(Q44963024)

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8302A/C and (TTA)n polymorphisms in the HMG-CoA reductase gene may be associated with some plasma lipid metabolic phenotypes in patients with coronary heart disease

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Europe PubMed Central

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7 December 2017

8302A/C and (TTA)n polymorphisms in the HMG-CoA reductase gene may be associated with some plasma lipid metabolic phenotypes in patients with coronary heart disease (English)

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Europe PubMed Central

PubMed publication ID

7 December 2017

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Zhiguang Su

4

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Yu Tong

1

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7 December 2017

Sizhong Zhang

2

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7 December 2017

Hai Li

3

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7 December 2017

Xiangdong Kong

5

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7 December 2017

Hekun Liu

6

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7 December 2017

Cuiying Xiao

7

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7 December 2017

Yan Sun

8

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7 December 2017

Jia Jun Shi

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7 December 2017

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1 March 2004

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7 December 2017

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7 December 2017

39

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7 December 2017

3

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7 December 2017

239-241

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7 December 2017

https://scigraph.springernature.com/pub.10.1007/s11745-004-1225-3

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A high frequency of length polymorphisms in repeated sequences adjacent to Alu sequences

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Pravastatin sodium, an inhibitor of 3-hydroxy-3-methylglutaryl coenzyme A reductase, decreases serum total cholesterol in Japanese White rabbits by two different mechanisms

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A simple salting out procedure for extracting DNA from human nucleated cells

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Inhibition of acyl-CoA: cholesterol acyltransferase decreases apolipoprotein B-100-containing lipoprotein secretion from HepG2 cells.

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Coronary heart disease: a disorder of growth

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Properties of triglyceride-rich and cholesterol-rich lipoproteins in the remnant-like particle fraction of human blood plasma.

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Differences in postprandial concentrations of very-low-density lipoprotein and chylomicron remnants between normotriglyceridemic and hypertriglyceridemic men with and without coronary heart disease.

1 reference

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ScrFI polymorphism in the 2nd intron of the HMGCR gene

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Lipid and fatty acid composition of canine lipoproteins

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Identifiers

10.1007/S11745-004-1225-3

1 reference

Europe PubMed Central

PubMed publication ID

7 December 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

7 December 2017

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