(Q44756792)

English

The origin of fluorescence in the neuronal ceroid lipofuscinoses (Batten disease) and neuron cultures from affected sheep for studies of neurodegeneration

scientific article published in May 2002

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scholarly article

1 reference

Europe PubMed Central

6 December 2017

The origin of fluorescence in the neuronal ceroid lipofuscinoses (Batten disease) and neuron cultures from affected sheep for studies of neurodegeneration (English)

1 reference

Europe PubMed Central

6 December 2017

1

object named as

David N Palmer

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Europe PubMed Central

6 December 2017

author name string

Manfred J Oswald

2

1 reference

Europe PubMed Central

6 December 2017

Valerie J Westlake

3

1 reference

Europe PubMed Central

6 December 2017

Graham W Kay

4

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Europe PubMed Central

6 December 2017

publication date

1 May 2002

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Europe PubMed Central

6 December 2017

number of pages

15

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Archives of Gerontology and Geriatrics

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Europe PubMed Central

6 December 2017

34

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Europe PubMed Central

6 December 2017

3

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Europe PubMed Central

6 December 2017

343-357

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Europe PubMed Central

6 December 2017

Canine ceroid lipofuscinosis, a model for ageing of the human isocortex

1 reference

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7 January 2021

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Synthesis and characterization of the fluorescent products derived from malonaldehyde and amino acids

1 reference

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7 January 2021

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Inactivation of ribonuclease and other enzymes by peroxidizing lipids and by malonaldehyde

1 reference

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7 January 2021

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Peroxidation of subcellular organelles: formation of lipofuscinlike fluorescent pigments

1 reference

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7 January 2021

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Apparent loss and hypertrophy of interneurons in a mouse model of neuronal ceroid lipofuscinosis: evidence for partial response to insulin-like growth factor-1 treatment

1 reference

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7 January 2021

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When lysosomes get old.

1 reference

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Lipofuscin and abnormalities in colloid in the equine thyroid gland in relation to age

1 reference

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7 January 2021

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A lysosomal proteinase, the late infantile neuronal ceroid lipofuscinosis gene (CLN2) product, is essential for degradation of a hydrophobic protein, the subunit c of ATP synthase.

1 reference

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7 January 2021

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The sequence of the major protein stored in ovine ceroid lipofuscinosis is identical with that of the dicyclohexylcarbodiimide-reactive proteolipid of mitochondrial ATP synthase

1 reference

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7 January 2021

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Measurement of fluorescent lipid peroxidation products in biological systems and tissues.

1 reference

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7 January 2021

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Analysis of dolichyl pyrophosphoryl oligosaccharides in purified storage cytosomes from ovine ceroid-lipofuscinosis

1 reference

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7 January 2021

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The properties of isolated human cardiac age pigment. I. Preparation and physical properties

1 reference

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THE PROPERTIES OF ISOLATED HUMAN CARDIAC AGE PIGMENT. II. CHEMICAL AND ENZYMATIC PROPERTIES

1 reference

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7 January 2021

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Disease-specific pathology in neurons cultured from sheep affected with ceroid lipofuscinosis

1 reference

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7 January 2021

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inferred from DOI database lookup

Inhibitors of lysosomal enzymes: accumulation of lipofuscin-like dense bodies in the brain

1 reference

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Lipofuscin in bovine muscle and brain: a model for studying age pigment

1 reference

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7 January 2021

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The analytical approach to the nature of lipofuscin (age pigment).

1 reference

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7 January 2021

based on heuristic

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Purification and characterization of bovine brain lysosomal pepstatin-insensitive proteinase, the gene product deficient in the human late-infantile neuronal ceroid lipofuscinosis

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4943%2802%2900011-0

7 January 2021

based on heuristic

inferred from DOI database lookup

In vitro culture of neurons from sheep with Batten disease.

1 reference

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7 January 2021

based on heuristic

inferred from DOI database lookup

Canine ceroid-lipofuscinosis-a model for human neuronal ceroid-lipofuscinosis and aging.

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4943%2802%2900011-0

7 January 2021

based on heuristic

inferred from DOI database lookup

The lysosomal system in neuronal cell death: a review

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4943%2802%2900011-0

7 January 2021

based on heuristic

inferred from DOI database lookup

Changes in GABAergic neuron distribution in situ and in neuron cultures in ovine (OCL6) Batten disease

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4943%2802%2900011-0

7 January 2021

based on heuristic

inferred from DOI database lookup

Phospholipid fatty acids in brains of normal sheep and sheep with ceroid-lipofuscinosis.

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4943%2802%2900011-0

7 January 2021

based on heuristic

inferred from DOI database lookup

Mitochondrial ATP synthase subunit c storage in the ceroid-lipofuscinoses (Batten disease)

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4943%2802%2900011-0

7 January 2021

based on heuristic

inferred from DOI database lookup

Storage bodies in the ceroid-lipofuscinoses (Batten disease): low-molecular-weight components, unusual amino acids and reconstitution of fluorescent bodies from non-fluorescent components

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4943%2802%2900011-0

7 January 2021

based on heuristic

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Batten disease and the ATP synthase subunit c turnover pathway: raising antibodies to subunit c.

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4943%2802%2900011-0

7 January 2021

based on heuristic

inferred from DOI database lookup

Different patterns of hydrophobic protein storage in different forms of neuronal ceroid lipofuscinosis (NCL, Batten disease)

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4943%2802%2900011-0

7 January 2021

based on heuristic

inferred from DOI database lookup

Electrospray ionisation mass spectrometry (ESI/MS) of ceroid lipofuscin protein; a model system for the study of F0 inhibitor interactions with mitochondrial subunit C

1 reference

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7 January 2021

based on heuristic

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A mutation in the ovine cathepsin D gene causes a congenital lysosomal storage disease with profound neurodegeneration

1 reference

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7 January 2021

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inferred from DOI database lookup

Identifiers

10.1016/S0167-4943(02)00011-0

1 reference

Europe PubMed Central

6 December 2017

1 reference

Europe PubMed Central

6 December 2017

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