(Q44939694)

English

Desensitization of the permeability transition pore by cyclosporin a prevents activation of the mitochondrial apoptotic pathway and liver damage by tumor necrosis factor-alpha

scientific article

Desensitization of the Permeability Transition Pore by Cyclosporin A Prevents Activation of the Mitochondrial Apoptotic Pathway and Liver Damage by Tumor Necrosis Factor-α

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scholarly article

1 reference

Europe PubMed Central

7 December 2017

Desensitization of the permeability transition pore by cyclosporin a prevents activation of the mitochondrial apoptotic pathway and liver damage by tumor necrosis factor-alpha (English)

1 reference

Europe PubMed Central

7 December 2017

apoptotic process

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desensitization

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based on heuristic

inferred from title

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based on heuristic

inferred from title

3

1 reference

Europe PubMed Central

7 December 2017

María Isabel Soriano

object named as

Maria Eugenia Soriano

1

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author name string

Luca Nicolosi

2

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Europe PubMed Central

7 December 2017

language of work or name

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publication date

16 June 2004

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Europe PubMed Central

7 December 2017

Journal of Biological Chemistry

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Europe PubMed Central

7 December 2017

279

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Europe PubMed Central

7 December 2017

35

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Europe PubMed Central

7 December 2017

36803-36808

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Europe PubMed Central

7 December 2017

Regulation of cell death: the calcium-apoptosis link

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Apoptosis-based therapies

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Mice deficient for the 55 kd tumor necrosis factor receptor are resistant to endotoxic shock, yet succumb to L. monocytogenes infection

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Mice lacking the tumour necrosis factor receptor 1 are resistant to TNF-mediated toxicity but highly susceptible to infection by Listeria monocytogenes

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Tumor necrosis factor receptor and Fas signaling mechanisms.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Caspase cleaved BID targets mitochondria and is required for cytochrome c release, while BCL-XL prevents this release but not tumor necrosis factor-R1/Fas death

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Prevention of apoptosis by Bcl-2: release of cytochrome c from mitochondria blocked

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

The release of cytochrome c from mitochondria: a primary site for Bcl-2 regulation of apoptosis

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Bax directly induces release of cytochrome c from isolated mitochondria

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Bax-induced cytochrome C release from mitochondria is independent of the permeability transition pore but highly dependent on Mg2+ ions

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Bcl-2 inhibits the mitochondrial release of an apoptogenic protease

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

The mitochondrial permeability transition is required for tumor necrosis factor alpha-mediated apoptosis and cytochrome c release

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Arachidonic acid causes cell death through the mitochondrial permeability transition. Implications for tumor necrosis factor-alpha aopototic signaling

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

A distinct pathway remodels mitochondrial cristae and mobilizes cytochrome c during apoptosis

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

A mitochondrial perspective on cell death

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Arachidonic acid released by phospholipase A(2) activation triggers Ca(2+)-dependent apoptosis through the mitochondrial pathway

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Collagen VI deficiency induces early onset myopathy in the mouse: an animal model for Bethlem myopathy.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Early resistance to cell death and to onset of the mitochondrial permeability transition during hepatocarcinogenesis with 2-acetylaminofluorene

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Requirement for GD3 ganglioside in CD95- and ceramide-induced apoptosis.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Ceramide induces hepatocyte cell death through disruption of mitochondrial function in the rat.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Commitment to apoptosis by GD3 ganglioside depends on opening of the mitochondrial permeability transition pore.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Ceramide 1-phosphate is a direct activator of cytosolic phospholipase A2

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Acidic sphingomyelinase downregulates the liver-specific methionine adenosyltransferase 1A, contributing to tumor necrosis factor-induced lethal hepatitis

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

On the effects of paraquat on isolated mitochondria. Evidence that paraquat causes opening of the cyclosporin A-sensitive permeability transition pore synergistically with nitric oxide.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Bid-deficient mice are resistant to Fas-induced hepatocellular apoptosis

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Role of FAN in tumor necrosis factor-alpha and lipopolysaccharide-induced interleukin-6 secretion and lethality in D-galactosamine-sensitized mice

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Activation of pro-death Bcl-2 family proteins and mitochondria apoptosis pathway in tumor necrosis factor-alpha-induced liver injury

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

A novel cytoplasmic domain of the p55 tumor necrosis factor receptor initiates the neutral sphingomyelinase pathway

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

FAN, a novel WD-repeat protein, couples the p55 TNF-receptor to neutral sphingomyelinase

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Four deaths and a funeral: from caspases to alternative mechanisms.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

The mitochondrial permeability transition, release of cytochrome c and cell death. Correlation with the duration of pore openings in situ

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Glutamate-induced neuronal death: A succession of necrosis or apoptosis depending on mitochondrial function

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Apoptosis to necrosis switching downstream of apoptosome formation requires inhibition of both glycolysis and oxidative phosphorylation in a BCL-X(L)- and PKB/AKT-independent fashion

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Cyclophilins and their possible role in the stress response

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Interactions of cyclophilin with the mitochondrial inner membrane and regulation of the permeability transition pore, and cyclosporin A-sensitive channel.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Vasopressin type 1A receptor up-regulation by cyclosporin A in vascular smooth muscle cells is mediated by superoxide.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Endotoxin-induced mitochondrial damage correlates with impaired respiratory activity

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Opening of the mitochondrial permeability transition pore causes matrix expansion and outer membrane rupture in Fas-mediated hepatic apoptosis in mice

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Direct Effect of Ceramide on the Mitochondrial Electron Transport Chain Leads to Generation of Reactive Oxygen Species

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Defective TNF-α–mediated hepatocellular apoptosis and liver damage in acidic sphingomyelinase knockout mice

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

A Ubiquinone-binding Site Regulates the Mitochondrial Permeability Transition Pore

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M405297200

21 January 2018

Identifiers

10.1074/JBC.M405297200

1 reference

Europe PubMed Central

7 December 2017

1 reference

Europe PubMed Central

7 December 2017

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