts stimulate mitogen-activated protein (MAP) kinases and ribosomal S6 kinases

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10.1016/J.ABB.2003.09.010

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Phosphatidylinositol 3-kinase requirement in activation of the ras/C-raf-1/MEK/ERK and p70(s6k) signaling cascade by the insulinomimetic agent vanadyl sulfate

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Vanadate augments insulin-stimulated insulin receptor kinase activity and prolongs insulin action in rat adipocytes. Evidence for transduction of amplitude of signaling into duration of response

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Activation of MAP kinase by insulin and vanadate in adipocytes from young and old rats.

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Vanadate-stimulated oxidation of NAD(P)H in the presence of biological membranes and other sources of O2-.

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Vanadium as a modulator of cellular regulatory cascades and oncogene expression.

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Hydrogen peroxide generated during cellular insulin stimulation is integral to activation of the distal insulin signaling cascade in 3T3-L1 adipocytes

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MKP-1 (3CH134), an immediate early gene product, is a dual specificity phosphatase that dephosphorylates MAP kinase in vivo

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Insulin-induced mitogen-activated protein (MAP) kinase phosphatase-1 (MKP-1) attenuates insulin-stimulated MAP kinase activity: a mechanism for the feedback inhibition of insulin signaling

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Insulin regulates MAP kinase phosphatase-1 induction in Hirc B cells via activation of both extracellular signal-regulated kinase (ERK) and c-Jun-N-terminal kinase (JNK).

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Antilipolytic actions of vanadate and insulin in rat adipocytes mediated by distinctly different mechanisms

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Vanadate fully stimulates insulin receptor substrate-1 associated phosphatidyl inositol 3-kinase activity in adipocytes from young and old rats

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Multifunctional actions of vanadium compounds on insulin signaling pathways: evidence for preferential enhancement of metabolic versus mitogenic effects

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10.1016/J.ABB.2003.09.010

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Europe PubMed Central

5 December 2017

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Europe PubMed Central

5 December 2017

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